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3. Adamts12 promotes fibrosis by restructuring extracellular matrix to enable activation of injury-responsive fibroblasts

4. Author Correction: Rps14 haploinsufficiency causes a block in erythroid differentiation mediated by S100A8 and S100A9

7. Collaborative effect of Csnk1a1 haploinsufficiency and mutant p53 in Myc induction can promote leukemic transformation

8. Proinflammatory phenotype of iPS cell-derived JAK2 V617F megakaryocytes induces fibrosis in 3D in vitro bone marrow niche

9. Non-canonical Hedgehog signaling mediates profibrotic hematopoiesis-stroma crosstalk in myeloproliferative neoplasms

10. Expansion-enhanced super-resolution radial fluctuations enable nanoscale molecular profiling of pathology specimens

11. Biological drivers of clinical phenotype in myelofibrosis

13. Sublethal necroptosis signaling promotes inflammation and liver cancer

14. Platelet-instructed SPP1+ macrophages drive myofibroblast activation in fibrosis in a CXCL4-dependent manner

15. Adult human kidney organoids originate from CD24+ cells and represent an advanced model for adult polycystic kidney disease

16. Spatial multi-omic map of human myocardial infarction

18. Genetic barcoding systematically compares genes in del(5q) MDS and reveals a central role for CSNK1A1 in clonal expansion

20. Mapping the cardiac vascular niche in heart failure

23. Heterogeneous bone-marrow stromal progenitors drive myelofibrosis via a druggable alarmin axis

24. Increased CXCL4 expression in hematopoietic cells links inflammation and progression of bone marrow fibrosis in MPN

25. Malignant Transformation Involving CXXC4 Mutations Identified in a Leukemic Progression Model of Severe Congenital Neutropenia

26. Decoding myofibroblast origins in human kidney fibrosis

28. Collaborative effect of Csnk1a1 haploinsufficiency and mutant p53 in Myc induction can promote leukemic transformation

29. Collaborative effect of Csnk1a1 haploinsufficiency and mutant p53 in Myc induction can promote leukemic transformation

32. Rps14, Csnk1a1 and miRNA145/miRNA146a deficiency cooperate in the clinical phenotype and activation of the innate immune system in the 5q- syndrome

34. Gli1+ Mesenchymal Stromal Cells Are a Key Driver of Bone Marrow Fibrosis and an Important Cellular Therapeutic Target

36. Physiologic Expression of Sf3b1K700E Causes Impaired Erythropoiesis, Aberrant Splicing, and Sensitivity to Therapeutic Spliceosome Modulation

37. Core Circadian Clock Genes Regulate Leukemia Stem Cells in AML

39. SRSF2-P95H decreases JAK/STAT signaling in hematopoietic cells and delays myelofibrosis development in mice

40. Data from Type I but Not Type II Calreticulin Mutations Activate the IRE1α/XBP1 Pathway of the Unfolded Protein Response to Drive Myeloproliferative Neoplasms

41. Supplementary Data from Type I but Not Type II Calreticulin Mutations Activate the IRE1α/XBP1 Pathway of the Unfolded Protein Response to Drive Myeloproliferative Neoplasms

42. Data from Mutant Calreticulin Requires Both Its Mutant C-terminus and the Thrombopoietin Receptor for Oncogenic Transformation

43. Supplementary Table S1 from Mutant Calreticulin Requires Both Its Mutant C-terminus and the Thrombopoietin Receptor for Oncogenic Transformation

44. Supplementary Figure S1 from Mutant Calreticulin Requires Both Its Mutant C-terminus and the Thrombopoietin Receptor for Oncogenic Transformation

45. Supplementary Methods, Figure Legends, Table Legends from Mutant Calreticulin Requires Both Its Mutant C-terminus and the Thrombopoietin Receptor for Oncogenic Transformation

47. Telomere dynamics in patients with del (5q) MDS before and under treatment with lenalidomide

50. Dissecting CD8+ T cell pathology of severe SARS-CoV-2 infection by single-cell immunoprofiling

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