117 results on '"Pursell, Zachary F"'
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2. Breast cancer cells survive chemotherapy by activating targetable immune-modulatory programs characterized by PD-L1 or CD80
3. Comprehensive Analysis of Hypermutation in Human Cancer
4. Light-strand bias and enriched zones of embedded ribonucleotides are associated with DNA replication and transcription in the human-mitochondrial genome
5. 2167. Mutational Signature Analysis to Predict Drug-resistant Pseudomonas aeruginosa
6. 2769. Treatment of Hypermutator Pseudomonas aeruginosa Induces Multidrug Resistance but Can Be Targeted by Combination Therapy
7. Light-strand bias and enriched zones of embedded ribonucleotides are associated with DNA replication and transcription in the human-mitochondrial genome.
8. DNA Precursor Asymmetries in Mammalian Tissue Mitochondria and Possible Contribution to Mutagenesis through Reduced Replication Fidelity
9. Replication DNA polymerases, genome instability and cancer therapies
10. Data from Cancers from Novel Pole-Mutant Mouse Models Provide Insights into Polymerase-Mediated Hypermutagenesis and Immune Checkpoint Blockade
11. Supplementary Table S3 from Cancers from Novel Pole-Mutant Mouse Models Provide Insights into Polymerase-Mediated Hypermutagenesis and Immune Checkpoint Blockade
12. Supplementary Data Figures 1-8 from Cancers from Novel Pole-Mutant Mouse Models Provide Insights into Polymerase-Mediated Hypermutagenesis and Immune Checkpoint Blockade
13. Supplementary Data from Cancers from Novel Pole-Mutant Mouse Models Provide Insights into Polymerase-Mediated Hypermutagenesis and Immune Checkpoint Blockade
14. RNA-mediated double-strand break repair in human cells
15. The role of the Pseudomonas aeruginosa hypermutator phenotype on the shift from acute to chronic virulence during respiratory infection
16. Abstract 1297: Chemotherapy-induced senescence activates robust, parallel programs of immune checkpoint expression that can be targeted with immunotherapy
17. Abstract 1504: Mouse model and human patient data suggest critical roles for Pten and p53 in suppressing POLE mutant tumor development
18. Yeast DNA Polymerase ε Participates in Leading-Strand DNA Replication
19. Mouse model and human patient data reveal critical roles for Pten and p53 in suppressing POLE mutant tumor development
20. Mouse model and human patient data suggest critical roles for Pten and p53 in suppressing POLE mutant tumor development
21. DNA Polymerase and Mismatch Repair Exert Distinct Microsatellite Instability Signatures in Normal and Malignant Human Cells.
22. Yeast DNA polymerase [epsilon] participates in leading-strand DNA replication
23. DNA Polymerase and Mismatch Repair Exert Distinct Microsatellite Instability Signatures in Normal and Malignant Human Cells
24. Cancers from Novel Pole-Mutant Mouse Models Provide Insights into Polymerase-Mediated Hypermutagenesis and Immune Checkpoint Blockade
25. Mechanisms for High Fidelity DNA Replication
26. DNA Polymerase ε: A Polymerase of Unusual Size (and Complexity)
27. The high fidelity and unique error signature of human DNA polymerase ε
28. POLE Mutation Spectra Are Shaped by the Mutant Allele Identity, Its Abundance, and Mismatch Repair Status
29. Trace amounts of 8-oxo-dGTP in mitochondrial dNTP pools reduce DNA polymerase γ replication fidelity
30. Regulation of B family DNA polymerase fidelity by a conserved active site residue: characterization of M644W, M644L and M644F mutants of yeast DNA polymerase ϵ
31. Bright G-Quadruplex Nanostructures Functionalized with Porphyrin Lanterns
32. POLE proofreading defects: Contributions to mutagenesis and cancer
33. Explosive mutation accumulation triggered by heterozygous human Pol ε proofreading-deficiency is driven by suppression of mismatch repair
34. Author response: Explosive mutation accumulation triggered by heterozygous human Pol ε proofreading-deficiency is driven by suppression of mismatch repair
35. In colonic ρ0 (rho0) cells reduced mitochondrial function mediates transcriptomic alterations associated with cancer
36. Abstract B09: DNA polymerase mutations trigger rapid onset of ultra-hypermutant malignant brain tumors in children with biallelic mismatch repair deficiency
37. Evidence for the Kinetic Partitioning of Polymerase Activity on G-Quadruplex DNA
38. Exonuclease mutations in DNA polymerase epsilon reveal replication strand specific mutation patterns and human origins of replication
39. DNA polymerase ε and its roles in genome stability
40. Human Pol ɛ-dependent replication errors and the influence of mismatch repair on their correction
41. Mismatch Repair Balances Leading and Lagging Strand DNA Replication Fidelity
42. Mismatch repair-independent increase in spontaneous mutagenesis in yeast lacking non-essential subunits of DNA polymerase ε
43. Yeast DNA polymerase epsilon participates in leading-strand DNA replication.
44. Regulation of B family DNA polymerase fidelity by a conserved active site residue : characterization of M644W, M644L and M644F mutants of yeast DNA polymerase epsilon.
45. Human DNA Polymerase ϵ Is Able to Efficiently Extend from Multiple Consecutive Ribonucleotides
46. Mismatch Repair Balances Leading and Lagging Strand DNA Replication Fidelity
47. An active site mutant allele of human Pol ε is a mutator in vitro and in vivo
48. Mismatch Repair–Independent Increase in Spontaneous Mutagenesis in Yeast Lacking Non-Essential Subunits of DNA Polymerase ε
49. Combined hereditary and somatic mutations of replication error repair genes result in rapid onset of ultra-hypermutated cancers.
50. Chapter 4 DNA Polymerase ɛ: A Polymerase of Unusual Size (and Complexity).
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