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Rhinophyma is a disease going with subcutaneous tissue thickening of the nose, including sebaceous gland hypertrophy and fibrosis. Progressive enlargement and irregular thickening of the nasal skin is characteristic of the disease. Surgical excision is commonly used treatment method. In the present case, nasal dorsal and alar defect after treated with totally excision and full-thickness skin graft of rhinophyma mass were repaired by using supratrochlear artery based myofascial forehead flap with a little skin island.

This is a case report about a 62-year-old gentleman who presented at the Otolaryngology department with a rhinophyma. Rhinophyma is a swelling of the soft tissue of the nose due to sebaceous gland hypertrophy, lymphoedema and fibrosis. Photography of the patient's condition highlights not only the features of the disease but is useful when comparing the contours and shape of the nose after surgical excision. Images of a relatively large rhinophyma are useful not only for their visual but also their educational value.

Otophyma is the term used for sebaceous gland hypertrophy and hyperplasia of the ear. It is usually the end stage of rosacea--a common, chronic, cutaneous disorder of unknown etiology. We hereby present a rare case of otophyma with its clinical features, its etiology, diagnosis, and review of medical and surgical therapy. This patient remained undiagnosed and mismanaged for many years before she was finally diagnosed with this rare condition and successfully managed by surgical technique. To our knowledge, this is the second case of otophyma presented in the otolaryngological literature.

An isolated affected 19-year-old male with hypohidrotic ectodermal dysplasia (HED) had rare features of the syndrome such as recurrent otitis and multiple sebaceous gland papules of the face. Sebaceous gland hypertrophy is puzzling in the context of HED.

In the hairless hamster, progesterone can antagonize dihydrotestosterone-mediated hypertrophy of the sebaceous gland.

The development of acne during puberty is intimately associated with and dependent upon the sebaceous gland hypertrophy that occurs at this time. It is our feeling that sebum provides the "fuel" for the development in a susceptible individual of the inflammatory lesions characteristic of the disease. 1 Our data indicates that the severest cases of acne occur in patients with large sebaceous glands and high sebum production. 2 Although there are undoubtedly other governing factors in the pathogenesis of the disease, the fact remains that suppression of the large postpubertal glands can favorably alter the course of the disease. For example, fractional superficial x-ray therapy exerts its beneficial effect on acne by suppression of sebaceous gland size. However, the response is only temporary, and after completion of a safely tolerated course of x-ray, the glands promptly regenerate, and as such the disorder may reappear. 3 It is well known that