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178 results on '"Rakitskaya, V."'

2. Improving the Efficiency of Grinding with Cylindrical Flap Wheels of Surfaces with Double Curvature

Author

Vinh, Le Tri, Koltsov, V. P., Rakitskaya, V. B., Cavas-Martínez, Francisco, Series Editor, Chaari, Fakher, Series Editor, di Mare, Francesca, Series Editor, Gherardini, Francesco, Series Editor, Haddar, Mohamed, Series Editor, Ivanov, Vitalii, Series Editor, Kwon, Young W., Series Editor, Trojanowska, Justyna, Series Editor, Radionov, Andrey A., editor, and Gasiyarov, Vadim R., editor

Published
2022
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6. Calculation of Allowance Value for Grinding with Flap Wheels of Shot-Treated Surface to Ensure Required Roughness

Author

Koltsov, V. P., Rakitskaya, V. B., Tardybaeva, E. V., Cavas-Martínez, Francisco, Series Editor, Chaari, Fakher, Series Editor, Gherardini, Francesco, Series Editor, Haddar, Mohamed, Series Editor, Ivanov, Vitalii, Series Editor, Kwon, Young W., Series Editor, Trojanowska, Justyna, Series Editor, di Mare, Francesca, Series Editor, Radionov, Andrey A., editor, and Gasiyarov, Vadim R., editor

Published
2021
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49. [ACTIVITY OF HYPOTHALAMIC-PITUITARY-ADRENAL AXIS OF PRENATALLY STRESSED MALE RATS IN EXPERIMENTAL MODEL OF DEPRESSION].

Author

Ordyan NE, Pivina SG, Rakitskaya VV, and Akulova VK

Subjects
Adrenocorticotropic Hormone metabolism, Animals, Corticosterone metabolism, Corticotropin-Releasing Hormone metabolism, Depressive Disorder metabolism, Female, Hypothalamo-Hypophyseal System physiology, Male, Pituitary-Adrenal System physiology, Pregnancy, Prenatal Exposure Delayed Effects metabolism, Rats, Rats, Wistar, Depressive Disorder physiopathology, Hypothalamo-Hypophyseal System metabolism, Pituitary-Adrenal System metabolism, Prenatal Exposure Delayed Effects physiopathology
Abstract

The hypothalamic-pituitary-adrenal (HPA) axis activity changes were examined in the adult, prenatally stressed male rats in the experimental depression model--the paradigm of "learned helplessness". It was shown that in males descending from intact mothers a depressive-like state was accompanied by an increase in HPA activity. The expression of corticotrophin-releasing hormone (CRH) in the hypothalamic paraventricular nucleus (PVN) increases, coupled with a rise in plasma levels of ACTH and corticosterone as well as in adrenal weight. At the same time in males born from mothers stressed during the last week of pregnancy we observed a decrease in activity of both the central (hypothalamus) and the peripheral (adrenal cortex) parts of regulation of this hormonal axis similar to that revealed for these animals in our previous study in "stress-restress" paradigm. It is concluded that prenatal stress modifies the sensitivity of animals to the inescapable intense stress impact, which manifests itself in a specific pattern of the HPA axis activity after stress load.

Published
2016

50. [STRESS-INDUCED PATTERNS OF THE HYPOTHALAMIC CRH AND VASOPRESSIN EXPRESSION IN FEMALE RATS IN A MODEL OF POSTTRAUMATIC STRESS DISORDER].

Author

Mironova VI, Rakitskaya VV, Pivina SG, and Ordyan NE

Subjects
Animals, Disease Models, Animal, Estrous Cycle, Female, Paraventricular Hypothalamic Nucleus pathology, Rats, Rats, Wistar, Stress Disorders, Post-Traumatic pathology, Stress, Psychological pathology, Corticotropin-Releasing Hormone biosynthesis, Gene Expression Regulation, Paraventricular Hypothalamic Nucleus metabolism, Stress Disorders, Post-Traumatic metabolism, Stress, Psychological metabolism, Vasopressins biosynthesis
Abstract

The neuroendocrine mechanisms underlying anxiety-like state development in cycling female rats with different plasma estradiol levels have been studied in a stress-restress paradigm, an animal model of posttraumatic stress disorder (PTSD). The effect of stress-restress on the hypothalamic expression of corticotropin-releasing hormone (CRH) and vasopressin was analyzed by quantitative immunocytochemistry. Stress-restress was found to increase CRH expression in the hypothalamic paraventricular nucleus (PVN) on the 10th post-restress day, but the level of CRH expression in the PVN restored to the basal values on the 30th post-restress day in all experimental groups. It was shown an increase in vasopressin immunoreactivity in the PVN from the 10th to the 30th post-restress days in female rats exposed to stress during the estrus phase (low plasma estradiol level). In summary, female rats with low plasma estradiol level exhibited the most significant changes in the hypothalamic neuroendocrine system following stress-restress exposure. It might be hypothesized that hyperactivity of the hypothalamic circuit of the central vasopressinergic system is one of the possible mechanisms underlying PTSD-like state development in female rats in a stress-restress paradigm.

Published
2015

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