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5. Glibenclamide reverses cardiovascular abnormalities of Cantu syndrome driven by [K.sub.ATP] channel overactivity

6. Loss of β-cell identity and dedifferentiation, not an irreversible process?

7. Preferential Gq signaling in diabetes: an electrical switch in incretin action and in diabetes progression?

9. KATP channels are necessary for glucose-dependent increases in amyloid-β and Alzheimer’s disease–related pathology

11. ABCC9-related Intellectual disability Myopathy Syndrome is a KATP channelopathy with loss-of-function mutations in ABCC9

18. Rpl13a small nucleolar RNAS regulate systemic glucose metabolism

25. Kir6.2-containing KATP channels are necessary for glucose dependent increases in amyloid-beta and Alzheimer's-related pathology

32. Sulfonylureas target the neurovascular response to decrease Alzheimer’s pathology

37. Complex consequences of Cantu syndrome SUR2 variant R1154Q in genetically modified mice

39. Genome-edited zebrafish model of ABCC8loss-of-function disease

41. Gain-of-Function Lrp5 Mutation Improves Bone Mass and Strength and Delays Hyperglycemia in a Mouse Model of Insulin-Deficient Diabetes

44. 1789-P: Inactivation of Acyl-Protein Thioesterase 1, a Major Mediator of Palmitoylation Cycling, Promotes Insulin Secretion and ß-Cell Failure

45. Kir6.1‐dependent KATP channels in lymphatic smooth muscle and vessel dysfunction in mice with Kir6.1 gain‐of‐function

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