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1. Pathway choice in the alternative telomere lengthening in neoplasia is dictated by replication fork processing mediated by EXD2’s nuclease activity

2. AGO2 localizes to cytokinetic protrusions in a p38-dependent manner and is needed for accurate cell division

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3. Correction to: A prototypical non-malignant epithelial model to study genome dynamics and concurrently monitor micro-RNAs and proteins in situ during oncogene-induced senescence

4. A prototypical non-malignant epithelial model to study genome dynamics and concurrently monitor micro-RNAs and proteins in situ during oncogene-induced senescence

5. Abstracts from the 3rd Conference on Aneuploidy and Cancer: Clinical and Experimental Aspects

6. SMUG1 Promotes Telomere Maintenance through Telomerase RNA Processing

7. Alternative Lengthening of Telomeres: Recurrent Cytogenetic Aberrations and Chromosome Stability under Extreme Telomere Dysfunction

8. The Roles of Telomerase in the Generation of Polyploidy during Neoplastic Cell Growth

9. Telomere reprogramming and maintenance in porcine iPS cells.

11. A non-canonical function of zebrafish telomerase reverse transcriptase is required for developmental hematopoiesis.

13. Supplementary Data from Doxorubicin Resistance in a Novel In vitro Model of Human Pleomorphic Liposarcoma Associated with Alternative Lengthening of Telomeres

14. Data from Doxorubicin Resistance in a Novel In vitro Model of Human Pleomorphic Liposarcoma Associated with Alternative Lengthening of Telomeres

15. Supplementary Methods and Legends for Movies and Figures from Distinct Roles of BARD1 Isoforms in Mitosis: Full-Length BARD1 Mediates Aurora B Degradation, Cancer-Associated BARD1β Scaffolds Aurora B and BRCA2

16. Supplementary Figures 1-4 from Pericentromeric Instability and Spontaneous Emergence of Human Neoacrocentric and Minute Chromosomes in the Alternative Pathway of Telomere Lengthening

17. Supplementary Methods, Legends and Table 1 from Pericentromeric Instability and Spontaneous Emergence of Human Neoacrocentric and Minute Chromosomes in the Alternative Pathway of Telomere Lengthening

18. Supplementary Movie 2 from Distinct Roles of BARD1 Isoforms in Mitosis: Full-Length BARD1 Mediates Aurora B Degradation, Cancer-Associated BARD1β Scaffolds Aurora B and BRCA2

19. Supplementary Movie 1 from Distinct Roles of BARD1 Isoforms in Mitosis: Full-Length BARD1 Mediates Aurora B Degradation, Cancer-Associated BARD1β Scaffolds Aurora B and BRCA2

20. Supplementary Table 1 from Distinct Roles of BARD1 Isoforms in Mitosis: Full-Length BARD1 Mediates Aurora B Degradation, Cancer-Associated BARD1β Scaffolds Aurora B and BRCA2

21. Supplementary Figures 1-8 from Distinct Roles of BARD1 Isoforms in Mitosis: Full-Length BARD1 Mediates Aurora B Degradation, Cancer-Associated BARD1β Scaffolds Aurora B and BRCA2

22. Data from Distinct Roles of BARD1 Isoforms in Mitosis: Full-Length BARD1 Mediates Aurora B Degradation, Cancer-Associated BARD1β Scaffolds Aurora B and BRCA2

23. Supplementary Movie 3 from Distinct Roles of BARD1 Isoforms in Mitosis: Full-Length BARD1 Mediates Aurora B Degradation, Cancer-Associated BARD1β Scaffolds Aurora B and BRCA2

24. Supplementary Movie 4 from Distinct Roles of BARD1 Isoforms in Mitosis: Full-Length BARD1 Mediates Aurora B Degradation, Cancer-Associated BARD1β Scaffolds Aurora B and BRCA2

25. Supplementary Figure 6 from Pericentromeric Instability and Spontaneous Emergence of Human Neoacrocentric and Minute Chromosomes in the Alternative Pathway of Telomere Lengthening

26. Supplementary Figure 7 from Pericentromeric Instability and Spontaneous Emergence of Human Neoacrocentric and Minute Chromosomes in the Alternative Pathway of Telomere Lengthening

27. Supplementary Figure 5 from Pericentromeric Instability and Spontaneous Emergence of Human Neoacrocentric and Minute Chromosomes in the Alternative Pathway of Telomere Lengthening

28. Selective pericentromeric heterochromatin dismantling caused by TP53 activation during senescence

29. HN1 interacts with gamma-tubulin to regulate centrosomes in advanced prostate cancer cells

30. Detection of a novel unbalanced X;21 translocation in a girl with Turner syndrome phenotype

31. Karyotypic Flexibility of the Complex Cancer Genome and the Role of Polyploidization in Maintenance of Structural Integrity of Cancer Chromosomes

32. The effect of intrauterine growth on leukocyte telomere length at birth

34. Chromosome extremities under the microscopy lens: molecular cytogenetics in telomere research

35. Childhood obesity and leucocyte telomere length

36. Alternative lengthening of human telomeres is a conservative <scp>DNA</scp> replication process with features of break‐induced replication

37. Unbalanced X;9 translocation in an infertile male with de novo duplication Xp22.31p22.33

38. EXD2 Protects Stressed Replication Forks and Is Required for Cell Viability in the Absence of BRCA1/2

39. Nuclear-Receptor-Mediated Telomere Insertion Leads to Genome Instability in ALT Cancers

40. Rif1 Maintains Telomere Length Homeostasis of ESCs by Mediating Heterochromatin Silencing

41. The Roles of Telomerase in the Generation of Polyploidy during Neoplastic Cell Growth

42. Chronic p53-independent p21 expression causes genomic instability by deregulating replication licensing

44. The helicase domain and C-terminus of human RecQL4 facilitate replication elongation on DNA templates damaged by ionizing radiation

45. Molecular insights into the heterogeneity of telomere reprogramming in induced pluripotent stem cells

46. Human UPF1 interacts with TPP1 and telomerase and sustains telomere leading-strand replication

47. Distinct Roles of BARD1 Isoforms in Mitosis: Full-Length BARD1 Mediates Aurora B Degradation, Cancer-Associated BARD1β Scaffolds Aurora B and BRCA2

48. The influence of 5-year therapy with tibolone on the lipid profile in postmenopausal women with mild hypercholesterolemia

49. Structure and Functions of Telomeres in Organismal Homeostasis and Disease

50. Inherited duplication Xq27-qter at Xp22.3 in severely affected males: Molecular cytogenetic evaluation and clinical description in three unrelated families