41 results on '"Reis-Mendes, Ana"'
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2. The Role of Nrf2 and Inflammation on the Dissimilar Cardiotoxicity of Doxorubicin in Two-Time Points: a Cardio-Oncology In Vivo Study Through Time
3. The role of inflammation and antioxidant defenses in the cardiotoxicity of doxorubicin in elderly CD-1 male mice
4. Enduring metabolic modulation in the cardiac tissue of elderly CD-1 mice two months post mitoxantrone treatment
5. Long-term effects of the chronic administration of doxorubicin on aged skeletal muscle: An exploratory study in mice
6. Doxorubicin-induced neurotoxicity differently affects the hippocampal formation subregions in adult mice
7. Autophagy (but not metabolism) is a key event in mitoxantrone-induced cytotoxicity in differentiated AC16 cardiac cells
8. Chemobrain: mitoxantrone-induced oxidative stress, apoptotic and autophagic neuronal death in adult CD-1 mice
9. Exploring the aging effect of the anticancer drugs doxorubicin and mitoxantrone on cardiac mitochondrial proteome using a murine model
10. The Metabolic Fingerprint of Doxorubicin-Induced Cardiotoxicity in Male CD-1 Mice Fades Away with Time While Autophagy Increases
11. The Role of Nrf2 and Inflammation on the Dissimilar Cardiotoxicity of Doxorubicin in Two-Time Points: a Cardio-Oncology In Vivo Study Through Time
12. A Clinically Relevant Dosage of Mitoxantrone Disrupts the Glutathione and Lipid Metabolic Pathways of the CD-1 Mice Brain: A Metabolomics Study
13. Perfil de resistência das infecções de corrente sanguínea em pacientes onco-hematológicos de um centro de referência no Norte do brasil
14. Cardiac Molecular Remodeling by Anticancer Drugs: Doxorubicin Affects More Metabolism While Mitoxantrone Impacts More Autophagy in Adult CD-1 Male Mice
15. Doxorubicin-induced neurotoxicity differently affects the hippocampal formation subregions in adult mice
16. Autophagy (but not metabolism) is a key event in mitoxantrone-induced cytotoxicity in differentiated AC16 cardiac cells
17. Splenic morphologic changes induced by a strenuous and exhaustive training program in Wistar rats
18. Mitoxantrone-induced neurotoxicity in CD-1 mice
19. Molecular alterations underlying Doxorubicin’s Chronic Cardiotoxicity in a mouse model
20. Role of Inflammation and Redox Status on Doxorubicin-Induced Cardiotoxicity in Infant and Adult CD-1 Male Mice
21. Inflammation as a Possible Trigger for Mitoxantrone-Induced Cardiotoxicity: An In Vivo Study in Adult and Infant Mice
22. Discovery of New Potent Positive Allosteric Modulators of Dopamine D2 Receptors: Insights into the Bioisosteric Replacement of Proline to 3-Furoic Acid in the Melanostatin Neuropeptide
23. Cardiac Structural Repercussions Induced by Exhaustive Physical Training in the Animal Model
24. Anticancer drugs-induced toxicity in different age male CD-1 mice
25. Effects of Doxorubicin and Mitoxantrone in the brain of differently aged mice: in vivo chemobrain study
26. Disclosing the effect of doxorubicin and mitoxantrone on cardiac mitochondrial proteome: an in vivo approach using a murine model
27. The Main Metabolites of Fluorouracil + Adriamycin + Cyclophosphamide (FAC) Are Not Major Contributors to FAC Toxicity in H9c2 Cardiac Differentiated Cells
28. Doxorubicin Is Key for the Cardiotoxicity of FAC (5-Fluorouracil + Adriamycin + Cyclophosphamide) Combination in Differentiated H9c2 Cells
29. Discovery of New Potent Positive Allosteric Modulators of Dopamine D2Receptors: Insights into the Bioisosteric Replacement of Proline to 3-Furoic Acid in the Melanostatin Neuropeptide
30. Pixantrone, a new anticancer drug with the same old cardiac problems? An in vitro study with differentiated and non-differentiated H9c2 cells
31. Old Pharmaceuticals with New Applications: the Case Studies of Lucanthone and Mitoxantrone
32. The Role of the Metabolism of Anticancer Drugs in Their Induced-Cardiotoxicity
33. At pharmacology relevant concentrations, mitoxantrone causes time- and concentration-dependent toxicity in H9c2 differentiated cells with autophagy involvement
34. Pixantrone, a new anticancer drug with the same old cardiac problems? An in vitrostudy with differentiated and non-differentiated H9c2 cells
35. The Role of the Metabolism of Anticancer Drugs in Their Induced-Cardiotoxicity
36. Evaluation of the toxicity induced by Doxorubicin in different age male CD-1 mice
37. Evaluation of the toxicity induced by Doxorubicin in different age male CD-1 mice
38. Mitoxantrone-induced toxicity in different age male CD-1 mice
39. Violência doméstica e aspectos socioeconômicos: enlaces, negociações e rupturas
40. Discovery of New Potent Positive Allosteric Modulators of Dopamine D 2 Receptors: Insights into the Bioisosteric Replacement of Proline to 3-Furoic Acid in the Melanostatin Neuropeptide.
41. The Role of the Metabolism of Anticancer Drugs in Their Induced-Cardiotoxicity.
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