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15. Small increases in insulin inhibit hepatic glucose production solely caused by an effect on glycogen metabolism.

16. 35th Annual Meeting of the European Association for the Study of Diabetes

17. The tracing of the pathway of mevalonate's metabolism to other than sterols.

18. The nature of the pentose pathway in liver.

19. Quantitative estimation of the pathways followed in the conversion to glycogen of glucose administered to the fasted rat.

20. Pentose pathway in human liver.

21. A method for quantitating the contributions of the pathways of acetoacetate formation and its application to diabetic ketosis in vivo.

22. Pathways of acetoacetate's formation in liver and kidney.

23. Pathways of acetone's metabolism in the rat.

24. 35th Annual Meeting of the European Association for the Study of Diabetes : Brussels, Belgium, 28 September-2 October 1999

31. Gluconeogenesis and glucuronidation in liver in vivo and the heterogeneity of hepatocyte function.

32. Contribution of gluconeogenesis and glycogenolysis to hepatic glucose production in acromegaly before and after pituitary microsurgery.

33. Sources of blood glycerol during fasting.

35. Quantitative contributions of gluconeogenesis to glucose production during fasting in type 2 diabetes mellitus.

36. A probing dose of phenylacetate does not affect glucose production and gluconeogenesis in humans.

37. Origins of the hydrogen bound to carbon 1 of glucose in fasting: significance in gluconeogenesis quantitation.

38. Contributions of net hepatic glycogenolysis and gluconeogenesis to glucose production in cirrhosis.

39. Quantifying gluconeogenesis during fasting.

40. Quantitation of glycogen/glucose-1-P cycling in liver.

41. Fructose-6-phosphate cycling and the pentose cycle in hyperthyroidism.

43. Pathways of hepatic glycogen formation in humans following ingestion of a glucose load in the fed state.

44. On the lack of formation of L-(+)-3-hydroxybutyrate by liver.

46. omega-Oxidation of fatty acids and the acetylation p-aminobenzoic acid.

47. Ketone body production in diabetic ketosis by other than liver.

48. A chemical degradation of 3-hydroxybutyric acid.

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