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1. Longitudinal analysis of the lung proteome reveals persistent repair months after mild to moderate COVID-19

2. Immune reconstitution inflammatory syndrome drives emergence of HIV drug resistance from multiple anatomic compartments in a person living with HIV

3. Long-term persistence of transcriptionally active ‘defective’ HIV-1 proviruses: implications for persistent immune activation during antiretroviral therapy

4. Defective HIV-1 proviruses produce viral proteins

5. Transcriptionally Active Defective HIV-1 Proviruses and Their Association With Immunological Nonresponse to Antiretroviral Therapy.

8. Clonally expanded CD4⁺ T cells can produce infectious HIV-1 in vivo

9. High-level dolutegravir resistance can emerge rapidly from few variants and spread by recombination: implications for integrase strand transfer inhibitor salvage therapy

11. Comprehensive analysis of unique cases with extraordinary control over HIV replication

15. A Combination of M50I and V151I Polymorphic Mutations in HIV-1 Subtype B Integrase Results in Defects in Autoprocessing

16. A Combination of Amino Acid Mutations Leads to Resistance to Multiple Nucleoside Analogs in Reverse Transcriptases from HIV-1 Subtypes B and C

18. Prolonged Posttreatment Virologic Control and Complete Seroreversion After Advanced Human Immunodeficiency Virus-1 Infection

20. Antigenic Restimulation of Virus-Specific Memory CD8 + T Cells Requires Days of Lytic Protein Accumulation for Maximal Cytotoxic Capacity

21. Dynamic Shifts in the HIV Proviral Landscape During Long Term Combination Antiretroviral Therapy: Implications for Persistence and Control of HIV Infections

25. Lytic Granule Loading of CD8+ T Cells Is Required for HIV-Infected Cell Elimination Associated with Immune Control

28. Longitudinal analysis of the lung proteome reveals persistent repair months after mild to moderate COVID-19

29. 2021 update to HIV-TRePS: a highly flexible and accurate system for the prediction of treatment response from incomplete baseline information in different healthcare settings.

30. 2018 update to the HIV-TRePS system: the development of new computational models to predict HIV treatment outcomes, with or without a genotype, with enhanced usability for low-income settings

31. Prolonged Posttreatment Virologic Control and Complete Seroreversion After Advanced Human Immunodeficiency Virus-1 Infection.

32. Defective HIV-1 proviruses produce viral proteins.

33. Early Presence of HIV-1 Subtype C in Washington, D.C.

35. Virion incorporation of integrin α4β7 facilitates HIV-1 infection and intestinal homing

36. Interleukin-15 (IL-15) Strongly Correlates with Increasing HIV-1 Viremia and Markers of Inflammation

37. Clonally expanded CD4 + T cells can produce infectious HIV-1 in vivo

38. CD8+ T-cell Cytotoxic Capacity Associated with Human Immunodeficiency Virus-1 Control Can Be Mediated through Various Epitopes and Human Leukocyte Antigen Types

39. IL-2 cycling causes transient increases in hsCRP and D-dimer that are not associated with plasma HIV-RNA levels

41. Plasma Interleukin-27 (IL-27) Levels Are Not Modulated in Patients with Chronic HIV-1 Infection

43. High Dose Atorvastatin Decreases Cellular Markers of Immune Activation Without Affecting HIV-1 RNA Levels: Results of a Double-Blind Randomized Placebo Controlled Clinical Trial

44. HIV Populations Are Large and Accumulate High Genetic Diversity in a Nonlinear Fashion

46. Microalbuminuria in HIV Disease

47. Defective HIV-1 proviruses produce novel proteincoding RNA species in HIV-infected patients on combination antiretroviral therapy.

48. The CD8+HLA-DR+T cells expanded in HIV-1 infection are qualitatively identical to those from healthy controls

50. CD8-EOMEShigh T cells define a subpopulation of CD8 memory cells that restore CD127high expression after cART treatment in HIV infected patients. (105.27)

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