Search

Your search keyword '"Roberts, Nicholas J."' showing total 503 results
Start Over Author "Roberts, Nicholas J."
503 results on '"Roberts, Nicholas J."'

2. 3D genomic mapping reveals multifocality of human pancreatic precancers

Author

Braxton, Alicia M., Kiemen, Ashley L., Grahn, Mia P., Forjaz, André, Parksong, Jeeun, Mahesh Babu, Jaanvi, Lai, Jiaying, Zheng, Lily, Niknafs, Noushin, Jiang, Liping, Cheng, Haixia, Song, Qianqian, Reichel, Rebecca, Graham, Sarah, Damanakis, Alexander I., Fischer, Catherine G., Mou, Stephanie, Metz, Cameron, Granger, Julie, Liu, Xiao-Ding, Bachmann, Niklas, Zhu, Yutong, Liu, YunZhou, Almagro-Pérez, Cristina, Jiang, Ann Chenyu, Yoo, Jeonghyun, Kim, Bridgette, Du, Scott, Foster, Eli, Hsu, Jocelyn Y., Rivera, Paula Andreu, Chu, Linda C., Liu, Fengze, Fishman, Elliot K., Yuille, Alan, Roberts, Nicholas J., Thompson, Elizabeth D., Scharpf, Robert B., Cornish, Toby C., Jiao, Yuchen, Karchin, Rachel, Hruban, Ralph H., Wu, Pei-Hsun, Wirtz, Denis, and Wood, Laura D.

Published
2024
Full Text
View/download PDF

3. Temporal and probabilistic comparisons of epidemic interventions

Author

Boudreau, Mariah C., Allen, Andrea J., Roberts, Nicholas J., Allard, Antoine, and Hébert-Dufresne, Laurent

Subjects
Quantitative Biology - Populations and Evolution
Abstract

Forecasting disease spread is a critical tool to help public health officials design and plan public health interventions.However, the expected future state of an epidemic is not necessarily well defined as disease spread is inherently stochastic, contact patterns within a population are heterogeneous, and behaviors change. In this work, we use time-dependent probability generating functions (PGFs) to capture these characteristics by modeling a stochastic branching process of the spread of a disease over a network of contacts in which public health interventions are introduced over time. To achieve this, we define a general transmissibility equation to account for varying transmission rates (e.g. masking), recovery rates (e.g. treatment), contact patterns (e.g. social distancing) and percentage of the population immunized (e.g. vaccination). The resulting framework allows for a temporal and probabilistic analysis of an intervention's impact on disease spread, which match continuous-time stochastic simulations that are much more computationally expensive.To aid policy making, we then define several metrics over which temporal and probabilistic intervention forecasts can be compared: Looking at the expected number of cases and the worst-case scenario over time, as well as the probability of reaching a critical level of cases and of not seeing any improvement following an intervention.Given that epidemics do not always follow their average expected trajectories and that the underlying dynamics can change over time, our work paves the way for more detailed short-term forecasts of disease spread and more informed comparison of intervention strategies., Comment: 20 pages, 5 figures

Published
2023
Full Text
View/download PDF

5. Predicting the diversity of early epidemic spread on networks

Author

Allen, Andrea J., Boudreau, Mariah C., Roberts, Nicholas J., Allard, Antoine, and Hébert-Dufresne, Laurent

Subjects
Quantitative Biology - Populations and Evolution, Physics - Physics and Society
Abstract

The interplay of biological, social, structural and random factors makes disease forecasting extraordinarily complex. The course of an epidemic exhibits average growth dynamics determined by features of the pathogen and the population, yet also features significant variability reflecting the stochastic nature of disease spread. In this work, we reframe a stochastic branching process analysis in terms of probability generating functions and compare it to continuous time epidemic simulations on networks. In doing so, we predict the diversity of emerging epidemic courses on both homogeneous and heterogeneous networks. We show how the challenge of inferring the early course of an epidemic falls on the randomness of disease spread more so than on the heterogeneity of contact patterns. We provide an analysis which helps quantify, in real time, the probability that an epidemic goes supercritical or conversely, dies stochastically. These probabilities are often assumed to be one and zero, respectively, if the basic reproduction number, or R0, is greater than 1, ignoring the heterogeneity and randomness inherent to disease spread. This framework can give more insight into early epidemic spread by weighting standard deterministic models with likelihood to inform pandemic preparedness with probabilistic forecasts.

Published
2021
Full Text
View/download PDF

7. Epichloë fungal endophyte interactions in perennial ryegrass (Lolium perenne L.) modified to accumulate foliar lipids for increased energy density

Author

Richardson, Kim A., de Bonth, Anouck C. M., Beechey-Gradwell, Zac, Kadam, Suhas, Cooney, Luke J., Nelson, Kelly A., Cookson, Ruth, Winichayakul, Somrutai, Reid, Michele, Anderson, Philip, Crowther, Tracey, Zou, Xiuying, Maher, Dorothy, Xue, Hong, Scott, Richard W., Allan, Anne, Johnson, Richard D., Card, Stuart D., Mace, Wade J., Roberts, Nicholas J., and Bryan, Gregory

Published
2023
Full Text
View/download PDF

8. Intraductal Transplantation Models of Human Pancreatic Ductal Adenocarcinoma Reveal Progressive Transition of Molecular Subtypes

Author

Miyabayashi, Koji, Baker, Lindsey A, Deschênes, Astrid, Traub, Benno, Caligiuri, Giuseppina, Plenker, Dennis, Alagesan, Brinda, Belleau, Pascal, Li, Siran, Kendall, Jude, Jang, Gun Ho, Kawaguchi, Risa Karakida, Somerville, Tim DD, Tiriac, Hervé, Hwang, Chang-Il, Burkhart, Richard A, Roberts, Nicholas J, Wood, Laura D, Hruban, Ralph H, Gillis, Jesse, Krasnitz, Alexander, Vakoc, Christopher R, Wigler, Michael, Notta, Faiyaz, Gallinger, Steven, Park, Youngkyu, and Tuveson, David A

Subjects
Rare Diseases, Clinical Research, Cancer, Pancreatic Cancer, Digestive Diseases, 2.1 Biological and endogenous factors, Aetiology, Adenocarcinoma, Animals, Carcinoma, Pancreatic Ductal, Disease Models, Animal, Gene Expression Regulation, Neoplastic, Humans, Mice, Pancreatic Ducts, Prognosis, Oncology and Carcinogenesis
Abstract

Pancreatic ductal adenocarcinoma (PDAC) is the most lethal common malignancy, with little improvement in patient outcomes over the past decades. Recently, subtypes of pancreatic cancer with different prognoses have been elaborated; however, the inability to model these subtypes has precluded mechanistic investigation of their origins. Here, we present a xenotransplantation model of PDAC in which neoplasms originate from patient-derived organoids injected directly into murine pancreatic ducts. Our model enables distinction of the two main PDAC subtypes: intraepithelial neoplasms from this model progress in an indolent or invasive manner representing the classical or basal-like subtypes of PDAC, respectively. Parameters that influence PDAC subtype specification in this intraductal model include cell plasticity and hyperactivation of the RAS pathway. Finally, through intratumoral dissection and the direct manipulation of RAS gene dosage, we identify a suite of RAS-regulated secreted and membrane-bound proteins that may represent potential candidates for therapeutic intervention in patients with PDAC. SIGNIFICANCE: Accurate modeling of the molecular subtypes of pancreatic cancer is crucial to facilitate the generation of effective therapies. We report the development of an intraductal organoid transplantation model of pancreatic cancer that models the progressive switching of subtypes, and identify stochastic and RAS-driven mechanisms that determine subtype specification.See related commentary by Pickering and Morton, p. 1448.This article is highlighted in the In This Issue feature, p. 1426.

Published
2020

11. First international workshop of the ATM and cancer risk group (4-5 December 2019)

Author

Lesueur, Fabienne, Easton, Douglas F., Renault, Anne-Laure, Tavtigian, Sean V., Bernstein, Jonine L., Kote-Jarai, Zsofia, Eeles, Rosalind A., Plaseska-Karanfia, Dijana, Feliubadaló, Lidia, Arun, Banu, Herold, Natalie, Versmold, Beatrix, Schmutzler, Rita Katharina, Nguyen-Dumont, Tú, Southey, Melissa C., Dorling, Leila, Dunning, Alison M., Ghiorzo, Paola, Dalmasso, Bruna Samia, Cavaciuti, Eve, Le Gal, Dorothée, Roberts, Nicholas J., Dominguez-Valentin, Mev, Rookus, Matti, Taylor, Alexander M. R., Goldstein, Alisa M., Goldgar, David E., Stoppa-Lyonnet, Dominique, and Andrieu, Nadine

Published
2022
Full Text
View/download PDF

17. Contributors

Author

Bovenga, F., primary, Bowman, E.T., additional, Brideau, Marc-André, additional, Clague, John J., additional, Crosta, Giovanni B., additional, Davies, Tim, additional, Delcamp, Audray, additional, Deline, P., additional, Hewitt, K., additional, Jakob, Matthias, additional, Korup, Oliver, additional, Massey, Chris, additional, McColl, Samuel T., additional, Moretti, Danilo, additional, Murphy, Bill, additional, Reznichenko, N., additional, Roberts, Nicholas J., additional, Rosser, Nick, additional, Shugar, D., additional, Sosio, Rosanna, additional, Utili, Stefano, additional, van Wyk de Vries, Benjamin, additional, Wang, Gonghui, additional, Warburton, Jeff, additional, and Wasowski, J., additional

Published
2022
Full Text
View/download PDF

25. Hydrogeological and geophysical properties of the very-slow-moving Ripley Landslide, Thompson River valley, British Columbia

Author

Huntley, David, Holmes, Jessica, Bobrowsky, Peter, Chambers, Jonathan, Meldrum, Philip, Wilkinson, Paul, Donohue, Shane, Elwood, David, Sattler, Kelvin, Hendry, Michael, Macciotta, Renato, and Roberts, Nicholas J.

Subjects
Prospecting -- Geophysical methods, Environmental monitoring -- Methods, Landslides -- Environmental aspects -- Models, Emergency management -- Methods, Disaster planning, Earth sciences
Abstract

Landslides along a 10 km reach ofThompson River southofAshcroft, British Columbia, have repeatedly damaged vital railway infrastructure, while also placing public safety, the environment, natural resources, and cultural heritage features at risk. Government agencies, universities, and the railway industry are focusing research efforts on a representative test site--the very-slow-moving Ripley Landslide--to manage better the geohazard risk in this corridor. We characterize the landslide's form and function through hydrogeological and geophysical mapping. Field mapping and exploratory drilling distinguish 10 hydrogeological units in surficial deposits and fractured bedrock. Electrical resistivity tomography, frequency domain electromagnetic conductivity measurements, ground-penetrating radar, seismic pressure wave refraction, and multispectral analysis of shear waves; in conjunction with downhole measurement of natural gamma radiation, induction conductivity, and magnetic susceptibility provide a detailed, static picture of soil moisture and groundwater conditions within the hydrogeological units. Differences in electrical resistivity of the units reflect a combination of hydrogeological characteristics and climatic factors, namely temperature and precipitation. Resistive earth materials include dry glaciofluvial outwash and non-fractured bedrock; whereas glaciolacustrine clay and silt, water-bearing fractured bedrock, and periodically saturated subglacial till and outwash are conductive. Dynamic, continuous real-time monitoring of electrical resistivity, now underway, will help characterize water-flow paths, and possible relationships to independently monitor pore pressures and slope creep. These new hydrogeological and geophysical data sets enhance understanding of the composition and internal structure of this landslide and provide important context to interpret multiyear slope stability monitoring ongoing in the valley. Key words: surficial mapping, geophysical surveys, landslide, geohazard monitoring, British Columbia. Les glissements de terrain le long d'un troncon de 10 km de la riviere Thompson au sud d'Ashcroft (Colombie-Britannique) ont cause des dommages a repetition a des infrastructures ferroviaires essentielles, tout en posant des risques pour la securite du public, le milieu ambiant, des ressources naturelles et des elements du patrimoine culturel. Des agences gouvernementales, des universites et le secteur ferroviaire ont axe des efforts de recherche sur un site d'essai representatif, le glissement tres lent de Ripley, afin de mieux gerer les georisques le long de ce corridor. Nous caracterisons la forme et la fonction de ce glissement a la lumiere de la cartographie hydrogeologique et geophysique. La cartographie de terrain et le forage d'exploration permettent de distinguer 10 unites hydrogeologiques dans les depots de surface et le roc fracture. La tomographie electrique, des mesures de conductivite electromagnetique en domaine de frequence, le georadar, la refraction des ondes sismiques de compression et l'analyse multispectrale des ondes de cisaillement, combines a la mesure au fond de puits du rayonnement gamma naturel, de la conductivite par induction et de la susceptibilite magnetique, fournissent un portait statique detaille de l'eau dans le sol et des conditions associees a l'eau souterraine dans les unites hydrogeologiques. Les differences de resistivite electrique entre unites refletent une combinaison de caracteristiques hydrogeologiques et de facteurs climatiques, a savoir la temperature et les precipitations. Les materiaux resistifs comprennent des depots d'epandage fluvioglaciaire secs et le roc non fracture, alors que les argiles et silts glaciolacustres, le roc fracture contenant de l'eau et du till infraglaciaire et des depots d'epandage periodiquement satures sont conducteurs. La surveillance continue et dynamique en temps reel de la resistivite electrique actuellement en cours aidera a caracteriser les voies d'ecoulement de l'eau et les relations possibles avec la pression interstitielle et la reptation de talus, qui font l'objet d'une surveillance independante. Ces nouveaux ensembles de donnees hydrogeologiques et geophysiques ameliorent la comprehension de la composition et de la structure interne de ce glissement de terrain et fournissent un important contexte pour l'interpretation des donnees issues de la surveillance pluriannuelle de la stabilite en cours dans la vallee. [Traduit par la Redaction] Mots-cles : cartographie de surface, leves geophysiques, glissement de terrain, surveillance des georisques, Colombie-Britannique., Introduction For many countries around the world, rail transport is the dominant mode for transporting natural resources to and from deepwater marine terminals, and transporting commuters and tourists to and [...]

Published
2020
Full Text
View/download PDF

26. RAD51B Harbors Germline Mutations Associated With Pancreatic Ductal Adenocarcinoma

Author

Xie, Fanfan, Ding, Ding, Lin, Cong, Cunningham, Dea, Wright, Michael, Javed, Ammar A., Azad, Nilo, Lee, Valerie, Donehower, Ross, De Jesus-Acosta, Ana, Le, Dung T., Pishvaian, Michael, Shin, Eun Ji, Lennon, Anne Marie, Khashab, Mouen, Singh, Vikesh, Klein, Alison P., Roberts, Nicholas J., Hacker-Prietz, Amy, McPhaul, Thomas, Burkhart, Richard A., Burns, William R., Narang, Amol, Zaheer, Atif, Fishman, Elliot K., Thompson, Elizabeth D., Anders, Robert, Yu, Jun, He, Jin, Wolfgang, Christopher L., Zheng, Lei, Liu, Dongbing, Wu, Kui, and Laheru, Daniel A.

Published
2022
Full Text
View/download PDF

28. Familial Pancreatic Cancer

Author

Roberts, Nicholas J., Klein, Alison P., Urrutia, Raul A., Section Editor, Büchler, Markus W., Section Editor, Neoptolemos, John, Section Editor, Neoptolemos, John P., editor, Urrutia, Raul, editor, Abbruzzese, James L., editor, and Büchler, Markus W., editor

Published
2018
Full Text
View/download PDF

30. Prevalence of Germline Mutations Associated With Cancer Risk in Patients With Intraductal Papillary Mucinous Neoplasms

Author

Skaro, Michael, Nanda, Neha, Gauthier, Christian, Felsenstein, Matthäus, Jiang, Zhengdong, Qiu, Miaozhen, Shindo, Koji, Yu, Jun, Hutchings, Danielle, Javed, Ammar A., Beckman, Ross, He, Jin, Wolfgang, Christopher L., Thompson, Elizabeth, Hruban, Ralph H., Klein, Alison P., Goggins, Michael, Wood, Laura D., and Roberts, Nicholas J.

Published
2019
Full Text
View/download PDF

31. CRISPR-Cas9 for selective targeting of somatic mutations in pancreatic cancers.

Author

Teh, Selina Shiqing K, Bowland, Kirsten, Halper-Stromberg, Eitan, Kotwal, Akhil, Bennett, Alexis, Skaist, Alyza, Tang, Jacqueline, Cai, Fidel, Macoretta, Antonella, Liang, Hong, Kamiyama, Hirohiko, Wheelan, Sarah, Lin, Ming-Tseh, Hruban, Ralph H, Hung, Chien-Fu, Goldstein, Michael, Scharpf, Robert B, Roberts, Nicholas J, and Eshleman, James R

Published
2024
Full Text
View/download PDF

34. Mutations in the pancreatic secretory enzymes CPA1 and CPB1 are associated with pancreatic cancer

Author

Tamura, Koji, Yu, Jun, Hata, Tatsuo, Suenaga, Masaya, Shindo, Koji, Abe, Toshiya, MacGregor-Das, Anne, Borges, Michael, Wolfgang, Christopher L., Weiss, Matthew J., He, Jin, Canto, Marcia Irene, Petersen, Gloria M., Gallinger, Steven, Syngal, Sapna, Brand, Randall E., Rustgi, Anil, Olson, Sara H., Stoffel, Elena, Cote, Michele L., Zogopoulos, George, Potash, James B., Goes, Fernando S., McCombie, Richard W., Zandi, Peter P., Pirooznia, Mehdi, Kramer, Melissa, Parla, Jennifer, Eshleman, James R., Roberts, Nicholas J., Hruban, Ralph H., Klein, Alison Patricia, and Goggins, Michael

Published
2018

36. Exome-Wide Association Study of Pancreatic Cancer Risk

Author

Grant, Robert C., Denroche, Robert E., Borgida, Ayelet, Virtanen, Carl, Cook, Natalie, Smith, Alyssa L., Connor, Ashton A., Wilson, Julie M., Peterson, Gloria, Roberts, Nicholas J., Klein, Alison P., Grimmond, Sean M., Biankin, Andrew, Cleary, Sean, Moore, Malcolm, Lemire, Mathieu, Zogopoulos, George, Stein, Lincoln, and Gallinger, Steven

Published
2018
Full Text
View/download PDF

38. Genomic characterization of malignant progression in neoplastic pancreatic cysts

Author

Noë, Michaël, Niknafs, Noushin, Fischer, Catherine G., Hackeng, Wenzel M., Beleva Guthrie, Violeta, Hosoda, Waki, Debeljak, Marija, Papp, Eniko, Adleff, Vilmos, White, James R., Luchini, Claudio, Pea, Antonio, Scarpa, Aldo, Butturini, Giovanni, Zamboni, Giuseppe, Castelli, Paola, Hong, Seung-Mo, Yachida, Shinichi, Hiraoka, Nobuyoshi, Gill, Anthony J., Samra, Jaswinder S., Offerhaus, G. Johan A., Hoorens, Anne, Verheij, Joanne, Jansen, Casper, Adsay, N. Volkan, Jiang, Wei, Winter, Jordan, Albores-Saavedra, Jorge, Terris, Benoit, Thompson, Elizabeth D., Roberts, Nicholas J., Hruban, Ralph H., Karchin, Rachel, Scharpf, Robert B., Brosens, Lodewijk A. A., Velculescu, Victor E., and Wood, Laura D.

Published
2020
Full Text
View/download PDF

41. Somatic loss ofATMis a late event in pancreatic tumorigenesis

Author

Paranal, Raymond M, primary, Jiang, Zhengdong, additional, Hutchings, Danielle, additional, Kryklyva, Valentyna, additional, Gauthier, Christian, additional, Fujikura, Kohei, additional, Nanda, Neha, additional, Huang, Bo, additional, Skaro, Michael, additional, Wolfgang, Christopher L, additional, He, Jin, additional, Klimstra, David S, additional, Brand, Randall E, additional, Singhi, Aatur D, additional, DeMarzo, Angelo, additional, Zheng, Lei, additional, Goggins, Michael, additional, Brosens, Lodewijk AA, additional, Hruban, Ralph H, additional, Klein, Alison P, additional, Lotan, Tamara, additional, Wood, Laura D, additional, and Roberts, Nicholas J, additional

Published
2023
Full Text
View/download PDF

43. Somatic protospacer adjacent motifs are numerous and selectively targetable in cancers

Author

Teh, Selina Shiqing K, primary, Bowland, Kirsten, additional, Bennett, Alexis, additional, Halper-Stromberg, Eitan, additional, Skaist, Alyza, additional, Pallavajjala, Aparna, additional, Tang, Jacqueline, additional, Cai, Fidel, additional, Macoretta, Antonella, additional, Liang, Hong, additional, Wheelan, Sarah, additional, Lin, Ming-Tseh, additional, Hruban, Ralph H, additional, Scharpf, Robert B, additional, Roberts, Nicholas J, additional, and Eshleman, James R, additional

Published
2023
Full Text
View/download PDF

45. Mechanism of delayed cell death following simultaneous CRISPR-Cas9 targeting in pancreatic cancers

Author

Teh, Selina Shiqing K, primary, Halper-Stromberg, Eitan, additional, Morsberger, Laura, additional, Bennett, Alexis, additional, Bowland, Kirsten, additional, Skaist, Alyza, additional, Cai, Fidel, additional, Liang, Hong, additional, Hruban, Ralph H, additional, Roberts, Nicholas J, additional, Scharpf, Robert B, additional, Zou, Ying S, additional, and Eshleman, James R, additional

Published
2023
Full Text
View/download PDF

47. Supplementary Figure S7 from Intraductal Transplantation Models of Human Pancreatic Ductal Adenocarcinoma Reveal Progressive Transition of Molecular Subtypes

Author

Miyabayashi, Koji, primary, Baker, Lindsey A., primary, Deschênes, Astrid, primary, Traub, Benno, primary, Caligiuri, Giuseppina, primary, Plenker, Dennis, primary, Alagesan, Brinda, primary, Belleau, Pascal, primary, Li, Siran, primary, Kendall, Jude, primary, Jang, Gun Ho, primary, Kawaguchi, Risa Karakida, primary, Somerville, Tim D.D., primary, Tiriac, Hervé, primary, Hwang, Chang-Il, primary, Burkhart, Richard A., primary, Roberts, Nicholas J., primary, Wood, Laura D., primary, Hruban, Ralph H., primary, Gillis, Jesse, primary, Krasnitz, Alexander, primary, Vakoc, Christopher R., primary, Wigler, Michael, primary, Notta, Faiyaz, primary, Gallinger, Steven, primary, Park, Youngkyu, primary, and Tuveson, David A., primary

Published
2023
Full Text
View/download PDF

48. Data from ATM Mutations in Patients with Hereditary Pancreatic Cancer

Author

Roberts, Nicholas J., primary, Jiao, Yuchen, primary, Yu, Jun, primary, Kopelovich, Levy, primary, Petersen, Gloria M., primary, Bondy, Melissa L., primary, Gallinger, Steven, primary, Schwartz, Ann G., primary, Syngal, Sapna, primary, Cote, Michele L., primary, Axilbund, Jennifer, primary, Schulick, Richard, primary, Ali, Syed Z., primary, Eshleman, James R., primary, Velculescu, Victor E., primary, Goggins, Michael, primary, Vogelstein, Bert, primary, Papadopoulos, Nickolas, primary, Hruban, Ralph H., primary, Kinzler, Kenneth W., primary, and Klein, Alison P., primary

Published
2023
Full Text
View/download PDF

49. Data from Intraductal Transplantation Models of Human Pancreatic Ductal Adenocarcinoma Reveal Progressive Transition of Molecular Subtypes

Author

Miyabayashi, Koji, primary, Baker, Lindsey A., primary, Deschênes, Astrid, primary, Traub, Benno, primary, Caligiuri, Giuseppina, primary, Plenker, Dennis, primary, Alagesan, Brinda, primary, Belleau, Pascal, primary, Li, Siran, primary, Kendall, Jude, primary, Jang, Gun Ho, primary, Kawaguchi, Risa Karakida, primary, Somerville, Tim D.D., primary, Tiriac, Hervé, primary, Hwang, Chang-Il, primary, Burkhart, Richard A., primary, Roberts, Nicholas J., primary, Wood, Laura D., primary, Hruban, Ralph H., primary, Gillis, Jesse, primary, Krasnitz, Alexander, primary, Vakoc, Christopher R., primary, Wigler, Michael, primary, Notta, Faiyaz, primary, Gallinger, Steven, primary, Park, Youngkyu, primary, and Tuveson, David A., primary

Published
2023
Full Text
View/download PDF

50. Supplementary Video3 from Intraductal Transplantation Models of Human Pancreatic Ductal Adenocarcinoma Reveal Progressive Transition of Molecular Subtypes

Author

Miyabayashi, Koji, primary, Baker, Lindsey A., primary, Deschênes, Astrid, primary, Traub, Benno, primary, Caligiuri, Giuseppina, primary, Plenker, Dennis, primary, Alagesan, Brinda, primary, Belleau, Pascal, primary, Li, Siran, primary, Kendall, Jude, primary, Jang, Gun Ho, primary, Kawaguchi, Risa Karakida, primary, Somerville, Tim D.D., primary, Tiriac, Hervé, primary, Hwang, Chang-Il, primary, Burkhart, Richard A., primary, Roberts, Nicholas J., primary, Wood, Laura D., primary, Hruban, Ralph H., primary, Gillis, Jesse, primary, Krasnitz, Alexander, primary, Vakoc, Christopher R., primary, Wigler, Michael, primary, Notta, Faiyaz, primary, Gallinger, Steven, primary, Park, Youngkyu, primary, and Tuveson, David A., primary

Published
2023
Full Text
View/download PDF

Catalog

Books, media, physical & digital resources
See catalog results