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Your search keyword '"Quelvennec, Erwann"' showing total 14 results
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14 results on '"Quelvennec, Erwann"'

2. Investigation of seven proposed regions of linkage in multiple sclerosis: an American and French collaborative study

Author

Pericak-Vance, Margaret A., Rimmler, Jackie B., Haines, Jonathan L., Garcia, Melissa E., Oksenberg, Jorge R., Barcellos, Lisa F., Lincoln, Robin, Hauser, Stephen L., Cournu-Rebeix, Isabelle, Azoulay-Cayla, Ariele, Lyon-Caen, Olivier, Fontaine, Bertrand, Duhamel, Emmanuelle, Coppin, Helene, Brassat, David, Roth, Marie-Paule, Clanet, Michel, Alizadeh, Mehdi, Yaouanq, Jacqueline, Quelvennec, Erwann, Semana, Gilbert, Edan, Gilles, Babron, Marie-Claude, Genin, Emmanuelle, and Clerget-Darpoux, Francoise

Published
2004
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9. HLA‐DRB3/4/5 mismatches are associated with increased risk of acute GVHD in 10/10 matched unrelated donor hematopoietic cell transplantation.

Author

Ducreux, Stéphanie, Dubois, Valérie, Amokrane, Kahina, Yakoub‐Agha, Ibrahim, Labalette, Myriam, Michallet, Mauricette, Rubio, Marie‐Thérèse, Kennel, Anne, Forcade, Edouard, Lafarge, Xavier, Bulabois, Claude‐Eric, Masson, Dominique, Daguindau, Etienne, Devys, Anne, Moalic, Virginie, Quelvennec, Erwann, Boudifa, Abdelali, Picard, Christophe, Van Endert, Peter, and de Matteis, Muriel

Published
2018
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10. Genetic interaction ofCTLA-4 with HLA-DR15 in multiple sclerosis patients

Author

Alizadeh, Mehdi, primary, Babron, Marie-Claude, additional, Birebent, Brigitte, additional, Matsuda, Fumihiko, additional, Quelvennec, Erwann, additional, Liblau, Roland, additional, Cournu-Rebeix, Isabelle, additional, Momigliano-Richiardi, Patricia, additional, Sequeiros, Jorge, additional, Yaouanq, Jacqueline, additional, Genin, Emmanuelle, additional, Vasilescu, Alexandre, additional, Bougerie, H�loise, additional, Trojano, Maria, additional, Martins Silva, Berta, additional, Maciel, Patricia, additional, Clerget-Darpoux, Fran�oise, additional, Clanet, Michel, additional, Edan, Gilles, additional, Fontaine, Bertrand, additional, and Semana, Gilbert, additional

Published
2003
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12. Genetic interaction of CTLA‐4with HLA‐DR15 in multiple sclerosis patients

Author

Alizadeh, Mehdi, Babron, Marie‐Claude, Birebent, Brigitte, Matsuda, Fumihiko, Quelvennec, Erwann, Liblau, Roland, Cournu‐Rebeix, Isabelle, Momigliano‐Richiardi, Patricia, Sequeiros, Jorge, Yaouanq, Jacqueline, Genin, Emmanuelle, Vasilescu, Alexandre, Bougerie, Héloise, Trojano, Maria, Martins Silva, Berta, Maciel, Patricia, Clerget‐Darpoux, Françoise, Clanet, Michel, Edan, Gilles, Fontaine, Bertrand, and Semana, Gilbert

Abstract

Multiple sclerosis is a chronic inflammatory disease of the central nervous system with a genetic component. Until now, the more consistent association with the disease is found with the major histocompatibility complex, especially HLA‐DRB1*1501‐DQB1*0602 haplotype. In this report, we demonstrate the interaction of Cytotoxic T Lymphocyte‐associated antigen 4 (CTLA‐4[CD152]) gene with DRB1*15 haplotype in multiple sclerosis genetic susceptibility. Our data were obtained from two European independent family‐based studies including 610 multiple sclerosis family trios. Ann Neurol 2003;54:119–122

Published
2003
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13. Genetic interaction of <TOGGLE>CTLA-4</TOGGLE> with HLA-DR15 in multiple sclerosis patients<FNR HREF="fn1"></FNR><FN ID="fn1"> Additional members of the Italian Group for MS Genetics and the Portuguese Group for MS Genetics are listed in the Appendix on page 122.</FN>

Author

Alizadeh, Mehdi, Babron, Marie-Claude, Birebent, Brigitte, Matsuda, Fumihiko, Quelvennec, Erwann, Liblau, Roland, Cournu-Rebeix, Isabelle, Momigliano-Richiardi, Patricia, Sequeiros, Jorge, Yaouanq, Jacqueline, Genin, Emmanuelle, Vasilescu, Alexandre, and Bougerie, Héloise

Abstract

Multiple sclerosis is a chronic inflammatory disease of the central nervous system with a genetic component. Until now, the more consistent association with the disease is found with the major histocompatibility complex, especially HLA-DRB1*1501-DQB1*0602 haplotype. In this report, we demonstrate the interaction of Cytotoxic T Lymphocyte-associated antigen 4 (CTLA-4 [CD152]) gene with DRB1*15 haplotype in multiple sclerosis genetic susceptibility. Our data were obtained from two European independent family-based studies including 610 multiple sclerosis family trios. Ann Neurol 2003;54:119–122

Published
2003
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14. Genetic interaction of CTLA-4 with HLA-DR15 in multiple sclerosis patients.

Author

Alizadeh M, Babron MC, Birebent B, Matsuda F, Quelvennec E, Liblau R, Cournu-Rebeix I, Momigliano-Richiardi P, Sequeiros J, Yaouanq J, Genin E, Vasilescu A, Bougerie H, Trojano M, Martins Silva B, Maciel P, Clerget-Darpoux F, Clanet M, Edan G, Fontaine B, and Semana G

Subjects
Abatacept, Antigens, CD, CTLA-4 Antigen, Cohort Studies, DNA Primers genetics, Genetic Predisposition to Disease, HLA-DR Serological Subtypes, Humans, Immunoblotting, Polymerase Chain Reaction, Polymorphism, Genetic genetics, Promoter Regions, Genetic genetics, Antigens, Differentiation genetics, HLA-DR Antigens genetics, Immunoconjugates, Multiple Sclerosis genetics
Abstract

Multiple sclerosis is a chronic inflammatory disease of the central nervous system with a genetic component. Until now, the more consistent association with the disease is found with the major histocompatibility complex, especially HLA-DRB1*1501-DQB1*0602 haplotype. In this report, we demonstrate the interaction of Cytotoxic T Lymphocyte-associated antigen 4 (CTLA-4 [CD152]) gene with DRB1*15 haplotype in multiple sclerosis genetic susceptibility. Our data were obtained from two European independent family-based studies including 610 multiple sclerosis family trios. Ann Neurol 2003;54:119-122

Published
2003
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