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4. Hematopoietic Stem Cell Transplantation as Treatment for Patients with DOCK8 Deficiency

7. Atopic dermatitis: Correlation of distinct risk factors with age of onset in adulthood compared to childhood

8. Heterozygous STAT1 gain-of-function mutations underlie an unexpectedly broad clinical phenotype

9. Atopic dermatitis: factors associated with age of onset in adulthood versus childhood [Abstract]

10. Atopic dermatitis: correlation of distinct risk factors with age of onset in adulthood compared to childhood

11. Atopic dermatitis: Correlation of distinct risk factors with age of onset in adulthood compared to childhood

14. Defective actin accumulation impairs human natural killer cell function in patients with dedicator of cytokinesis 8 deficiency

16. IL‐13, periostin and dipeptidyl‐peptidase‐4 reveal endotype‐phenotype associations in atopic dermatitis.

17. DOCK8 Deficiency: Clinical and Immunological Phenotype and Treatment Options - a Review of 136 Patients

18. Key findings to expedite the diagnosis of hyper-IgE syndromes in infants and young children

19. Diagnostic approach to the hyper-IgE syndromes: Immunologic and clinical key findings to differentiate hyper-IgE syndromes from atopic dermatitis

22. A Novel Gain-of-Function IKBA Mutation Underlies Ectodermal Dysplasia with Immunodeficiency and Polyendocrinopathy

23. Lung Parenchyma Surgery in Autosomal Dominant Hyper-IgE Syndrome

24. Commensal bacteria-derived signals regulate basophil hematopoiesis and allergic inflammation

25. Novel signal transducer and activator of transcription 3 (STAT3) mutations, reduced TH17 cell numbers, and variably defective STAT3 phosphorylation in hyper-IgE syndrome

27. Electrical impedance spectroscopy for the characterization of skin barrier in atopic dermatitis

28. Rescue of STAT3 Function in Hyper-IgE Syndrome Using Adenine Base Editing

30. Additional file 2 of Retained primary teeth in STAT3 hyper-IgE syndrome: early intervention in childhood is essential

31. Additional file 3 of Retained primary teeth in STAT3 hyper-IgE syndrome: early intervention in childhood is essential

34. Retained primary teeth in STAT3 hyper-IgE syndrome: early intervention in childhood is essential

36. Molecular Analysis of the MVK and TNFRSF1A Genes in Patients With a Clinical Presentation Typical of the Hyperimmunoglobulinemia D With Periodic Fever Syndrome: A Low-Penetrance TNFRSF1A Variant in a Heterozygous MVK Carrier Possibly Influences the Phenotype of Hyperimmunoglobulinemia D With Periodic Fever Syndrome or Vice Versa

39. Impact of high‐altitude therapy on type‐2 immune responses in asthma patients

40. Impaired memory B‐cell development and antibody maturation with a skewing toward IgE in patients with STAT3 hyper‐IgE syndrome

42. Lung disease inSTAT3 hyper‐IgE syndrome requires intense therapy

43. Somatic alterations compromised molecular diagnosis of DOCK8 hyper-IgE syndrome caused by a novel intronic splice site mutation

44. Impact of high‐altitude therapy on type‐2 immune responses in asthma patients.

45. Perception of climate change in patients with chronic lung disease

46. Lung disease in STAT3 hyper‐IgE syndrome requires intense therapy.

47. Heterozygous STAT1 gain-of-function mutations underlie an unexpectedly broad clinical phenotype

48. Outcome of HSCT in Adolescents and Young Adults with Non-SCID Primary Immunodeficiencies

49. DOCK8 Deficiency: Clinical and Immunological Phenotype and Treatment Options - a Review of 136 Patients

50. Targeted next-generation sequencing: a novel diagnostic tool for primary immunodeficiencies

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