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63 results on '"Quisqualic Acid toxicity"'

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1. Geranium intoxication induces detoxification enzymes in the Japanese beetle, Popillia japonica Newman.

2. Agrin requires specific proteins to selectively activate γ-aminobutyric acid neurons for pain suppression.

3. Excitotoxic injury to thoracolumbar gray matter alters sympathetic activation and thermal pain sensitivity.

4. Rare excitatory amino acid from flowers of zonal geranium responsible for paralyzing the Japanese beetle.

5. Neuroprotective and anticonvulsant effects of EGIS-8332, a non-competitive AMPA receptor antagonist, in a range of animal models.

6. Spinal and supraspinal changes in tumor necrosis factor-alpha expression following excitotoxic spinal cord injury.

7. Effects of estrone on quisqualate-induced toxicity in primary cultures of rat cortical neurons.

8. The role of excitotoxic injury in post-traumatic syringomyelia.

9. Excitotoxic model of post-traumatic syringomyelia in the rat.

10. Effects of interleukin-10 (IL-10) on pain behavior and gene expression following excitotoxic spinal cord injury in the rat.

11. Selective vulnerability of spinal cord motor neurons to non-NMDA toxicity.

12. Cholinergic and noradrenergic afferents influence the functional properties of the postnatal visual cortex in rats.

13. Neuroprotective effects of interleukin-10 following excitotoxic spinal cord injury.

14. Diffusion-weighted MR imaging in a rat model of syringomyelia after excitotoxic spinal cord injury.

15. Lysosphingomyelin prevents behavioral aberrations and hippocampal neuron loss induced by the metabotropic glutamate receptor agonist quisqualate.

16. Reduced mitochondrial manganese-superoxide dismutase activity exacerbates glutamate toxicity in cultured mouse cortical neurons.

17. Simple and configural association learning in rats with bilateral quisqualic acid lesions of the nucleus basalis magnocellularis.

18. Antagonism of glutamate receptors by a chromatographic fraction from the exudate of the sea anemone Phyllactis flosculifera.

19. Protection against non-NMDA receptor-mediated excitotoxicity by GYKI 52466 in mature telencephalic cultures of the rat.

20. The effect of quisqualic acid-induced lesions of the nucleus basalis magnocellularis on latent inhibition.

21. A defective control of small-amplitude movements in monkeys with globus pallidus lesions: an experimental study on one component of pallidal bradykinesia.

22. Epidermal growth factor and basic fibroblast growth factor protect dopaminergic neurons from glutamate toxicity in culture.

23. Neurotoxicity of acute glutamate transport blockade depends on coactivation of both NMDA and AMPA/Kainate receptors in organotypic hippocampal cultures.

24. Differential brain area vulnerability to long-term subcortical excitotoxic lesions.

25. Transplants to the cerebral cortex of nucleus basalis magnocellularis-lesioned rats: effects on deficits in latent inhibition.

26. Investigations of neurotoxicity and neuroprotection within the nucleus basalis of the rat.

27. Effects of excitotoxic lesions of the septum and vertical limb nucleus of the diagonal band of Broca on conditional visual discrimination: relationship between performance and choline acetyltransferase activity in the cingulate cortex.

28. Re-expression of glia-derived nexin/protease nexin 1 depends on mode of lesion-induction or terminal degeneration: observations after excitotoxin or 6-hydroxydopamine lesions of rat substantia nigra.

29. Inhibitory effects of salmon calcitonin on the tail-biting and scratching behavior induced by substance P and three excitatory amino acids.

30. Mnemonic deficits in the double Y-maze are related to the effects of nucleus basalis injections of ibotenic and quisqualic acid on choline acetyltransferase in the rat amygdala.

31. Neuronal degeneration and spinal cavitation following intraspinal injections of quisqualic acid in the rat.

32. Comparative effects of excitotoxic lesions of the hippocampus and septum/diagonal band on conditional visual discrimination and spatial learning.

33. Selective excitotoxic pathology in the rat hippocampus.

34. The neuroprotective properties of ES-242s, novel NMDA receptor antagonists, in neuronal cell culture toxicity studies.

35. Galanin-immunoreactivity in the nucleus basalis of Meynert in the rat: age-related changes and differential response to lesion-induced cholinergic cell loss.

36. Effects of NBM lesions with two neurotoxins on spatial memory and autoshaping.

37. Glutamate neurotoxicity: a role for non-N-methyl-D-aspartate receptors?

38. Excitatory amino acid neurotoxicity in the developing brain.

39. Quisqualic acid-induced neurotoxicity is protected by NMDA and non-NMDA receptor antagonists.

40. Susceptibility of brain to AMPA induced excitotoxicity transiently peaks during early postnatal development.

41. Differential effect of excitotoxins in the basal forebrain on choline acetyltransferase activity in the cortex and amygdala.

42. Neurophysiological consequences of combined cholinergic and noradrenergic lesions.

43. Mobilization of dantrolene-sensitive intracellular calcium pools is involved in the cytotoxicity induced by quisqualate and N-methyl-D-aspartate but not by 2-amino-3-(3-hydroxy-5-methylisoxazol-4-yl)propionate and kainate in cultured cerebral cortical neurons.

44. Quisqualate neurotoxicity in rat cortical cultures: pharmacology and mechanisms.

45. Loss of cholinergic neurons in the nucleus basalis induces neocortical electroencephalographic and passive avoidance deficits.

46. Attentional functions of the forebrain cholinergic systems: effects of intraventricular hemicholinium, physostigmine, basal forebrain lesions and intracortical grafts on a multiple-choice serial reaction time task.

47. EEG changes induced by acute and chronic quisqualic or ibotenic acid nucleus basalis lesions are stabilized by tacridine.

48. Review: avian models for experimental myopia.

49. Effects of THA on passive avoidance retention performance of intact, nucleus basalis, frontal cortex and nucleus basalis + frontal cortex-lesioned rats.

50. Behavioral impairments after lesions of the nucleus basalis by ibotenic acid and quisqualic acid.

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