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1. Phase II trial of afatinib in patients with advanced urothelial carcinoma with genetic alterations in ERBB1-3 (LUX-Bladder 1)

3. Vitamin D opposes multilineage cell differentiation induced by Notch inhibition and BMP4 pathway activation in human colon organoids

4. List of contributors

8. Genome-wide Association Study of Bladder Cancer Reveals New Biological and Translational Insights

10. GATA6 identifies an immune-enriched phenotype linked to favorable outcomes in patients with pancreatic cancer undergoing upfront surgery

11. A renewed model of pancreatic cancer evolution based on genomic rearrangement patterns

14. An integrated multi-omics analysis identifies prognostic molecular subtypes of non-muscle-invasive bladder cancer

15. The GATA3 X308_Splice breast cancer mutation is a hormone context-dependent oncogenic driver

19. Genome-wide association study identifies multiple susceptibility loci for pancreatic cancer

22. The protective effect of atopic diseases against pancreatic cancer is not driven by Th2-biomarkers

23. Integration of metabolome and microbiome omics layers toward pancreatic cancer risk prediction

24. Complement system genetic variability shapes pancreatic cancer risk

25. Table S1 from Analysis of Several Common APOBEC-type Mutations in Bladder Tumors Suggests Links to Viral Infection

26. Data from Analysis of Several Common APOBEC-type Mutations in Bladder Tumors Suggests Links to Viral Infection

27. Figure S1 from Analysis of Several Common APOBEC-type Mutations in Bladder Tumors Suggests Links to Viral Infection

28. Supplementary Figure S2 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization

29. Supplementary Table S6 from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization

30. Data from Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization

32. PDAC heterogeneity resolved spatially at the single‐cell level: new biological answers, new questions on optimal translation†.

35. Predictive signature of response to neoadjuvant chemotherapy in muscle-invasive bladder cancer integrating mRNA expression, taxonomic subtypes, and clinicopathological features

36. Analysis of Several Common APOBEC-type Mutations in Bladder Tumors Suggests Links to Viral Infection

37. Genetic Susceptibility to Nonalcoholic Fatty Liver Disease and Risk for Pancreatic Cancer: Mendelian Randomization

39. Reply to ‘Mosaic loss of chromosome Y in leukocytes matters’

44. Abstract 6075: Comprehensive genomic characterization of early-stage bladder cancer from 438 patients by whole genome- and exome sequencing

45. Supplementary Figure 2 from Choline Kinase Alpha (CHKα) as a Therapeutic Target in Pancreatic Ductal Adenocarcinoma: Expression, Predictive Value, and Sensitivity to Inhibitors

46. Supplementary Figure 4 from Choline Kinase Alpha (CHKα) as a Therapeutic Target in Pancreatic Ductal Adenocarcinoma: Expression, Predictive Value, and Sensitivity to Inhibitors

47. Supplementary Figure 6 from Choline Kinase Alpha (CHKα) as a Therapeutic Target in Pancreatic Ductal Adenocarcinoma: Expression, Predictive Value, and Sensitivity to Inhibitors

48. Supplementary Figure 1 from Choline Kinase Alpha (CHKα) as a Therapeutic Target in Pancreatic Ductal Adenocarcinoma: Expression, Predictive Value, and Sensitivity to Inhibitors

49. Data from Choline Kinase Alpha (CHKα) as a Therapeutic Target in Pancreatic Ductal Adenocarcinoma: Expression, Predictive Value, and Sensitivity to Inhibitors

50. Supplementary Figure 3 from Choline Kinase Alpha (CHKα) as a Therapeutic Target in Pancreatic Ductal Adenocarcinoma: Expression, Predictive Value, and Sensitivity to Inhibitors

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