17 results on '"Qian, Junliang"'
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2. A PLC communication characteristics‐based fault location method in medium voltage meshed distribution networks
3. Potential convergence of olfactory dysfunction in Parkinson’s disease and COVID-19: The role of neuroinflammation
4. Chemogenetic and optogenetic stimulation of zona incerta GABAergic neurons ameliorates motor impairment in Parkinson’s disease
5. Therapeutic effects of masitinib on abnormal mechanoreception in a mouse model of tourniquet-induced extremity ischemia-reperfusion
6. Chronic morphine-mediated upregulation of high mobility group box 1 in the spinal cord contributes to analgesic tolerance and hyperalgesia in rats
7. A PLC communication characteristics-based fault location method in MV meshed distribution networks
8. Analysis of Intrinsic Stock Value Based on Discounted Cash Flow (DCF) Model -- Take China Construction Bank (601939) as an Example
9. Different responses of skeletal muscles to femoral artery ligation-induced ischemia identified in BABL/c and C57BL/6 mice
10. Toll-like receptor 4-mediated nuclear factor-κB activation in spinal cord contributes to chronic morphine-induced analgesic tolerance and hyperalgesia in rats
11. Hyperbaric oxygen therapy does not alleviate tourniquet-induced acute ischemia-reperfusion injury in mouse skeletal muscles
12. A comparison of acute mouse hindlimb injuries between tourniquet- and femoral artery ligation-induced ischemia-reperfusion
13. Does hyperbaric oxygen pretreatment with 100% oxygen attenuate tourniquet‐induced acute ischemia‐reperfusion injury in mouse hindlimb?
14. The therapeutic effects of masitinib in complex regional pain syndrome in a mouse model of tourniquet‐induced extremity ischemia‐reperfusion
15. Chronic morphine-mediated upregulation of high mobility group box 1 in the spinal cord contributes to analgesic tolerance and hyperalgesia in rats
16. A Comparison of Ischemia‐Reperfusion Injuries Induced by Tourniquet and Femoral Artery Ligation in Mouse Hindlimb
17. CXCL12/CXCR4 signaling-mediated ERK1/2 activation in spinal cord contributes to the pathogenesis of postsurgical pain in rats
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