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1. Genetic risk impacts the association of menopausal hormone therapy with colorectal cancer risk

3. Fine-mapping analysis including over 254,000 East Asian and European descendants identifies 136 putative colorectal cancer susceptibility genes

4. Genome-Wide Interaction Analysis of Genetic Variants With Menopausal Hormone Therapy for Colorectal Cancer Risk.

6. Probing the diabetes and colorectal cancer relationship using gene – environment interaction analyses

7. Genetically proxied glucose-lowering drug target perturbation and risk of cancer: a Mendelian randomisation analysis

9. Prospective and Mendelian randomization analyses on the association of circulating fatty acid binding protein 4 (FABP-4) and risk of colorectal cancer

10. Combining Asian and European genome-wide association studies of colorectal cancer improves risk prediction across racial and ethnic populations

11. Body size and risk of colorectal cancer molecular defined subtypes and pathways: Mendelian randomization analyses

12. Prognostic role of detailed colorectal location and tumor molecular features: analyses of 13,101 colorectal cancer patients including 2994 early-onset cases

13. Elucidating the Risk of Colorectal Cancer for Variants in Hereditary Colorectal Cancer Genes

14. Two genome-wide interaction loci modify the association of nonsteroidal anti-inflammatory drugs with colorectal cancer

15. Pre-vaccination transcriptomic profiles of immune responders to the MUC1 peptide vaccine for colon cancer prevention

16. Supplementary Figure S2 from Randomized, Double-Blind, Placebo-Controlled Trial of MUC1 Peptide Vaccine for Prevention of Recurrent Colorectal Adenoma

17. Supplementary Table S2 from Randomized, Double-Blind, Placebo-Controlled Trial of MUC1 Peptide Vaccine for Prevention of Recurrent Colorectal Adenoma

18. Supplementary Methods S2 from Randomized, Double-Blind, Placebo-Controlled Trial of MUC1 Peptide Vaccine for Prevention of Recurrent Colorectal Adenoma

19. Identifying colorectal cancer caused by biallelic MUTYH pathogenic variants using tumor mutational signatures

21. Mendelian randomisation study of age at menarche and age at menopause and the risk of colorectal cancer

23. Effectiveness of Colonoscopy Screening vs Sigmoidoscopy Screening in Colorectal Cancer

24. Assessing aneuploidy with repetitive element sequencing

25. Identifying Novel Susceptibility Genes for Colorectal Cancer Risk From a Transcriptome-Wide Association Study of 125,478 Subjects

32. Relationship of prediagnostic body mass index with survival after colorectal cancer: Stage‐specific associations

33. UNSEG: unsupervised segmentation of cells and their nuclei in complex tissue samples

35. Proceedings of the fourth international molecular pathological epidemiology (MPE) meeting

36. Corrigendum: genome-wide association study of colorectal cancer identifies six new susceptibility loci.

37. Erratum: Corrigendum: Genome-wide association study of colorectal cancer identifies six new susceptibility loci

38. Identification of a common variant with potential pleiotropic effect on risk of inflammatory bowel disease and colorectal cancer

39. Genome-wide association study of colorectal cancer identifies six new susceptibility loci.

44. Leisure time television watching, computer use and risks of breast, colorectal and prostate cancer: A Mendelian randomisation analysis.

45. Supplemental Table 4 from Elevated EVL Methylation Level in the Normal Colon Mucosa Is a Potential Risk Biomarker for Developing Recurrent Adenomas

46. Supplemental Table 6 from Elevated EVL Methylation Level in the Normal Colon Mucosa Is a Potential Risk Biomarker for Developing Recurrent Adenomas

47. Supplemental Table 7 from Elevated EVL Methylation Level in the Normal Colon Mucosa Is a Potential Risk Biomarker for Developing Recurrent Adenomas

48. Supplemental Figure 3 from Elevated EVL Methylation Level in the Normal Colon Mucosa Is a Potential Risk Biomarker for Developing Recurrent Adenomas

49. Supplemental Table 2 from Elevated EVL Methylation Level in the Normal Colon Mucosa Is a Potential Risk Biomarker for Developing Recurrent Adenomas

50. Supplemental Figure 2 from Elevated EVL Methylation Level in the Normal Colon Mucosa Is a Potential Risk Biomarker for Developing Recurrent Adenomas

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