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6. Spleen tyrosine kinase inhibition mitigates hemin-induced thromboinflammation in the lung and kidney of sickle cell mice

7. S100A8/A9 drives the formation of procoagulant platelets through GPIbα

8. Illustrated State‐of‐the‐Art Capsules of the ISTH 2022 Congress

11. High factor VIII concentrations interfere with glycoprotein VI-mediated platelet activation in vitro

12. Discrete and conserved inflammatory signatures drive thrombosis in different organs afterSalmonellainfection

13. Hydroxychloroquine inhibits hemolysis-induced arterial thrombosis ex vivoand improves lung perfusion in hemin-treated mice

16. Modelling the pathology and treatment of cardiac fibrosis in vascularised atrial and ventricular cardiac microtissues

19. Functional significance of the platelet immune receptors GPVI and CLEC-2

20. Data from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

21. Supp Table 1 from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

22. Supp Table 2 from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

23. Supp Table 3 from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

24. Supp Table 4 from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

25. Supp Table 5 from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

26. Supplementary Information from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

27. Contributors

28. GPVI and CLEC-2

31. Human Bone Marrow Organoids Enable the Study of Hematopoietic Cell-Stromal Interactions and Support the Survival of Malignant Cells from Patients

32. Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

33. Severity of SARS-CoV-2 infection is associated with high numbers of alveolar mast cells and their degranulation

35. SARS-CoV-2 Spike- and Nucleoprotein-Specific Antibodies Induced After Vaccination or Infection Promote Classical Complement Activation

36. Preferential uptake of SARS-CoV-2 by pericytes potentiates vascular damage and permeability in an organoid model of the microvasculature

38. Complement Factor H Inhibits CD47-Mediated Resolution of Inflammation

40. Platelets and Antiplatelet Medication in COVID-19-Related Thrombotic Complications

41. Adverse Outcome in COVID-19 Is Associated With an Aggravating Hypo-Responsive Platelet Phenotype

42. In vitro, classical complement activation differs by disease severity and between SARS-CoV-2 antigens

44. CLEC-2 Prevents Accumulation and Retention of Inflammatory Macrophages During Murine Peritonitis

45. Stimulation of vascular organoids with SARS-CoV-2 antigens increases endothelial permeability and regulates vasculopathy

47. The podoplanin-CLEC-2 axis inhibits inflammation in sepsis

48. CLEC-2 promotes inflammatory peritoneal macrophage emigration to draining lymph nodes during endotoxemia

49. Post-translational polymodification of β1-tubulin regulates motor protein localization in platelet production and function

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