37 results on '"San Martin, Alejandra"'
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2. HERPUD1 governs tumor cell mitochondrial function via inositol 1,4,5-trisphosphate receptor-mediated calcium signaling
3. The Role of Fatty Acid Synthase in the Vascular Smooth Muscle Cell to Foam Cell Transition
4. The cofilin phosphatase slingshot homolog 1 restrains angiotensin II-induced vascular hypertrophy and fibrosis in vivo
5. Nox1-based NADPH oxidase regulates the Par protein complex activity to control cell polarization
6. Abstract P3029: The Role Of Fatty Acid Synthase In The Modulation Of Vascular Smooth Muscle Cells Into A Foam Cell Phenotype
7. Poldip2 is an oxygen-sensitive protein that controls PDH and αKGDH lipoylation and activation to support metabolic adaptation in hypoxia and cancer
8. Abstract 407: The Role Of Peroxisomes And Bile Acid-initiated Signaling In Vascular Smooth Muscle Cell Phenotype
9. Mitochondrial Protein Poldip2 (Polymerase Delta Interacting Protein 2) Controls Vascular Smooth Muscle Differentiated Phenotype by O-Linked GlcNAc (N-Acetylglucosamine) Transferase–Dependent Inhibition of a Ubiquitin Proteasome System
10. Metabolic regulation of the proteasome under hypoxia by Poldip2 controls fibrotic signaling in vascular smooth muscle cells
11. The Role of Fatty Acid Synthase in Atherosclerosis
12. The Role of Bile Acid Signaling in Differentiation of Vascular Smooth Muscle Cells
13. Regulation of total LC3 levels by angiotensin II in vascular smooth muscle cells
14. The mitochondrial protease ClpXP regulates the phenotype of VSMC by a SIRT1-dependent mechanism
15. PGC-1alpha deficiency attenuates vascular smooth muscle atherogenic phenotypic plasticity
16. HERPUD1 protects against oxidative stress-induced apoptosis through downregulation of the inositol 1,4,5-trisphosphate receptor
17. Metabolic adaptation in hypoxia and cancer
18. Reactive oxygen species-selective regulation of aortic inflammatory gene expression in Type 2 diabetes
19. Nox1-based NADPH oxidase-derived superoxide is required for VSMC activation by advanced glycation end-products
20. Quiescin/sulfhydryl oxidase 1b (QSOX1b) induces migration and proliferation of vascular smooth muscle cells by distinct redox pathways
21. Angiotensin II-Regulated Autophagy Is Required for Vascular Smooth Muscle Cell Hypertrophy
22. PPARγ Regulates Mitochondrial Structure and Function and Human Pulmonary Artery Smooth Muscle Cell Proliferation
23. Color and Antioxidant Capacity Preservation of Opuntia spp. Juices by Spray-drying Microencapsulation.
24. Hic-5 Mediates TGFβ–Induced Adhesion in Vascular Smooth Muscle Cells by a Nox4-Dependent Mechanism
25. NADPH Oxidases: Progress and Opportunities
26. Role of βPIX in PDGF-Induced Lamellipodia Dynamics in VSMC
27. TGF-β Mediates Focal Adhesion Maturation by a Smad/Nox4-Dependent Mechanism that Involves Regulation of Hsp27 and Hic5
28. Vascular smooth muscle insulin resistance, but not hypertrophic signaling, is independent of angiotensin II-induced IRS-1 phosphorylation by JNK
29. PGC-1α Serine 570 Phosphorylation and GCN5-mediated Acetylation by Angiotensin II Drive Catalase Down-regulation and Vascular Hypertrophy
30. The role of Nox-mediated oxidation in the regulation of cytoskeletal dynamics
31. Abstract 15221: Digoxin Reverses Angiogenic Switch During LVAD Support by Inhibiting the Hypoxia Triggered HIF1-? Signaling Cascade: In vivoand in vitroEvidence
32. PGC-1α Serine 570 Phosphorylation and GCN5-mediated Acetylation by Angiotensin II Drive Catalase Down-regulation and Vascular Hypertrophy.
33. Early Endosomal Antigen 1 (EEA1) Is an Obligate Scaffold for Angiotensin II-induced; PKC-α-dependent Akt Activation in Endosomes.
34. Vascular smooth muscle insulin resistance, but not hypertrophic signaling, is independent of angiotensin II-induced IRS-1 phosphorylation by JNK.
35. PGC-1 alpha serine 570 phosphorylation and GCN5-mediated acetylation by angiotensin II drive catalase down-regulation and vascular hypertrophy.
36. Mechanisms of vascular smooth muscle NADPH oxidase 1 (Nox1) contribution to injury-induced neointimal formation.
37. Nox1 overexpression potentiates angiotensin II-induced hypertension and vascular smooth muscle hypertrophy in transgenic mice.
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