24 results on '"Sadleir, Katherine R."'
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2. Aberrant glial activation and synaptic defects in CaMKIIα-iCre and nestin-Cre transgenic mouse models
3. The innate immunity protein IFITM3 modulates γ-secretase in Alzheimer’s disease
4. Connections between ApoE, sleep, and Aβ and tau pathologies in Alzheimer’s disease
5. Aberrant glial activation and synaptic defects in CaMKIIα-iCre and nestin-Cre transgenic mouse models
6. Death induced by survival gene elimination (DISE) contributes to neurotoxicity in Alzheimer’s disease
7. Modeling genetic diversity in Alzheimer’s disease
8. Presynaptic dystrophic neurites surrounding amyloid plaques are sites of microtubule disruption, BACE1 elevation, and increased Aβ generation in Alzheimer’s disease
9. A conserved annexin A6–mediated membrane repair mechanism in muscle, heart, and nerve
10. Oral nimodipine treatment has no effect on amyloid pathology or neuritic dystrophy in the 5XFAD mouse model of amyloidosis
11. The Alzheimer’s β-secretase BACE1 localizes to normal presynaptic terminals and to dystrophic presynaptic terminals surrounding amyloid plaques
12. Pregabalin Treatment does not Affect Amyloid Pathology in 5XFAD Mice
13. Elevated Aβ42 in Aged, Non-demented Individuals with Cerebral Atherosclerosis
14. RPS23RG1 May Prevent Ubiquitin-Proteosomal Degradation of Postsynaptic Densities-93 and -95 to Protect Synaptic Function: Implications for Alzheimer’s Disease
15. Erratum to: The Alzheimer’s β-secretase BACE1 localizes to normal presynaptic terminals and to dystrophic presynaptic terminals surrounding amyloid plaques
16. ER stress is not elevated in the 5XFAD mouse model of Alzheimer's disease
17. P1‐163: MOLECULAR MECHANISMS OF THE ALZHEIMER'S RISK GENE UNC5C IN NEURONAL DEATH
18. MOLECULAR MECHANISMS OF THE ALZHEIMER’S RISK GENE UNC5C IN NEURONAL DEATH
19. Dimebon alters hippocampal amyloid pathology in 3xTg-AD mice
20. Aβ reduction in BACE1 heterozygous null 5XFAD mice is associated with transgenic APP level
21. Genetic Inhibition of Phosphorylation of the Translation Initiation Factor eIF2α Does Not Block Aβ-Dependent Elevation of BACE1 and APP Levels or Reduce Amyloid Pathology in a Mouse Model of Alzheimer’s Disease
22. Cdk5 Protein Inhibition and Aβ42 Increase BACE1 Protein Level in Primary Neurons by a Post-transcriptional Mechanism
23. Phosphorylation of the Translation Initiation Factor eIF2α Increases BACE1 Levels and Promotes Amyloidogenesis
24. Dimebon alters hippocampal amyloid pathology in 3xTg-AD mice.
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