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2. A prospective longitudinal cohort study on risk factors for COVID-19 vaccination failure (RisCoin): methods, procedures and characterization of the cohort

5. Diet prevents the expansion of segmented filamentous bacteria and ileo-colonic inflammation in a model of Crohn’s disease

8. Author Correction: Somatic mosaicism and common genetic variation contribute to the risk of very-early-onset inflammatory bowel disease

11. NBAS Variants Are Associated with Quantitative and Qualitative NK and B Cell Deficiency

14. Infantile and Very Early Onset Inflammatory Bowel Disease: A Multicenter Study.

15. Exclusive enteral nutrition initiates individual protective microbiome changes to induce remission in pediatric Crohn’s disease

16. Children with Localized Crohn's Disease Benefit from Early Ileocecal Resection and Perioperative Anti-Tumor Necrosis Factor Therapy.

17. Exclusive Enteral Nutrition Initiates Protective Microbiome Changes to Induce Remission in Pediatric Crohn's Disease

18. Clinical Genomics for the Diagnosis of Monogenic forms of Inflammatory Bowel Disease: A Position Paper from The Paediatric IBD Porto Group of ESPGHAN

19. Somatic mosaicism and common genetic variation contribute to the risk of very-early-onset inflammatory bowel disease

20. A variant in IL6ST with a selective IL-11 signaling defect in human and mouse

21. Is Autologous Fecal Microbiota Transfer after Exclusive Enteral Nutrition in Pediatric Crohn’s Disease Patients Rational and Feasible? Data from a Feasibility Test

22. Fecal Microbiota Transplantation for Recurrent Clostridium difficile Infection and Other Conditions in Children: A Joint Position Paper From the North American Society for Pediatric Gastroenterology, Hepatology, and Nutrition and the European Society for Pediatric Gastroenterology, Hepatology, and Nutrition

23. Fecal Microbiota Transplantation for Recurrent Clostridium difficile Infection and Other Conditions in Children: A Joint Position Paper From the North American Society for Pediatric Gastroenterology, Hepatology, and Nutrition and the European Society for Pediatric Gastroenterology, Hepatology, and Nutrition

27. Characterization of Crohn disease in X-linked inhibitor of apoptosis–deficient male patients and female symptomatic carriers

31. Diet prevents the expansion of segmented filamentous bacteria and ileo-colonic inflammation in a model of Crohn’s disease

32. Author Correction: Somatic mosaicism and common genetic variation contribute to the risk of very-early-onset inflammatory bowel disease.

33. Fecal Microbiota Transfer (FMT) in Children and Adolescents - Review and statement by the GPGE microbiome working group

37. 608: DIET CONTROLS SEGMENTED FILAMENTOUS BACTERIA IN DRIVING CROHN'S DISEASE-LIKE INFLAMMATION IN TNFΔARE MICE

40. Anti-TNF therapy for inflammatory bowel disease in patients with neurodegenerative Niemann-Pick disease Type C

42. Esophageal Perforation and EVAC in Pediatric Patients: A Case Series of Four Children

43. Deficiency in X-linked inhibitor of apoptosis protein promotes susceptibility to microbial triggers of intestinal inflammation

45. Selective loss of function variants in IL6ST cause Hyper-IgE syndrome with distinct impairments of T-cell phenotype and function

46. Following Pediatric and Adult IBD Patients through the COVID-19 Pandemic: Changes in Psychosocial Burden and Perception of Infection Risk and Harm over Time

47. Clinical Genomics for the Diagnosis of Monogenic Forms of Inflammatory Bowel Disease:A Position Paper From the Paediatric IBD Porto Group of European Society of Paediatric Gastroenterology, Hepatology and Nutrition

48. 5'-triphosphate-siRNA: turning gene silencing and Rig-I activation against melanoma

49. sj-pdf-1-jla-10.1177_2472630320969147 – Supplemental material for Metabolomic Signatures in Pediatric Crohn’s Disease Patients with Mild or Quiescent Disease Treated with Partial Enteral Nutrition: A Feasibility Study

50. Crohn' s Disease Exclusion Diet - an alternative to exlusive enteral nutritional therapy in children and adolescentswith Crohn 's disease? Statement of the GPGE working groups CEDATA and Nutrition/Nutrition Medicine

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