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427 results on '"Ross, Fiona M."'

1. The radial distribution of supernovae compared to star formation tracers

Author

Audcent-Ross, Fiona M., Meurer, Gerhardt R., Audcent, James R., Ryder, Stuart D., Wong, O. Ivy, Phan, J., Williamson, A., and Kim, J. H.

Subjects
Astrophysics - Astrophysics of Galaxies
Abstract

Given the limited availability of direct evidence (pre-explosion observations) for supernova (SN) progenitors, the location of supernovae (SNe) within their host galaxies can be used to set limits on one of their most fundamental characteristics, their initial progenitor mass. We present our constraints on SN progenitors derived by comparing the radial distributions of 80 SNe in the SINGG and SUNGG surveys to the R-band, Halpha, and UV light distributions of the 55 host galaxies. The strong correlation of Type Ia SNe with R-band light is consistent with models containing only low mass progenitors, reflecting earlier findings. When we limit the analysis of Type II SNe to apertures containing 90 per cent of the total flux, the radial distribution of these SNe best traces far ultraviolet (FUV) emission, consistent with recent direct detections indicating Type II SNe have moderately massive red supergiant progenitors. Stripped Envelope (SE) SNe have the strongest correlation with Halpha fluxes, indicative of very massive progenitors (M* > 20 M_solar). This result contradicts a small, but growing, number of direct detections of SE SN progenitors indicating they are moderately massive binary systems. Our result is consistent, however, with a recent population analysis suggesting binary SE SN progenitor masses are regularly underestimated. SE SNe are centralised with respect to Type II SNe and there are no SE SNe recorded beyond half the maximum disc radius in the optical and one third the disc radius in the ultraviolet. The absence of SE SNe beyond these distances is consistent with reduced massive star formation efficiencies in the outskirts of the host galaxies., Comment: 17 pages

Published
2019
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2. Near-identical star formation rate densities from H$\alpha$ and FUV at redshift zero

Author

Audcent-Ross, Fiona M., Meurer, Gerhardt R., Wong, O. I., Zheng, Z., Hanish, D., Zwaan, M. A., Bland-Hawthorn, J., Elagali, A., Meyer, M., Putman, M. E., Ryan-Webber, E. V., Sweet, S. M., Thilker, D. A., Seibert, M., Allen, R., Dopita, M. A., Doyle-Pegg, M. T., Drinkwater, M., Ferguson, H. C., Freeman, K. C., Heckman, T. M., Kennicutt Jr, R. C., Kilborn, V. A., Kim, J. H., Knezek, P. M., Koribalski, B., Smith, R. C., Staveley-Smith, L., Webster, R. L., and Werk, J. K.

Subjects
Astrophysics - Astrophysics of Galaxies
Abstract

For the first time both H$\alpha$ and far-ultraviolet (FUV) observations from an HI-selected sample are used to determine the dust-corrected star formation rate density (SFRD: $\dot{\rho}$) in the local Universe. Applying the two star formation rate indicators on 294 local galaxies we determine log($\dot{\rho}$$ _{H\alpha}) = -1.68~^{+0.13}_{-0.05}$ [M$_{\odot} $ yr$^{-1} $ Mpc$^{-3}]$ and log($\dot{\rho}_{FUV}$) $ = -1.71~^{+0.12}_{-0.13}$ [M$_\odot $ yr$^{-1} $ Mpc$^{-3}]$. These values are derived from scaling H$\alpha$ and FUV observations to the HI mass function. Galaxies were selected to uniformly sample the full HI mass (M$_{HI}$) range of the HI Parkes All-Sky Survey (M$_{HI} \sim10^{7}$ to $\sim10^{10.7}$ M$_{\odot}$). The approach leads to relatively larger sampling of dwarf galaxies compared to optically-selected surveys. The low HI mass, low luminosity and low surface brightness galaxy populations have, on average, lower H$\alpha$/FUV flux ratios than the remaining galaxy populations, consistent with the earlier results of Meurer. The near-identical H$\alpha$- and FUV-derived SFRD values arise with the low H$\alpha$/FUV flux ratios of some galaxies being offset by enhanced H$\alpha$ from the brightest and high mass galaxy populations. Our findings confirm the necessity to fully sample the HI mass range for a complete census of local star formation to include lower stellar mass galaxies which dominate the local Universe., Comment: 17 pages, 7 figures

Published
2018
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3. Cosmic clocks: A Tight Radius - Velocity Relationship for HI-Selected Galaxies

Author

Meurer, Gerhardt R., Obreschkow, Danail, Wong, O. Ivy, Zheng, Zheng, Audcent-Ross, Fiona M., and Hanish, D. J.

Subjects
Astrophysics - Astrophysics of Galaxies, Astrophysics - Cosmology and Nongalactic Astrophysics
Abstract

HI-Selected galaxies obey a linear relationship between their maximum detected radius Rmax and rotational velocity. This result covers measurements in the optical, ultraviolet, and HI emission in galaxies spanning a factor of 30 in size and velocity, from small dwarf irregulars to the largest spirals. Hence, galaxies behave as clocks, rotating once a Gyr at the very outskirts of their discs. Observations of a large optically-selected sample are consistent, implying this relationship is generic to disc galaxies in the low redshift Universe. A linear RV relationship is expected from simple models of galaxy formation and evolution. The total mass within Rmax has collapsed by a factor of 37 compared to the present mean density of the Universe. Adopting standard assumptions we find a mean halo spin parameter lambda in the range 0.020 to 0.035. The dispersion in lambda, 0.16 dex, is smaller than expected from simulations. This may be due to the biases in our selection of disc galaxies rather than all halos. The estimated mass densities of stars and atomic gas at Rmax are similar (~0.5 Msun/pc^2) indicating outer discs are highly evolved. The gas consumption and stellar population build time-scales are hundreds of Gyr, hence star formation is not driving the current evolution of outer discs. The estimated ratio between Rmax and disc scale length is consistent with long-standing predictions from monolithic collapse models. Hence, it remains unclear whether disc extent results from continual accretion, a rapid initial collapse, secular evolution or a combination thereof., Comment: 14 pages, 7 figures, 3 in colour. Published in MNRAS. This v2 corrects wrong journal in the references section (all instances of "Astrophysics and Space Sciences" should have been ApJ). The Posti+2017 has also been updated. An erratum has been submitted to MNRAS

Published
2018
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4. Author Correction: Identification of multiple risk loci and regulatory mechanisms influencing susceptibility to multiple myeloma

Author

Went, Molly, Sud, Amit, Försti, Asta, Halvarsson, Britt-Marie, Weinhold, Niels, Kimber, Scott, van Duin, Mark, Thorleifsson, Gudmar, Holroyd, Amy, Johnson, David C., Li, Ni, Orlando, Giulia, Law, Philip J., Ali, Mina, Chen, Bowang, Mitchell, Jonathan S., Gudbjartsson, Daniel F., Kuiper, Rowan, Stephens, Owen W., Bertsch, Uta, Broderick, Peter, Campo, Chiara, Bandapalli, Obul R, Einsele, Hermann, Gregory, Walter A., Gullberg, Urban, Hillengass, Jens, Hoffmann, Per, Jackson, Graham H., Jöckel, Karl-Heinz, Johnsson, Ellinor, Kristinsson, Sigurður Y., Mellqvist, Ulf-Henrik, Nahi, Hareth, Easton, Douglas, Pharoah, Paul, Dunning, Alison, Peto, Julian, Canzian, Federico, Swerdlow, Anthony, Eeles, Rosalind A., Kote-Jarai, Zsofia, Muir, Kenneth, Pashayan, Nora, The PRACTICAL consortium, Nickel, Jolanta, Nöthen, Markus M., Rafnar, Thorunn, Ross, Fiona M., da Silva Filho, Miguel Inacio, Thomsen, Hauke, Turesson, Ingemar, Vangsted, Annette, Andersen, Niels Frost, Waage, Anders, Walker, Brian A., Wihlborg, Anna-Karin, Broyl, Annemiek, Davies, Faith E., Thorsteinsdottir, Unnur, Langer, Christian, Hansson, Markus, Goldschmidt, Hartmut, Kaiser, Martin, Sonneveld, Pieter, Stefansson, Kari, Morgan, Gareth J., Hemminki, Kari, Nilsson, Björn, and Houlston, Richard S.

Published
2019
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9. Data from Homozygous Deletion Mapping in Myeloma Samples Identifies Genes and an Expression Signature Relevant to Pathogenesis and Outcome

Author

Dickens, Nicholas J., primary, Walker, Brian A., primary, Leone, Paola E., primary, Johnson, David C., primary, Brito, José L., primary, Zeisig, Athanasia, primary, Jenner, Matthew W., primary, Boyd, Kevin D., primary, Gonzalez, David, primary, Gregory, Walter M., primary, Ross, Fiona M., primary, Davies, Faith E., primary, and Morgan, Gareth J., primary

Published
2023
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11. Supplementary Table 1 from Mapping of Chromosome 1p Deletions in Myeloma Identifies FAM46C at 1p12 and CDKN2C at 1p32.3 as Being Genes in Regions Associated with Adverse Survival

Author

Boyd, Kevin D., primary, Ross, Fiona M., primary, Walker, Brian A., primary, Wardell, Christopher P., primary, Tapper, William J., primary, Chiecchio, Laura, primary, Dagrada, GianPaolo, primary, Konn, Zoe J., primary, Gregory, Walter M., primary, Jackson, Graham H., primary, Child, J. Anthony, primary, Davies, Faith E., primary, and Morgan, Gareth J., primary

Published
2023
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14. Supplementary Methods, References, Legends for Table 1 and Figure 1 from Gender Disparities in the Tumor Genetics and Clinical Outcome of Multiple Myeloma

Author

Boyd, Kevin D., primary, Ross, Fiona M., primary, Chiecchio, Laura, primary, Dagrada, GianPaolo, primary, Konn, Zoe J., primary, Tapper, William J., primary, Walker, Brian A., primary, Wardell, Christopher P., primary, Gregory, Walter M., primary, Szubert, Alex J., primary, Davies, Faith E., primary, and Morgan, Gareth J., primary

Published
2023
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15. Adobe PDF - Supplementary_Data.pdf from Homozygous Deletion Mapping in Myeloma Samples Identifies Genes and an Expression Signature Relevant to Pathogenesis and Outcome

Author

Dickens, Nicholas J., primary, Walker, Brian A., primary, Leone, Paola E., primary, Johnson, David C., primary, Brito, José L., primary, Zeisig, Athanasia, primary, Jenner, Matthew W., primary, Boyd, Kevin D., primary, Gonzalez, David, primary, Gregory, Walter M., primary, Ross, Fiona M., primary, Davies, Faith E., primary, and Morgan, Gareth J., primary

Published
2023
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16. Supplementary Table 3 from Mapping of Chromosome 1p Deletions in Myeloma Identifies FAM46C at 1p12 and CDKN2C at 1p32.3 as Being Genes in Regions Associated with Adverse Survival

Author

Boyd, Kevin D., primary, Ross, Fiona M., primary, Walker, Brian A., primary, Wardell, Christopher P., primary, Tapper, William J., primary, Chiecchio, Laura, primary, Dagrada, GianPaolo, primary, Konn, Zoe J., primary, Gregory, Walter M., primary, Jackson, Graham H., primary, Child, J. Anthony, primary, Davies, Faith E., primary, and Morgan, Gareth J., primary

Published
2023
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17. Supplementary Table 2 from Mapping of Chromosome 1p Deletions in Myeloma Identifies FAM46C at 1p12 and CDKN2C at 1p32.3 as Being Genes in Regions Associated with Adverse Survival

Author

Boyd, Kevin D., primary, Ross, Fiona M., primary, Walker, Brian A., primary, Wardell, Christopher P., primary, Tapper, William J., primary, Chiecchio, Laura, primary, Dagrada, GianPaolo, primary, Konn, Zoe J., primary, Gregory, Walter M., primary, Jackson, Graham H., primary, Child, J. Anthony, primary, Davies, Faith E., primary, and Morgan, Gareth J., primary

Published
2023
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18. The modulation of epileptiform activity in rat hippocampal slices by adenine nucleotides

Author

Ross, Fiona M.

Subjects
610, Neurotransmitters, Epilepsy
Abstract

In this study the most consistent method of inducing epileptiform activity in hippocampal slices involved recording from the pyramidal cell layer of the CA3 region whilst bathing the slice in a medium containing no added magnesium and 4-aminopyridine (4AP) (50?M). The bursts produced were interictal in nature and inhibited by kynurenate (ImM) or 2-amino-5-phosphonopentanoic acid (20 and 40muM) suggesting that both NMDA and non-NMDA receptors were involved. The effect of ATP and other adenine nucleotides on the rate of spontaneous epileptiform activity was investigated. ATP and adenosine were equipotent at decreasing discharge rate at concentrations above 10muM. The depression produced by ATP was characterised by being inhibited by the A1 receptor antagonist 8-cyclopentyl, 1,3-dimethylxanthine (CPT) but resistant to adenosine deaminase (at a concentration which annulled the effect of adenosine). P2 receptor antagonists (pyridoxalphosphate-6-azophenyl-2',4'-disulphonic acid (PPADS) and suramin) were also unable to block ATP depression of bursting. AMP depressed epileptiform activity similarly to ATP. Its effect was inhibited by CPT and 5'-adenylic acid deaminase (AMPase). AMPase also blocked the depression in discharge rate elicited by ATP. 5'-nucleotidase is involved in the metabolism of AMP to adenosine. The combination of 5'-nucleotidase and adenosine deaminase with AMP or ATP resulted in no significant change in the frequency of activity. These later results, therefore, suggest that ATP acts through metabolism to AMP. The antagonism by CPT supports the involvement of A1 receptors. The ATP analogues alpha,beta-methyleneATP and 2-methylthioATP were also tested as well as uridine triphosphate. Only alpha,beta-methyleneATP (10muM) altered the discharge rate producing an increase in the frequency of spontaneous activity which was inhibited by suramin and PPADS. These results are indicative of an excitatory P2X receptor in the hippocampal CA3 region. Several have been cloned and found expressed in the hippocampus but none with the characteristics displayed in this study. BzATP, an agonist at the P2Z receptor, decreased the discharge rate during a 10 minute perfusion. This depression was not reversed upon washing but continued to progress. The combination of BzATP and CPT produced an increase in discharge rate which was smaller than that caused by CPT alone. This suggests that A1 receptors may not be involved in producing the effect of BzATP. The alpha, omega-adenine dinucleotides Ap4A and Ap5A produced a concentration-dependent depression in discharge rate. Adenosine deaminase inhibited the effect of 1muM but not the early stages of the depression produced by 10?M Ap4A and Ap5A. CPT but not AMPase antagonised the depression in rate produced by Ap4A. This suggests that in the CA3 region of the hippocampus AP4A and Ap5A act partly by stimulating xanthine sensitive receptors directly and partly through the formation of the metabolite, adenosine. AMPase initially elevated the discharge rate to a small extent but a subsequent fall in rate occurred during the wash period which continued until a plateau was reached at which the rate remained for up to 90 minutes. IMP was ineffective. Dialysis of AMPase neither affected the enzymatic activity of AMPase nor the following depression. Boiling the enzyme rendered it inactive in that it could no longer prevent the effect of AMP. The sustained depression in activity produced by AMPase was also inhibited by denaturisation. It was proposed, therefore, that AMPase induces a form of long term depression (LTD). The induction of this LTD, as measured by an effect on discharge rate, was not altered by antagonists of A1, opioid or GABAA receptors nor inhibitors of cyclo-oxygenase, nitric oxide synthase or protein kinase.

Published
1997

19. Identification of multiple risk loci and regulatory mechanisms influencing susceptibility to multiple myeloma

Author

Went, Molly, Sud, Amit, Försti, Asta, Halvarsson, Britt-Marie, Weinhold, Niels, Kimber, Scott, van Duin, Mark, Thorleifsson, Gudmar, Holroyd, Amy, Johnson, David C., Li, Ni, Orlando, Giulia, Law, Philip J., Ali, Mina, Chen, Bowang, Mitchell, Jonathan S., Gudbjartsson, Daniel F., Kuiper, Rowan, Stephens, Owen W., Bertsch, Uta, Broderick, Peter, Campo, Chiara, Bandapalli, Obul R, Einsele, Hermann, Gregory, Walter A., Gullberg, Urban, Hillengass, Jens, Hoffmann, Per, Jackson, Graham H., Jöckel, Karl-Heinz, Johnsson, Ellinor, Kristinsson, Sigurður Y., Mellqvist, Ulf-Henrik, Nahi, Hareth, Easton, Douglas, Pharoah, Paul, Dunning, Alison, Peto, Julian, Canzian, Federico, Swerdlow, Anthony, Eeles, Rosalind A., Kote-Jarai, ZSofia, Muir, Kenneth, Pashayan, Nora, Nickel, Jolanta, Nöthen, Markus M., Rafnar, Thorunn, Ross, Fiona M., da Silva Filho, Miguel Inacio, Thomsen, Hauke, Turesson, Ingemar, Vangsted, Annette, Andersen, Niels Frost, Waage, Anders, Walker, Brian A., Wihlborg, Anna-Karin, Broyl, Annemiek, Davies, Faith E., Thorsteinsdottir, Unnur, Langer, Christian, Hansson, Markus, Goldschmidt, Hartmut, Kaiser, Martin, Sonneveld, Pieter, Stefansson, Kari, Morgan, Gareth J., Hemminki, Kari, Nilsson, Björn, Houlston, Richard S., and The PRACTICAL consortium

Published
2018
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29. Effects of zoledronic acid versus clodronic acid on skeletal morbidity in patients with newly diagnosed multiple myeloma (MRC Myeloma IX): secondary outcomes from a randomised controlled trial

Author

Morgan, Gareth J, Child, J Anthony, Gregory, Walter M, Szubert, Alex J, Cocks, Kim, Bell, Sue E, Navarro-Coy, Nuria, Drayson, Mark T, Owen, Roger G, Feyler, Sylvia, Ashcroft, A John, Ross, Fiona M, Byrne, Jennifer, Roddie, Huw, Rudin, Claudius, Cook, Gordon, Jackson, Graham H, Wu, Ping, and Davies, Faith E

Published
2011
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31. Osteonecrosis of the jaw and renal safety in patients with newly diagnosed multiple myeloma: Medical Research Council Myeloma IX Study results

Author

Jackson, Graham H., Morgan, Gareth J., Davies, Faith E., Wu, Ping, Gregory, Walter M., Bell, Sue E., Szubert, Alexander J., Coy, Nuria Navarro, Drayson, Mark T., Owen, Roger G., Feyler, Sylvia, Ashcroft, Andrew J., Ross, Fiona M., Byrne, Jennifer, Roddie, Huw, Rudin, Claudius, Boyd, Kevin D., Osborne, Wendy L., Cook, Gordon, and Child, Anthony J.

Published
2014
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32. Constitutional and somatic rearrangement of chromosome 21 in acute lymphoblastic leukaemia

Author

Li, Yilong, Schwab, Claire, Ryan, Sarra L., Papaemmanuil, Elli, Robinson, Hazel M., Jacobs, Patricia, Moorman, Anthony V., Dyer, Sara, Borrow, Julian, Griffiths, Mike, Heerema, Nyla A., Carroll, Andrew J., Talley, Polly, Bown, Nick, Telford, Nick, Ross, Fiona M., Gaunt, Lorraine, McNally, Richard J. Q., Young, Bryan D., Sinclair, Paul, Rand, Vikki, Teixeira, Manuel R., Joseph, Olivia, Robinson, Ben, Maddison, Mark, Dastugue, Nicole, Vandenberghe, Peter, Haferlach, Claudia, Stephens, Philip J., Cheng, Jiqiu, Van Loo, Peter, Stratton, Michael R., Campbell, Peter J., and Harrison, Christine J.

Published
2014
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49. Patient compliance - whose responsibility?

Author

Ross, Fiona M.

Subjects
Physician and patient -- Research, Aged patients -- Care and treatment, Drug utilization -- Research, Nurse and patient -- Research, Patient compliance -- Research, Health, Social sciences
Abstract

This paper focuses on general practitioner and district nurse knowledge of prescribed drugs taken by their elderly patients, and the evaluation of a method of improving that knowledge by a patient held drug guide. Serious differences were identified in the knowledge of doctors, nurses and patients about drugs. The drug guide was associated with an improvement in patient knowledge of drugs as well as a reduction in disagreements between doctors and nurses. A key issue addressed is patient culpability in compliance. It is argued that this is a simplification which overlooks important factors such as inadequate communication and lack of knowledge on the part of doctors and nurses as well as the difficulties the patient may have with the regime.

Published
1991

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