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3. Dysregulation of mitochondrial and proteolysosomal genes in Parkinson’s disease myeloid cells

9. Dystonia

10. Differences in Sex‐Specific Frequency of Glucocerebrosidase Variant Carriers and Familial Parkinsonism

11. Association of Olfactory Performance With Motor Decline and Age at Onset in People With Parkinson Disease and the LRRK2 G2019S Variant

12. Parkinson Disease and Subthalamic Nucleus Deep Brain Stimulation: Cognitive Effects inGBAMutation Carriers

14. Association of Olfactory Performance With Motor Decline and Age at Onset in People With Parkinson Disease and the G2019S Variant.

16. Cognitive Functioning of Glucocerebrosidase (GBA) Non-manifesting Carriers

17. Parkinson disease phenotype in Ashkenazi jews with and without LRRK2 G2019S mutations

18. Association of Olfactory Performance With Motor Decline and Age at Onset in People With Parkinson Disease and the LRRK2G2019S Variant

19. Parkinson Disease and Subthalamic Nucleus Deep Brain Stimulation: Cognitive Effects in GBA Mutation Carriers.

21. Discordant transcriptional signatures of mitochondrial genes in Parkinson’s disease human myeloid cells

26. Longitudinal outcomes of LRRK2 gene mutation parkinsonism undergoing either Subthalamic Nucleus (STN) or Globus Pallidus Internus (GPi) Deep-Brain stimulation (DBS): a multi-group comparison (P1.8-029)

32. Neuropsychiatric characteristics of GBA-associated Parkinson disease

34. Glucocerebrosidase enzyme activity in GBA mutation Parkinson’s disease

36. Spatial Discrimination Threshold Abnormalities are not Detected in a Pilot Study of DYT6 Dystonia Mutation Carriers

39. Reorganization of corticostriatal circuits in healthy G2019S LRRK2 carriers

41. Neuropsychiatric Features of GBA-Associated Parkinson Disease (P2.024)

43. Glucocerebrosidase Enzyme Activity in GBA1 Mutation Parkinson Disease (P4.042)

47. Lower cognitive performance in healthy G2019S LRRK2 mutation carriers

50. LRRK2 G2019S mutations are associated with an increased cancer risk in Parkinson disease.

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