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1. Interferon-gamma is quintessential for NOS2 and COX2 expression in ER- breast tumors that lead to poor outcome

2. Systemic Nos2 Depletion and Cox inhibition limits TNBC disease progression and alters lymphoid cell spatial orientation and density

3. A divergent mode of activation of a nitrosyl iron complex with unusual antiangiogenic activity

6. NOS2 and COX-2 Co-Expression Promotes Cancer Progression: A Potential Target for Developing Agents to Prevent or Treat Highly Aggressive Breast Cancer.

7. NOS2 and COX2 Provide Key Spatial Targets that Determine Outcome in ER-Breast Cancer

8. Spatial analysis of NOS2 and COX2 interaction with T-effector cells reveals immunosuppressive landscapes associated with poor outcome in ER- breast cancer patients

18. Itaconic acid mediates crosstalk between macrophage metabolism and peritoneal tumors

21. IL-27 induces an IFN-like signature in murine macrophages which in turn modulate colonic epithelium

28. Supplementary Data from A Mitochondrial Target Sequence Polymorphism in Manganese Superoxide Dismutase Predicts Inferior Survival in Breast Cancer Patients Treated with Cyclophosphamide

29. Supplementary Tables 1 - 2 from NOS Inhibition Modulates Immune Polarization and Improves Radiation-Induced Tumor Growth Delay

30. Supplementary Methods from Dithiolethiones Inhibit NF-κB Activity via Covalent Modification in Human Estrogen Receptor–Negative Breast Cancer

32. Data from NOS Inhibition Modulates Immune Polarization and Improves Radiation-Induced Tumor Growth Delay

36. Supplementary Table 1 from Dithiolethiones Inhibit NF-κB Activity via Covalent Modification in Human Estrogen Receptor–Negative Breast Cancer

41. New nitrosyl ruthenium complexes with combined activities for multiple cardiovascular disorders

44. Nitric Oxide and Cancer: An Overview

46. Interferon-gamma is quintessential for NOS2 and COX2 expression in ER- breast tumors that lead to poor outcome.

47. Contributors

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