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415 results on '"Rucci, Nadia"'

15. Supplementary Table 1 from A Six-Gene Signature Predicting Breast Cancer Lung Metastasis

Author

Landemaine, Thomas, primary, Jackson, Amanda, primary, Bellahcène, Akeila, primary, Rucci, Nadia, primary, Sin, Soraya, primary, Abad, Berta Martin, primary, Sierra, Angels, primary, Boudinet, Alain, primary, Guinebretière, Jean-Marc, primary, Ricevuto, Enrico, primary, Noguès, Catherine, primary, Briffod, Marianne, primary, Bièche, Ivan, primary, Cherel, Pascal, primary, Garcia, Teresa, primary, Castronovo, Vincent, primary, Teti, Anna, primary, Lidereau, Rosette, primary, and Driouch, Keltouma, primary

Published
2023
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16. Data from A Six-Gene Signature Predicting Breast Cancer Lung Metastasis

Author

Landemaine, Thomas, primary, Jackson, Amanda, primary, Bellahcène, Akeila, primary, Rucci, Nadia, primary, Sin, Soraya, primary, Abad, Berta Martin, primary, Sierra, Angels, primary, Boudinet, Alain, primary, Guinebretière, Jean-Marc, primary, Ricevuto, Enrico, primary, Noguès, Catherine, primary, Briffod, Marianne, primary, Bièche, Ivan, primary, Cherel, Pascal, primary, Garcia, Teresa, primary, Castronovo, Vincent, primary, Teti, Anna, primary, Lidereau, Rosette, primary, and Driouch, Keltouma, primary

Published
2023
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29. HistoEnder : A 3D printer-based histological slide autostainer that retains 3D printer functions

Author

Ponzetti, Marco, Chinna Rao Devarapu, Ganga, Rucci, Nadia, Carlone, Armando, Saggiomo, Vittorio, Ponzetti, Marco, Chinna Rao Devarapu, Ganga, Rucci, Nadia, Carlone, Armando, and Saggiomo, Vittorio

Abstract

Automated microscope slide stainers are usually very expensive and unless the laboratory performs heavy histological work it is difficult to justify buying a 2000-10000€ machine. As a result, histology and pathology labs around the world lose thousands of working hours for following procedures that could be easily automated. Herein, we propose a simple modification of an open-source 3D printer, the Creality Ender-3, into an automated microscope slide autostainer, the HistoEnder. The HistoEnder is cheap (less than 200€), modular, and easy to set up, with only two 3D-printed parts needed. Additionally, the 3D printer retains its full functionality, and it can be reverted back into 3D printing in less than 1 min. The g-code associated with the procedure is extremely simple, and can be written by anyone. The HistoEnder can also be used in chemistry and material science laboratories for automating surface modifications and dip coating.

Published
2022

39. The glycosaminoglycan-binding domain of PRELP acts as a cell type-specific NF-[kappa]B inhibitor that impairs osteoclastogenesis

Author

Rucci, Nadia, Rufo, Anna, Alamanou, Marina, Capulli, Mattia, Del Fattore, Andrea, Ahrman, Emma, Capece, Daria, Iansante, Valeria, Zazzeroni, Francesca, Alesse, Edoardo, Heinegard, Dick, and Teti, Anna

Subjects
Glycosaminoglycans -- Physiological aspects, Gene expression -- Physiological aspects, Biological sciences
Abstract

Proline/arginine-rich end leucine-rich repeat protein (PRELP) is a glycosaminoglycan (GAG)- and collagen-binding anchor protein highly expressed in cartilage, basement membranes, and developing bone. We observed that PRELP inhibited in vitro and in vivo mouse osteoclastogenesis through its GAG-binding domain ([sup.hbd]PRELP), involving (a) cell internalization through a chondroitin sulfate- and annexin Il-dependent mechanism, (b) nuclear translocation, (c) interaction with p65 nuclear factor [kappa]B (NF-[kappa]B) and inhibition of its DNA binding, and (d) impairment of NF-[kappa]B transcriptional activity and reduction of osteoclast-specific gene expression, [sup.hbd]PRELP does not disrupt the mitogen-activated protein kinase signaling nor does it impair cell survival, [sup.hbd]PRELP activity is cell type specific, given that it is internalized by the RAW264.7 osteoclast-like cell line but fails to affect calvarial osteoblasts, bone marrow macrophages, and epithelial cell lines. In vivo, hbdpRELP reduces osteoclast number and activity in ovariectomized mice, underlying its physiological and/or pathological importance in skeletal remodeling. doi/10.1083/jcb.200906014

Published
2009

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