128 results on '"Schuhmacher, Swenja"'
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2. Nitrate tolerance as a model of vascular dysfunction: Roles for mitochondrial aldehyde dehydrogenase and mitochondrial oxidative stress
3. AT 1-receptor blockade by telmisartan upregulates GTP-cyclohydrolase I and protects eNOS in diabetic rats
4. ALDH-2 deficiency increases cardiovascular oxidative stress---Evidence for indirect antioxidative properties
5. Comparison of Linagliptin, Sitagliptin and Liraglutide Effects on Survival and Vascular Complications in Experimental Sepsis: 128
6. Vascular Dysfunction in Experimental Diabetes Is Improved by Pentaerithrityl Tetranitrate but Not Isosorbide-5-Mononitrate Therapy
7. Vascular Dysfunction in Streptozotocin-Induced Experimental Diabetes Strictly Depends on Insulin Deficiency
8. Comparison of Direct and Indirect Antioxidant Effects of Linagliptin (BI 1356, ONDERO) with other Gliptins - Evidence for Anti-inflammatory Properties of Linagliptin: 557
9. Vascular Dysfunction in Nitroglycerin-induced Nitrate Tolerance is Improved by Telmisartan Therapy - Suppression of the RAAS and PKC Pathway: 322
10. Vascular Dysfunction in Streptozotocin-induced Experimental Diabetes Strictly Depends on Insulin Deficiency: 73
11. Lysozyme-M Positive Myelomonocytic cells Mediate Angiotensin-II induced Arterial Hypertension: 33
12. Effects of the Organic Nitrates PETN and ISDN on Vascular Dysfunction and Oxidative Stress in an Experimental Model of Diabetes Mellitus type 1: 159
13. Manganese superoxide dismutase and aldehyde dehydrogenase deficiency increase mitochondrial oxidative stress and aggravate age-dependent vascular dysfunction
14. AT1-receptor blockade by telmisartan upregulates GTP-cyclohydrolase I and protects eNOS in diabetic rats
15. Telmisartan Co-Therapy Inhibits all Important Sources of Oxidative Stress and Improves Endothelial Dysfunction in an Experimental Model of Diabetes Mellitus (Type 1): 75
16. Role of the β1-Subunit of the Large Conductance Potassium Chanel on Nitrate Tolerance and on Angiotensis II Induced Oxidative Stress: 74
17. Mitochondrial oxidative stress and nitrate tolerance – comparison of nitroglycerin and pentaerithrityl tetranitrate in Mn-SOD+/- mice
18. Organic Nitrates and Nitrate Resistance in Diabetes: The Role of Vascular Dysfunction and Oxidative Stress with Emphasis on Antioxidant Properties of Pentaerithrityl Tetranitrate
19. Einfluss der AMPK-Aktivität auf Endothelfunktion, oxidativen Stress und vaskuläre Inflammation
20. Nitroglycerin-induced endothelial dysfunction and tolerance involve adverse phosphorylation and S-glutathionylation of endothelial nitric oxide synthase — Beneficial effects of therapy with the AT1-receptor blocker telmisartan
21. Lysozyme M positive monocytes mediate angiotensin II-induced arterial hypertension and vascular dysfunction
22. Molecular mechanisms of the crosstalk between mitochondrial and NADPH oxidase derived reactive oxygen species in white blood cells — Implications for cardiovascular diseases
23. Comparison of direct and indirect antioxidant effects of linagliptin with other gliptins — Evidence for antioxidant and antiinflammatory properties of linagliptin
24. Molecular mechanisms of the crosstalk between mitochondrial and NADPH oxidase derived reactive oxygen species in white blood cells – implications for cardiovascular diseases
25. Glutathione peroxidase‐1‐deficiency enhances age‐dependent vascular dysfunction
26. Abstract 16785: Molecular Mechanisms of the Crosstalk Between Mitochondrial and NADPH Oxidase Derived Reactive Oxygen Species in White Blood Cells - Implications for Cardiovascular Diseases
27. Nitroglycerin-Induced Endothelial Dysfunction and Tolerance Involve Adverse Phosphorylation and S -Glutathionylation of Endothelial Nitric Oxide Synthase
28. Lysozyme M–Positive Monocytes Mediate Angiotensin II–Induced Arterial Hypertension and Vascular Dysfunction
29. CIRCULATING AND INFILTRATING MONOCYTIC CELLS MEDIATE ANGIOTENSIN-II INDUCED ARTERIAL HYPERTENSION
30. α1AMP-Activated Protein Kinase Preserves Endothelial Function During Chronic Angiotensin II Treatment by Limiting Nox2 Upregulation
31. Effects of PETN and ISDN on vascular dysfunction and oxidative stress in an experimental model of Diabetes
32. Pentaerythritol Tetranitrate Improves Angiotensin II–Induced Vascular Dysfunction via Induction of Heme Oxygenase-1
33. Vascular Dysfunction in Streptozotocininduced Experimental Diabetes Strictly Depends on Insulin Deficiency
34. Vascular Dysfunction in Nitroglycerininduced Nitrate Tolerance is Improved by Telmisartan Therapy — Suppression of the RAAS and PKC Pathway
35. Abstract 5522: Improvement of Angiotensin-II-Induced Vascular Dysfunction and Oxidative Stress by the Organic Nitrate Pentaerithrityl Tetranitrate Depends on Heme Oxygenase-1-Induction
36. When Metabolism Rules Perfusion
37. Suppression of the JNK Pathway by Induction of a Metabolic Stress Response Prevents Vascular Injury and Dysfunction
38. First Evidence for a Crosstalk Between Mitochondrial and NADPH Oxidase-Derived Reactive Oxygen Species in Nitroglycerin-Triggered Vascular Dysfunction
39. MnSOD and ALDH‐2 deficiency potentiate aging‐associated vascular dysfunction, mitochondrial oxidative stress and mtDNA strand breaks
40. Mitochondrial oxidative stress and nitrate tolerance – comparison of nitroglycerin and pentaerithrityl tetranitrate in Mn-SOD+/-mice
41. 393: Could the Active Tumor Suppressor PTEN Function as a Prognostic Marker for Metastasis in Renal Cell Carcinoma?
42. PSS128 - Comparison of Linagliptin, Sitagliptin and Liraglutide Effects on Survival and Vascular Complications in Experimental Sepsis
43. Mitochondrial oxidative stress and nitrate tolerance -- comparison of nitroglycerin and pentaerithrityl tetranitrate in Mn-SOD+/- mice.
44. Effects of the Organic Nitrates PETN and ISDN on Vascular Dysfunction and Oxidative Stress in an Experimental Model of Diabetes Mellitus type 1
45. Role of the beta 1-subunit of the large conductance potassium chanel on nitrate tolerance and on angiotensis II induced oxidative stress
46. The Th1 Transcription Factor T Box Expressed in T Cells (T-bet) mediates Angiotensin II Induced Vascular Dysfunction and Oxidative Stress
47. AMP-activated Protein Kinase Mediates Vascular Protective Effects During Exercise Training
48. Knockout of vascular AMPK leads to increased mitochondrial stress and decreased vasodilator sensitivity towards organic nitrates
49. Pleiotropic Antioxidant Effects of Telmisartan Improve Nitroglycerin-Triggered Vascular Dysfunction
50. Comparison of the effects of PETN, ISMN and ISDN in experimental diabetes mellitus type 1 and in arterial hypertension
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