205 results on '"Rieckmann T"'
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2. P08 Radiosensitivity and impaired DNA double-strand break repair in HPV-positive OPSCC: The role of smoking and the ATM kinase as observed in ex vivo and in vitro studies
3. Current studies of immunotherapy in head and neck cancer
4. SP-1002 Radiosenstivity of HPV-related SCC
5. Radiation DNA damage and use in cancer/therapeutics-translation of radiation modifiers
6. p53 modulates homologous recombination at I-SceI-induced double-strand breaks through cell-cycle regulation
7. Analyse von Antikörpern gegen frühe HPV16-Antigene und Follow-up von HPV-16 E6-positiven Patienten in der prospektiven Kohorte der Hamburg City Health Study (HCHS)
8. Analysis of Human papillomavirus (HPV) 16 early protein antibodies and follow-up of HPV-16 E6-positive patients in the prospective cohort of the Hamburg City Health Study (HCHS)
9. Nachweis eines DNA-Reparaturdefekts HPV-positiver OPSCC mittels ex vivo kultivierter, humaner Tumorschnitte
10. Patient derived e x vivo slice cultures demonstrate a profound DNA double-strand break repair defect in HPV-positive OPSCC
11. A lack of effectiveness in the ATM-orchestrated DNA damage response contributes to the enhanced radiation sensitivity of HPV-positive HNSCC cell lines
12. Eingeschränkte Effektivität der ATM-gesteuerten DNA Schadensantwort als Ursache der erhöhten zellulären Strahlenempfindlichkeit HPV-positiver HNSCC Zellen
13. Radiosensitization of HPV Positive Head and Neck Squamous Cell Carcinomas (HNSCC) by Chk1 Inhibition: ID 301
14. The predictive role of Axl and Gas6 in patients with squamous cell carcinoma of the head and neck
15. Analyse von Antikörpern gegen frühe HPV16-Antigene und Identifizierung von Patienten mit hohem Risiko für HPV-assoziierte Oropharynxkarzinome in der prospektiven Hamburg City Health Study
16. Analysis of HPV 16 early protein antibodies and identification of patients at high risk for HPV-driven oropharyngeal cancer in the prospective cohort of the Hamburg City Health Study
17. Ex-vivo-Kulturen humaner HNSCC Tumoren bestätigen eine eingeschränkte DNA-Doppelstrangbruch-Reparatur HPV-positiver OPSCC und weisen auf einen Defekt der ATM-abhängigen Schadensantwort hin
18. Ex-vivo slice cultures of tumor tissues confirm the DNA double-strand break repair defect of HPV-positive OPSCC and suggest a defect in the ATM-mediated DNA damage response
19. Die prädiktive Rolle von Axl und Gas6 bei Patienten mit Plattenepithelkarzinomen des Kopf-Halsbereiches
20. ADVANCING RECOVERY: STATE AND PROVIDER PARTNERSHIPS TO PROMOTE MEDICATIONS FOR THE TREATMENT OF ALCOHOL USE DISORDERS: 160
21. Tissue micro array based evaluation of the central DNA double-strand break repair protein KU80 as a prognostic marker in HNSCC
22. Tissue Micro Array basierte Evaluation des zentralen DNA Doppelstrangbruch-Reparaturproteins KU80 als möglicher prognostischer Marker in HNSCC
23. PO-1085 Prolonged trifluridine/tipiracil treatment radiosensitises colorectal cancer cells
24. A tissue micro array analysis of Src kinase expression and activity in HNSCC
25. Tissue Micro-Array basierte Analyse der Expression und Aktivität von Src-Kinasen in HNSCC
26. PO-1083 The dual inhibitor BEZ235 radiosensitizes HNSCC cells due to impairment of the DSB repair
27. Dielectric relaxation spectroscopy of reactively blended amorphous poly(ethylene terephthalate)–poly(ethylene naphthalate) films
28. Duale Inhibition von PARP1 und dem intra-S/G2-Zellzykluskontrollpunkt als neue Strategie zur hoch wirksamen Strahlensensitivierung HPV+ HNSCC
29. Massenspektrometrischer Vergleich HPV+ und HPV- HNSCC Tumoren und Zelllinien
30. OC-0490: Targeting PARP1 and the intra-S/G2 checkpoints for highly effective radiosensitization of HPV+ HNSCC
31. Dual inhibition of PARP1 and the intra-S/G2 cell cycle checkpoints as a novel strategy for highly effective radiosensitization of HPV+ HNSCC
32. Untersuchung der Rolle der ATM-Kinase für die erhöhte Strahlenempfindlichkeit HPV-positiver HNSCC
33. Mass spectrometric comparison of HPV+ and HPV- HNSCC tumors and cell lines
34. A possible role of the ATM kinase for the enhanced radiation sensitivity of HPV+ HNSCC
35. Chapter 11 - Radiation DNA damage and use in cancer/therapeutics-translation of radiation modifiers
36. Identifizierung funktioneller Proteomsignaturen in HPV positiven Kopf-Hals Karzinomen
37. Radiosensitivierung HPV-positiver HNSCC Zellen durch alleinige und kombinierte Inhibition von Chk1 und Wee1
38. Current studies of immunotherapy in head and neck cancer
39. Chemosensitivität von HPV-positiven und HPV-negativen Kopf-Hals-Tumorzelllinien
40. G2-checkpoint targeting and radiosensitization of HPV/p16-positive HNSCC cells through the inhibition of Chk1 and Wee1
41. Radiosensitization of NSCLC cells by EGFR inhibition is the result of an enhanced p53-dependent G1 arrest.
42. Radiosensitivierung von HPV-positiven Kopf-Hals-Karzinomen durch PARP- und EGFR-Inhibition
43. Oregon's Medicaid Transformation -- Observations on Organizational Structure and Strategy
44. Strahlensensibilisierung durch Chk1 Inhibitor bei HPV positiven Kopf-Hals-Karzinomen
45. 173 - G2-checkpoint targeting and radiosensitization of HPV/p16-positive HNSCC cells through the inhibition of Chk1 and Wee1
46. SP-0557: Radiobiology of HPV-positive HNSCC cells: Mechanisms of radiosensitivity and implications for therapy
47. OC-0499: HNSCC cell lines positive for HPV and p16 possess high cellular radiosensitivity due to impaired DSB repair capacity
48. PD-0094: EGFR inhibition radiosensitizes NSCLC cells via permanent G1 arrest but only when p53/p21 signaling is intact
49. Treatment needs of American Indians with substance use disorders
50. p53 modulates homologous recombination at I-SceI-induced double-strand breaks through cell-cycle regulation
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