15 results on '"Senapati, Shantibhushan"'
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2. Data from MUC4 Mucin Potentiates Pancreatic Tumor Cell Proliferation, Survival, and Invasive Properties and Interferes with Its Interaction to Extracellular Matrix Proteins
3. Supplementary Data, Chaturvedi, et al. from MUC4 Mucin Potentiates Pancreatic Tumor Cell Proliferation, Survival, and Invasive Properties and Interferes with Its Interaction to Extracellular Matrix Proteins
4. SMRT and NCoR1 fine-tune inflammatory versus tolerogenic balance in dendritic cells by differentially regulating STAT3 signaling
5. NCoR1 and SMRT Fine-Tune Inflammatory Versus Tolerogenic Balance in Dendritic Cells By Differentially Regulating STAT3 Signaling
6. Long-read 16S-seq reveals nasopharynx microbial dysbiosis and enrichment of Mycobacterium and Mycoplasma in COVID-19 patients: a potential source of co-infection
7. Long-read 16S-seq reveals nasopharynx microbial dysbiosis and enrichment of Mycobacterium and Mycoplasma in COVID-19 patients: A source of co-infection
8. Nanopore 16S rRNA sequencing reveals alterations in nasopharyngeal microbiome and enrichment ofMycobacteriumandMycoplasmain patients with COVID 19
9. Tumours: Targeting of Monoclonal Antibodies for Imaging and Potential for Therapy
10. S1951 MUC 4 Suppression is an Early Event in Colorectal Carcinogenesis (CRC): Potential Biomarker for Screening and Chemoprevention
11. W1747 MUC4 and AKT/β-Catenin Mediated Control of Cell Proliferation in Early Colon Carcinogenesis
12. S2044 MUC17: A Novel Tumor Suppressor Gene in Colon Carcinogenesis?
13. W1878 Muc4: A Novel Tumor Suppressor Gene Impacts Upon Initiation and Progression Phases of Colorectal Carcinogenesis
14. MUC4 Mucin Potentiates Pancreatic Tumor Cell Proliferation, Survival, and Invasive Properties and Interferes with Its Interaction to Extracellular Matrix Proteins
15. Analysis of Indian SARS-CoV-2 Genomes Reveals Prevalence of D614G Mutation in Spike Protein Predicting an Increase in Interaction With TMPRSS2 and Virus Infectivity.
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