504 results on '"Regnault V"'
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2. Erratum to: “Microvascular manifestations revealing vaccine-induced thrombotic thrombocytopenia after COVID-19 vaccination” [J. Med. Vasc. 47 (2022) 36–38]
3. Microvascular manifestations revealing vaccine-induced thrombotic thrombocytopenia after COVID-19 vaccination
4. An international multicentre‐laboratory evaluation of a new assay to detect specifically lupus anticoagulants dependent on the presence of anti‐beta2‐glycoprotein autoantibodies
5. Characterisation of a hypercoagulable phenotype with increased thrombin generation in acute heart failure: 134
6. Standardisation of thrombin generation test - which reference plasma for TGT?: An international multicentre study
7. Activated protein C resistance determined by thrombin generation is predictive of thrombosis in antiphospholipid-positive patients and associated auto-immune diseases: a multicenter prospective cohort study: OR186
8. Effects of antiphospholipid antibodies on vascular smooth muscle cells: OR091
9. Mild hemophilia A with factor VIII assay discrepancy: using thrombin generation assay to assess the bleeding phenotype
10. Chemical reports and memoirs ...
11. Increased fibrinogen rises thrombin generation and fibrin clot formation in obese Zucker rats: PB 4.64–1
12. Variability in exposure of epitope G40-R43 of domain I in commercial anti-β2-glycoprotein I IgG ELISAs influences the diagnosis of the antiphospholipid syndrome: PB 4.62–1
13. Vascular smooth muscle cells confer a prothrombotic phenotype within the vessel wall of spontaneously hypertensive rats: PA 2.17–2
14. Aldosterone decreases thrombin generation via enhancement of thrombomodulin-mediated protein C activation: OC 54.2
15. P150Hypercoagulable phenotype of the arterial wall of spontaneously hypertensive rats: involvement of vascular smooth muscle cells
16. Role of focal adhesions of vascular smooth muscle cells in thrombin generation
17. P6495Role of vascular smooth muscles cells in naked mole rats during ageing (Heterocephalus glaber)
18. The risk of heart valve disease associated with antiphospholipid antibodies in patients with systemic lupus erythematosus: a meta-analysis of echocardiographic studies: OC-MO-029
19. Nonbacterial purpura fulminans and severe autoimmune acquired protein S deficiency associated with human herpesvirus-6 active replication
20. Pursuing the debate on the serologic criteria that define the antiphospholipid syndrome
21. Anti-protein S antibodies following a varicella infection: detection, characterization and influence on thrombin generation
22. Nouvelles Recherches surla Compressibilite des gaz, et sur les Forces Elastiques des Vapeurs
23. Antistreptokinase platelet-activating antibodies are common and heterogeneous
24. 5.3 Platelet Aggregation is Modulated by Arterial Stiffness in End Stage Renal Disease
25. Both kinetic data and epitope mapping provide clues for understanding the anti-coagulant effect of five murine monoclonal antibodies to human β2-glycoprotein I
26. Interaction between hypertension and arterial stiffness an expert reappraisal
27. Omega-3 Fatty Acids Inhibit Thrombin Formation In Vascular Smooth Muscle Cells
28. Antibodies against phospholipids and beta2-glycoprotein I increase the risk of recurrent venous thromboembolism in patients without systemic lupus erythematosus
29. P3.06 Cyclic Stretch-Induced Thrombin Generation by Rat Vascular Smooth Muscle Cells is Mediated by the Integrin (V(3) Signaling Pathway)
30. P2.04: Paradoxical Involvement of the Endothelial Mineralocorticoid Receptor in Platelet Activation and Vascular Thrombosis in Mouse
31. P4.32: Inactivation of Serum Response Factor Contributes to Decrease Vascular Muscular Tone and Arterial Stiffness in Mice
32. P4.31: Arterial Stiffness and Haemostasis Changes in Obese Zucker Rats
33. Role of TGF-β co-receptor CD109 in extracellular matrix production of smooth muscle cells
34. Control of coagulation by the RhoA pathway and the exchange factor Arhgef1
35. Involvement of avß3 integrin and PAR signaling in thrombin generation and proliferation of vascular smooth muscle cells from thoracic and abdominal aortic aneurysms
36. P3.04 Increased Thrombin Generation and Vascular Remodeling in Obese Zucker Rats
37. P3.01 Phenotypic Modulation of Vascular Smooth Muscle Cells in Response to Hypertension Confers a Prothrombotic State Within the Vessel Wall
38. 7.6: Tissue Factor Pathway Inhibitor: A New Link Between Arterial Stiffness, Pulse Pressure and Coagulation in Postmenopausal Women
39. P7.06: Gender Difference in Cardiovascular Risk: Role of Blood Pressure Amplification
40. P11.04 Vascular Stiffness is Increased in Antiphospholipid Syndrome: Significance and Clinical Associations
41. 4803Differential implication of PAR signaling in thrombin generation and proliferation of vascular smooth muscle cells from human thoracic and abdominal aortic aneurysms
42. Conditional inactivation of integrin alpha v subunit in vascular smooth muscle cells decreases thrombin generation in vessels and blood
43. [PP.16.24] EFFECTS OF ANTIPHOSPHOLIPID ANTIBODIES ON VASCULAR SMOOTH MUSCLE CELLS
44. 23 Hypercoagulabilité dans l’hypertension pulmonaire : intérêt du potentiel thrombinique
45. Sul calorico specifico di alcuni corpi semplici
46. Risque d’hypertension pulmonaire associé aux anticorps antiphospholipides chez les patients atteints de lupus érythémateux systémique. Revue systématique et méta-analyse des études échocardiographiques
47. La résistance à la protéine C activée determinée par thrombinographie est prédictive du risque thrombotique chez les patients porteurs d’anticorps antiphospholipides et atteints de maladies auto-immunes associées. Une étude de cohorte prospective multicentrique
48. Les anticorps dirigés contre le domaine I de la β2-glycoprotéine I sont prédictifs des complications thrombotiques chez les patients atteints du syndrome des antiphospholipides : une étude prospective
49. Increased risk for heart valve disease associated with antiphospholipid antibodies in patients with systemic lupus erythematosus: meta-analysis of echocardiographic studies.
50. Un nouveau composé pyrroline sélectif des récepteurs imidazoliniques I1 améliore le syndrome métabolique d’un modèle de rongeur
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