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2,443 results on '"Sauerbruch T"'

1. Feasibility and safety of long-term photodynamic therapy (PDT) in the palliative treatment of patients with hilar cholangiocarcinoma

Author

Höblinger A, Gerhardt T, González-Carmona MA, Hüneburg R, Sauerbruch T, and Schmitz V

Subjects
cholangiocarcinoma, palliative care, photodynamic therapy, chemotherapy, Medicine
Abstract

Abstract Background and Aim PDT is an important palliative option for patients with unresectable extrahepatic cholangiocarcinoma (CC). However, the results published to date reported on studies with no more than 6 (mostly up to 4) PDT procedures. Furthermore, the clinical experience of PDT in combination with chemotherapy is limited. The purpose of this retrospective analysis was to evaluate the feasibility and safety of multiple (4 to 14) settings of PDT, combined with biliary drainage, and (in some cases) with chemotherapy. Methods Ten patients with unresectable extrahepatic CC were treated with biliary stenting and at least 4 PDT procedures in our department between 10/2005 and 08/2010. Results Ten patients (male/female = 5/5), mean age 68.8 years (range, 54 - 81 years) who received at least 4 PDT procedures were analyzed. All patients underwent endoscopic biliary drainage. Nine patients received metallic stents and one patient a plastic stent. In 4 patients (40%) bilateral metal stenting (JoStent SelfX®) was performed. The mean number of PDT sessions was 7.9 ± 3.9 (range: 4 - 14). Eight patients had elevated bilirubin levels with a mean bilirubin at admission of 9.9 ± 11.3 mg/dL, which had decreased to an average minimum of 1.2 ± 0.9 mg/dL after 3 months. No severe toxicity was noted. Two patients received concomitant chemotherapy (GEMCIS as 1st line, GEMOX plus cetuximab as 2nd line). The median overall survival has not been reached, whereas the estimated survival of all patients was 47.6 months, 95% CI 25.9 - 48.1. Conclusion Long-term PDT in patients with extrahepatic CC is feasible and effective and is accompanied - at least in this cohort- by a survival time of more than 2 years.

Published
2011
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2. Progression of liver fibrosis in HIV/HCV genotype 1 co-infected patients is related to the T allele of the rsI2979860 polymorphism of the IL28B gene

Author

Lutz P, Wasmuth J-C, Nischalke H-D, Vidovic N, Grünhagei F, Lammert F, Oldenburg J, Rockstroh JK, Sauerbruch T, and Spcngler U

Subjects
IL28B, polymorphism, liver fibrosis, transient elastography, HIV, HCV, Medicine
Abstract

Abstract Objective HIV/HCV co-infection is characterised by accelerated progression of liver disease. Recently, the rsl2979860 C/T polymorphism in the IL28B gene has been linked to progression towards cirrhosis in HCV mono-infected patients and to treatment response of HCV-infection in HIV/HCV co-infected patients. Our aim was to clarify by non-invasive techniques if this polymorphism affects fibrosis progression in HIV/HCV co-infection. Methods In a cross-sectional design, liver stiffness (transient elastography), surrogate markers of liver fibrosis (APRI and FIB-4 scores) and rsl2979860 genotypes were analysed in 84 HCV/H1V co-infected patients. IL28B genotypes were determined by real-time PCR using a light cycler. In 56 HIV/HCV co-infected patients we also studied progression of fibrosis in relation to rsl2979860 C/T genotypes over two years. Results 82% of the patients were on HAART (74% without detectable HI viremia) and 67% were haemophiliacs, respectively. HCV genotype 1 was present in 62%. Cross-sectional median liver stiffness was 7.4 kPa and correlated with APRI and FIB-4 scores (r = 0.6 each, p < 0.001). Frequencies of IL28B genotypes were: CC 50%, CT 43% and TT 7%. In the cross-sectional analysis liver stiffness values were not different between the various IL28B-genotypes. Upon follow-up under HAART carriers of a C allele did not show further progression, while liver stiffness significantly increased in HIV/HCV co-infected patients with the T allele (p = 0.047). Conclusion Although progression of liver fibrosis was low under HAART in our cohort, progression was more pronounced in HIV/HCV genotype 1 co-infected patients with the T allele.

Published
2011
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3. Cancer risk in HIV-infected individuals on HAART is largely attributed to oncogenic infections and state of immunocompetence

Author

Vogel M, Friedrich O, Lüchters G, Holleczek B, Wasmuth JC, Anadol E, Schwarze-Zander C, Nattermann J, Oldenburg J, Sauerbruch T, Rockstroh JK, and Spengler U

Subjects
Lymphoma, Carcinoma, HIV, CD4-T cells, HAART, Medicine
Abstract

Abstract Objectives To estimate the cancer risk of HIV-infected patients in the HAART era with respect to a general reference population and to determine risk factors for malignancy. Methods Long term (1996-2009) cancer incidence of the Bonn single centre HIV cohort was compared to the incidence of the reference population of Saarland using standardized incidence ratios (SIR). Poisson regression analysis was used to identify predictors of cancer risk. Results 1,476 patients entered the cohort, enabling 8,772 person years of observation. 121 tumours in 114 patients, 7 in-situ and 114 invasive cancers, were identified. Malignancies associated with infectious agents such as Kaposi sarcoma (SIRs: male: 5,683; female: 277), non-Hodgkin lymphoma (SIRs male: 35; female: 18), anal cancer (SIRs male: 88; female: 115) as well a cervical carcinoma (SIR female: 4) and Hodgkin's disease (SIR male: 39) and liver cancer (SIR male: 18) were substantially more frequent in HIV-infected patients than in the general population (p < 0.001, each), whereas all other types of cancer were not increased. Poisson regression identified HAART (incidence rate ratio IRR (95% CI): 0.28 (0.19-0.41), p < 0.001), CD4 count (IRR per 100 cells/μl increase: 0.66 (0.57-0.76), p < 0.001), hepatitis B (IRR: 2.15 (1.10-4.20), p = 0.046) and age (IRR per 10 year increase: 1.23 (1.03 - 1.46), p = 0.023) as independent predictors for the occurrence of any type of cancer. Conclusions HAART and preserved CD4 cells preferentially reduce the risk of malignancies associated with oncogenic infections.

Published
2011
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4. Transient elastography discloses identical distribution of liver fibrosis in chronic hepatitis C between HIV-negative and HIV-positive patients on HAART

Author

Grünhage F, Wasmuth J-C, Herkenrath S, Vidovic N, Goldmann G, Rockstroh J, Lammert F, Oldenburg J, Sauerbruch T, and Spengler U

Subjects
Liver fibrosis, HIV, HCV, transelastography, Medicine
Abstract

Abstract Objective Progressive immunodeficiency associated with HIV-infection leads to a progressive course of liver disease in HIV/HCV-co-infected patients. Highly active antiretroviral therapy (HAART) efficiently restores and preserves immune functions and has recently been demonstrated to also result in reduced liver-related mortality in HIV/HCV-co-infected patients. Methods To analyse differences in current liver fibrosis as a possible effect of HAART on fibrosis progression we assessed hepatic fibrosis by transient elastography in a cross-sectional comparison between HCV-mono-infected and HIV/HCV-co-infected patients presenting at our outpatient department in 2007. Results Overall, we did not find any difference in the distribution of liver stiffness between mono- (n = 84) and double-infected (n = 57) patients (14.4 kPa (10.8 - 18.2) versus 12.4 kPa (9.1 - 16.1), mean (95%-CI)). However, in the 8 HIV+ patients with CD4 counts < 200/μl liver stiffness was markedly greater (18.4 kPa (0.8 - 36.0)) than in HIV+ patients with preserved immunity (11.5 kPa (8.4 - 15.0)). Conclusions These findings are in line with other data that show an improved prognosis of chronic hepatitis C in HIV+ patients under effective HAART, and may be a hint that fibrosis progression in well-treated HIV+ patients will no longer be different from that in HCV-mono-infected patients.

Published
2010
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5. Renal impairment after liver transplantation - a pilot trial of calcineurin inhibitor-free vs. calcineurin inhibitor sparing immunosuppression in patients with mildly impaired renal function after liver transplantation

Author

Gerhardt T, Terjung B, Knipper P, Palmedo H, Woitas RP, Kalff J, Sauerbruch T, and Spengler U

Subjects
calcineurin inhibitor, GFR, impaired renal function, liver transplantation, mycophenolate mofetil, Medicine
Abstract

Abstract Objectives Chronic kidney disease is frequent in patients after orthotopic liver transplantation (OLT) and has impact on survival. Patients receiving calcineurin inhibitors (CNI) are at increased risk to develop impaired renal function. Early CNI reduction and concomitant use of mycophenolat mofetil (MMF) has been shown to improve renal function. Methods The aim of this trial was to compare dose-reduced CNI/MMF versus CNI-free MMF/prednisone-based treatment in stable patients after OLT with respect to glomerular filtration rate (GFR). 21 patients [GFR 44.9 ± 9.9 mL/min/1.73 m2 measured by 99m-Tc-DTPA-clearance, serum creatinine (SCr) 1.5 ± 0.42 mg/dL] were randomized either to exchange CNI for 10 mg prednisone (group 1; n = 8) or to receive CNI at 25% of the initial dose (group 2; n = 13) each in combination with 1000 mg MMF b.i.d. Results At month 12 mean SCr (-0.3 ± 0.4 mg/dL, p = 0.031) and GFR improved (8.6 ± 13.1 mL/min/1.73 m2, p = 0.015) in group 2 but remained unchanged in group 1. Main side effects were gastroinstestinal symptoms (14.3%) and infections (4.8%). Two biopsy proven, steroid-responsive rejections occurred. In group 1 mean diastolic blood pressure (BP) increased by 11 ± 22 mmHg (p = 0.03). Conclusions Reduced dose CNI in combination with MMF but not CNI-free-immunosuppression leads to improvement of GFR in patients with moderately elevated SCr levels after OLT. Addition of steroids resulted in increased diastolic blood pressure presumably counterbalancing the benefits of CNI withdrawal on renal function.

Published
2009
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6. Coinheritance of hereditary spherocytosis and reversibility of cirrhosis in a young female patient with hereditary hemochromatosis

Author

Höblinger A, Erdmann C, Strassburg CP, Sauerbruch T, and Lammert F

Subjects
hereditary hemochromatosis, hereditary spherocytosis, iron overload, liver cirrhosis, Medicine
Abstract

Abstract Here we report a 33-years-old woman with hereditary spherocytosis and hemochromatosis due to homozygosity for the C282Y mutation of the HFE gene. The coinheritance of both conditions led to severe iron overload and liver cirrhosis at young age. The patient was treated by repeated phlebotomy, and reversibility of cirrhosis was documented by transient elastography. This report discusses the pathophysiology of iron accumulation in patients with hemolytic anemia combined with HFE C282Y homozygosity. The case indicates that patients with hematological disorders characterized by increased erythropoetic activity should be screened for HFE mutations.

Published
2009
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9. Herausgeber

Author

Niebling, W., primary, Manger, B., additional, Ghadimi, B.M., additional, Bieber, T., additional, Weber, M.M., additional, Schulz, R.-J., additional, Kölbl, Heinz, additional, Serve, H., additional, Nickenig, G., additional, Sauerbruch, T., additional, Benzing, T., additional, Diener, H.C., additional, Rascher, W., additional, Nauck, F., additional, Vogelmeier, C., additional, Falkai, P., additional, Voderholzer, U., additional, Parhofer, K., additional, and Müller, S.C., additional

Published
2019
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10. Autoren

Author

Andrié, R.P., primary, Angenendt, J., additional, Aubin, H., additional, Baldus, S., additional, Balzer, J., additional, Baltzer, J., additional, Behr, J., additional, Benz, Marcus, additional, Berger, M., additional, Blank, N., additional, Blind, E., additional, Buchfelder, M., additional, Burk, A., additional, Detter, C., additional, Diel, R., additional, Diener, H.-C., additional, Domschke, W., additional, Doppler, K., additional, Ellinger, J., additional, Epple, H.-J., additional, Erbel, R., additional, Fichtner-Feigl, S., additional, Fischereder, M., additional, Forsting, M., additional, Friedrich, M., additional, Frommberger, U., additional, Frye, B.C., additional, Goebel, F.-D., additional, Grabitz, K., additional, Groeneveld, A., additional, Gundling, F., additional, Gutt, C.N., additional, Haidl, G., additional, Hartmann, U., additional, Hausteiner-Wiehle, C., additional, Herpertz-Dahlmann, B., additional, Heuß, D., additional, Heußner, P., additional, Hiller, E., additional, Hohenfellner, U., additional, Honegger, J., additional, Hornung, T., additional, Jansen, F., additional, Jelinek, T., additional, Jordan, B., additional, Keck, C., additional, Kelm, M., additional, Kilian, A., additional, Kleindienst, A., additional, Kölbl, H., additional, Kreis, M.E., additional, Kroener-Herwig, B., additional, Külz, A.K., additional, Lammert, F., additional, Lichtenberg, A., additional, Limmroth, V., additional, Lindner, K., additional, Loddenkemper, R., additional, Lorenz, H.-M., additional, Lügering, N., additional, Mallmann, P., additional, Max, R., additional, Merle, U., additional, Mellert, F., additional, Michels, G., additional, Müller, R.-U., additional, Müller, S.C., additional, Müller-Lissner, S., additional, Müller-Quernheim, J., additional, Neuhof, C., additional, Parhofer, K.G., additional, Petri, E., additional, Pfeiffer, T., additional, Pilatz, Adrian, additional, Pizarro, C., additional, Probst, C., additional, Rascher, W., additional, Raue, F., additional, Reindl, C., additional, Riedner, C., additional, Riemann, D., additional, Röckelein, V., additional, Sandmann, W., additional, Sauerbruch, T., additional, Schahab, N., additional, Schaper, A., additional, Schepp, W., additional, Schliep, S., additional, Schnürch, H.-G., additional, Schölmerich, J., additional, Schönermarck, Ulf, additional, Schopohl, J., additional, Schuchert, A., additional, Schulze-Koops, H., additional, Schupp, J., additional, Segerer, S.E., additional, Senninger, N., additional, Seybold, U., additional, Siegmund, B., additional, Skowasch, D., additional, Sommer, C., additional, Stahl, M., additional, Stallmach, A., additional, Sticherling, M., additional, Stremmel, W., additional, Sure, U., additional, Tacke, F., additional, Vloet, T., additional, Voderholzer, U., additional, von Kodolitsch, Y., additional, Wagenlehner, F., additional, Wahlers, T., additional, Weber, L.T., additional, Weidner, W., additional, Weis-Müller, B.T., additional, Werner, N., additional, Wintergerst, U., additional, Wolf, Gunter, additional, Ziegler, D., additional, Zierz, S., additional, and Zimmermann, Th., additional

Published
2019
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13. Autoren

Author

Albers, P., primary, Andrié, R.P., additional, Angenendt, J., additional, Appenrodt, B., additional, Arkudas, A., additional, Aubin, H., additional, Autschbach, R., additional, Baldus, S., additional, Balzer, J., additional, Baltzer, J., additional, Behr, J., additional, Behrens, A., additional, Benz, Marcus, additional, Berger, M., additional, Berr, F., additional, Bischoff, S.C., additional, Böhm, A., additional, Böhm, M., additional, Bokemeyer, D., additional, Borowitzka, F., additional, Brüwer, M., additional, Buchfelder, M., additional, Burk, A., additional, Caca, K., additional, Daum, S., additional, Detter, C., additional, Diener, H.-C., additional, Domschke, W., additional, Doppler, K., additional, Elger, C.E., additional, Ell, C., additional, Ellinger, J., additional, Epple, H.-J., additional, Esser, B., additional, Ewen, Sebastian, additional, Fibbe, C., additional, Fichtner-Feigl, S., additional, Fiegel, P., additional, Fischbach, W., additional, Fischereder, M., additional, Fölsch, U.R., additional, Forsting, M., additional, Frey, N., additional, Freys, S.M., additional, Friedrich, M., additional, Frieling, T., additional, Frommberger, U., additional, Frühmorgen, P., additional, Geidel, S., additional, Germer, C.-T., additional, Goebel, F.-D., additional, Goetzenich, A., additional, Grabitz, K., additional, Greetfeld, M., additional, Groeneveld, A., additional, Groha, P.K., additional, Gundling, F., additional, Gutt, C.N., additional, Haak, T., additional, Hartmann, U., additional, Hauner, H., additional, Hausteiner-Wiehle, C., additional, Henningsen, P., additional, Herpertz-Dahlmann, B., additional, Heuß, D., additional, Heußner, P., additional, Hoffmann, J., additional, Hohenfellner, U., additional, Horch, R.E., additional, Hüll, M., additional, John, S., additional, Jordan, B., additional, Jurowich, C., additional, Käberich, A., additional, Katus, H., additional, Keck, C., additional, Kelm, M., additional, Kiehne, K., additional, Kilian, Adrienne, additional, Kindermann, I., additional, Kleindienst, A., additional, Klingel, K., additional, von Kodolitsch, Y., additional, Kölbl, H., additional, Koletzko, S., additional, Kollmar, O., additional, Konstantinidis, S., additional, Kramer, H.J., additional, Kreis, M.E., additional, Kroener-Herwig, B., additional, Kurschat, C., additional, Lammert, F., additional, Lamprecht, G., additional, Langosch, J.M., additional, Layer, P., additional, Leifeld, L., additional, Lichtenberg, A., additional, Lindner, K., additional, Link, A., additional, Lügering, N., additional, Lusch, Achim, additional, Marth, T., additional, Merle, U., additional, Mellert, F., additional, Meyer-Marcotty, M., additional, Michels, G., additional, Middeke, J.M., additional, Möddel, G., additional, Mössner, J., additional, Müller, A., additional, Müller, J.L., additional, Müller, R.-U., additional, Müller, S.C., additional, Müller-Lissner, S., additional, Müller-Werdan, U., additional, Neubrand, M., additional, Neuhof, C., additional, Parhofer, K.G., additional, Petri, E., additional, Pfeiffer, T., additional, Pilatz, Adrian, additional, Pizarro, C., additional, Pohle, T., additional, Polykandriotis, E., additional, Probst, C., additional, Rascher, W., additional, Rauch, B., additional, Reuss-Borst, M., additional, Riedner, C., additional, Rietschel, E., additional, Röckelein, V., additional, Rüster, Ch., additional, Ruß, M., additional, Sailer, M., additional, Sandmann, W., additional, Sauerbruch, T., additional, Schaaf, B., additional, Schaefer, C.A., additional, Schahab, N., additional, Schaper, A., additional, Schetelig, J., additional, Schmitz, M., additional, Schnürch, H.-G., additional, Schölmerich, J., additional, Schönermarck, Ulf, additional, Schrickel, J.W., additional, Schuchert, A., additional, Schunkert, H., additional, Segerer, S.E., additional, Senninger, N., additional, Seybold, U., additional, Siegmund, B., additional, Skowasch, D., additional, Sommer, C., additional, Spengler, U., additional, Stahl, M., additional, Stallmach, A., additional, Stolpmann, G., additional, Stremmel, W., additional, Sure, U., additional, Tacke, F., additional, te Wildt, B.T., additional, Trinkler, P., additional, Tröger, H., additional, Unnewehr, M., additional, Vloet, T., additional, Voderholzer, U., additional, Vogt, P.M., additional, von Scheidt, W., additional, Wagenlehner, F., additional, Wahlers, T., additional, Weber, L.T., additional, Wehrmann, T., additional, Weis-Müller, B.T., additional, Werdan, K., additional, Wintergerst, U., additional, Wittwer, T., additional, Wolf, Gunter, additional, Wölfling, K., additional, Wolkersdörfer, G., additional, Ziegler, D., additional, and Zierz, S., additional

Published
2018
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14. Herausgeber

Author

Niebling, W., primary, Manger, B., additional, Ghadimi, B.M., additional, Bieber, T., additional, Weber, M.M., additional, Schulz, R.-J., additional, Kölbl, Heinz, additional, Serve, H., additional, Nickenig, G., additional, Sauerbruch, T., additional, Benzing, T., additional, Diener, H.C., additional, Rascher, W., additional, Nauck, F., additional, Vogelmeier, C., additional, Falkai, P., additional, Voderholzer, U., additional, Parhofer, K., additional, and Müller, S.C., additional

Published
2018
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16. Variceal bleeding

Author

Sauerbruch, T., Trebicka, J., Ferkolj, I., editor, Gangl, A., editor, Galle, P. R., editor, and Vucelic, B., editor

Published
2008
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17. Leberzirrhose

Author

Sauerbruch, T., Steinbeck, Gerhard, editor, Paumgartner, Gustav, editor, Brandt, T., editor, Göke, B., editor, Greten, N., editor, Hiddemann, W., editor, Lode, H., editor, Mann, K., editor, Riess, H., editor, Risler, T., editor, Schattenkirchner, M., editor, Seeger, W., editor, and Wehling, M., editor

Published
2005
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18. Benign Liver Tumors

Author

Biecker, E., Fischer, H.P., Strunk, H., Sauerbruch, T., Scheppach, Wolfgang, editor, Bresalier, Robert S., editor, and Tytgat, Guido N. J., editor

Published
2004
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20. Gallenwegstents

Author

Scheurlen, C., Sauerbruch, T., Boese-Landgraf, J., editor, Gallkowski, Uwe, editor, Layer, G., editor, and Schalhorn, A., editor

Published
2003
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21. Dysphagie

Author

Neubrand, M., Schepke, M., Sauerbruch, T., König, Benno, editor, Reinhardt, Dietrich, editor, and Schuster, Hans-Peter, editor

Published
2000
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22. Gastroenterologie

Author

Mössner, J., Riemann, J. F., Schwörer, H., Ramadori, G., Keller, K. M., Arnold, R., Jansen, T., Plewig, G., Dumoulin, F. L., Sauerbruch, T., Hartmann, H., Köppe, Britta, Adler, Guido, Köhne, C.-H., Benter, T., Ritzel, U., Leonhardt, U., and Hiddemann, Wolfgang, editor

Published
2000
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25. Leberkrankheiten

Author

Pape, G. R., Sauerbruch, T., Paumgartner, Gustav, editor, Brandt, Th., editor, Greten, H., editor, Koch, K. M., editor, Lode, H., editor, van de Loo, J., editor, Schattenkirchner, M., editor, Seeger, W., editor, Steinbeck, G., editor, and Ziegler, R., editor

Published
1999
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26. Erkrankungen der Speiseröhre

Author

Sauerbruch, T., Schepke, M., Paumgartner, Gustav, editor, Brandt, Th., editor, Greten, H., editor, Koch, K. M., editor, Lode, H., editor, van de Loo, J., editor, Schattenkirchner, M., editor, Seeger, W., editor, Steinbeck, G., editor, and Ziegler, R., editor

Published
1999
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27. Dysphagie

Author

Neubrand, M., Schepke, M., Sauerbruch, T., Schuster, H.-P., editor, Wilms, K., editor, Lydtin, H., editor, and Lindner, U. K., editor

Published
1999
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38. Clinical Course of acute-on-chronic liver failure syndrome and effects on prognosis

Author

Gustot, Thierry, Fernandez, Javier, Garcia, Elisabet, Morando, Filippo, Caraceni, Paolo, Alessandria, Carlo, Laleman, Wim, Trebicka, Jonel, Elkrief, Laure, Hopf, Corinna, Solís-Munoz, Pablo, Saliba, Faouzi, Zeuzem, Stefan, Albillos, Augustin, Benten, Daniel, Montero-Alvarez, José Luis, Chivas, Maria Teresa, Concepción, Mar, Córdoba, Juan, McCormick, Aiden, Stauber, Rudolf, Vogel, Wolfgang, de Gottardi, Andrea, Welzel, Tania M., Domenicali, Marco, Risso, Alessandro, Wendon, Julia, Deulofeu, Carme, Angeli, Paolo, Durand, François, Pavesi, Marco, Gerbes, Alexander, Jalan, Rajiv, Moreau, Richard, Ginés, Pere, Bernardi, Mauro, Arroyo, Vicente, Aguilar-Melero, P, Bañares, R, Bocci, M, Catalina, M V, Chin, J L, Coenraad, M J, Coilly, A, Dorn, L, Gatta, A, Gerber, L, Grenbæk, H, Graupera, I, Guevara, M, Hausen, A, Karlsen, S, Lohse, A W, Maggioli, C, Markwardt, D, Martinez, J, Marzano, A, de la Mata García, M, Mesonero, F, Mookerjee, R P, Moreno, C, Morrell, B, Mortensen, C, Nevens, F, Peck-Radosavljevic, M, Rizzetto, M, Romano, A, Samuel, D, Sauerbruch, T, Simon-Talero, M, Solà, E, Soriano, G, Sperl, J, Spindelboeck, W, Steib, C, Valla, D, Verbeke, L, Van Vlierberghe, H, Wege, H, Willars, C, Baenas, M Y, and Zaccherini, G.

Published
2015
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44. Mechanisms of extrahepatic vasodilation in portal hypertension

Author

Hennenberg, M., Trebicka, J., Sauerbruch, T., and Heller, J.

Subjects
Portal hypertension -- Development and progression, Portal hypertension -- Forecasts and trends, Protein kinases -- Physiological aspects, Blood vessels -- Dilatation, Blood vessels -- Physiological aspects, Market trend/market analysis, Health
Published
2008

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