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3. Loss of Cdkn1a protects against MASLD alone or with alcohol intake by preserving lipid homeostasis

Author

Lamas-Paz, Arantza, Hionides-Gutiérrez, Alejandro, Guo, Feifei, Jorquera, Gonzalo, Morán-Blanco, Laura, Benedé-Ubieto, Raquel, Mesquita, Mariana, Estévez-Vázquez, Olga, Zheng, Kang, Mazariegos, Marina, Vázquez-Ogando, Elena, Blázquez-López, Elena, Asensio, Iris, Mutlu, Beste, Gomez-Santos, Beatriz, Peligros, María Isabel, Vaquero, Javier, Bañares, Rafael, Delgado, Teresa C., Martínez-Chantar, María Luz, Martínez-Naves, Eduardo, Sanz-García, Carlos, Mohamed, Mohamed Ramadan, Tesolato, Sofía, Iniesta, Pilar, Gallego-Durán, Rocío, Maya-Miles, Douglas, Ampuero, Javier, Romero-Gómez, Manuel, Martínez-Alcocer, Ana, Sanfeliu-Redondo, David, Fernández-Iglesias, Anabel, Gracia-Sancho, Jordi, Coll, Mar, Graupera, Isabel, Ginès, Pere, Ciudin, Andrea, Rivera-Esteban, Jesús, Pericàs, Juan M., Ávila, Matías A., Frutos, Maria Dolores, Martínez-Cáceres, Carlos Manuel, Ramos-Molina, Bruno, Aspichueta, Patricia, Puigserver, Pere, Nevzorova, Yulia A., and Cubero, Francisco Javier

Published
2025
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6. Transcriptome-wide analysis of PGC-1α–binding RNAs identifies genes linked to glucagon metabolic action

Author

Tavares, Clint DJ, Aigner, Stefan, Sharabi, Kfir, Sathe, Shashank, Mutlu, Beste, Yeo, Gene W, and Puigserver, Pere

Subjects
Biochemistry and Cell Biology, Biological Sciences, Biotechnology, Genetics, Underpinning research, 2.1 Biological and endogenous factors, 1.1 Normal biological development and functioning, Aetiology, Adenosine Triphosphate, Animals, Calcium-Binding Proteins, Cells, Cultured, Gene Expression Profiling, Gene Expression Regulation, Glucagon, Gluconeogenesis, Glucose, Guanosine Triphosphate, Hepatocytes, Liver, Male, Metabolomics, Mice, Mice, Inbred C57BL, Mitochondrial Membrane Transport Proteins, Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha, Protein Binding, Transcriptome, PGC-1 alpha, RNA binding, glucagon, liver, mitochondria, PGC-1α
Abstract

The peroxisome proliferator-activated receptor gamma coactivator 1-alpha (PGC-1α) is a transcriptional coactivator that controls expression of metabolic/energetic genes, programming cellular responses to nutrient and environmental adaptations such as fasting, cold, or exercise. Unlike other coactivators, PGC-1α contains protein domains involved in RNA regulation such as serine/arginine (SR) and RNA recognition motifs (RRMs). However, the RNA targets of PGC-1α and how they pertain to metabolism are unknown. To address this, we performed enhanced ultraviolet (UV) cross-linking and immunoprecipitation followed by sequencing (eCLIP-seq) in primary hepatocytes induced with glucagon. A large fraction of RNAs bound to PGC-1α were intronic sequences of genes involved in transcriptional, signaling, or metabolic function linked to glucagon and fasting responses, but were not the canonical direct transcriptional PGC-1α targets such as OXPHOS or gluconeogenic genes. Among the top-scoring RNA sequences bound to PGC-1α were Foxo1, Camk1δ, Per1, Klf15, Pln4, Cluh, Trpc5, Gfra1, and Slc25a25 PGC-1α depletion decreased a fraction of these glucagon-induced messenger RNA (mRNA) transcript levels. Importantly, knockdown of several of these genes affected glucagon-dependent glucose production, a PGC-1α-regulated metabolic pathway. These studies show that PGC-1α binds to intronic RNA sequences, some of them controlling transcript levels associated with glucagon action.

Published
2020

13. Cardiomyocyte PGC-1α enables physiological adaptations to endurance exercise through suppression of GDF15 and cardiac atrophy

Author

Khetarpal, Sumeet A., primary, Li, Haobo, additional, Vitale, Tevis, additional, Rhee, James, additional, Grauvogel, Louisa, additional, Castro, Claire, additional, Mittenbühler, Melanie J., additional, Houstis, Nicholas E., additional, Vargas-Castillo, Ariana, additional, Smythers, Amanda L., additional, Liu, Jing, additional, Curtin, Casie, additional, Sprenger, Hans-Georg, additional, Blackmore, Katherine A., additional, Kuznetsov, Alexandra, additional, Freeman, Rebecca, additional, Bogoslavski, Dina, additional, Ellinor, Patrick T., additional, Asnani, Aarti, additional, Dumesic, Phillip A., additional, Puigserver, Pere, additional, Roh, Jason D., additional, Spiegelman, Bruce M., additional, and Rosenzweig, Anthony, additional

Published
2024
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14. Structural basis of respiratory complexes adaptation to cold temperatures

Author

Shin, Young-Cheul, primary, Latorre-Muro, Pedro, additional, Djurabekova, Amina, additional, Zdorevskyi, Oleksii, additional, Bennett, Christopher, additional, Burger, Nils, additional, Song, Kangkang, additional, Xu, Chen, additional, Sharma, Vivek, additional, Liao, Maofu, additional, and Puigserver, Pere, additional

Published
2024
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16. H3K27me3-mediated PGC1[alpha] gene silencing promotes melanoma invasion through WNT5A and YAP

Author

Luo, Chi, Balsa, Eduardo, Perry, Elizabeth A., Liang, Jiaxin, Tavares, Clint D., Vazquez, Francisca, Widlund, Hans R., and Puigserver, Pere

Subjects
Genetic engineering, Epigenetic inheritance, Melanoma, Cancer metastasis, Chromatin, Tumors, Diseases, Health care industry
Abstract

Oncogene-targeted and immune checkpoint the rapies have revolutionized the clinical management of malignant melanoma and now offer hope to patients with advanced disease. Intimately connected to patients' overall clinical risk is whether the initial primary melanoma lesion will metastasize and cause advanced disease, but underlying mechanisms are not entirely understood. A subset of melanomas display heightened peroxisome proliferator-activated receptor y coactivator 1-a (PGC1[alpha]) expression that maintains cell survival cues by promoting mitochondrial function, but also suppresses metastatic spread. Here, we show that PGC1[alpha] expression in melanoma cells was silenced by chromatin modifications that involve promoter H3K27 trimethylation. Pharmacological EZH2 inhibition diminished H3K27me3 histone markers, increased PGC1[alpha] expression, and functionally suppressed invasion within PGC1[alpha]-silenced melanoma cells. Mechanistically, PGC1[alpha] silencing activated transcription factor 12 (TCF12), to increase expression of WNT5A, which in turn stabilized YAP protein levels to promote melanoma migration and metastasis. Accordingly, inhibition of components of this transcription-signaling axis, including TCF12, WNT5A, or YAP, blocked melanoma migration in vitro and metastasis in vivo. These results indicate that epigenetic control of melanoma metastasis involved altered expression of PGC1[alpha] and an association with the inherent metabolic state of the tumor., Introduction Recent breakthroughs in the clinical management of malignant melanoma, which include treatment using BRAF(V600E)-targeted drugs and immune checkpoint-blocking antibodies, now improve survival for patients with advanced stage disease (1). [...]

Published
2020
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18. Nicotinamide N-methyltransferase knockdown protects against diet-induced obesity.

Author

Kraus, Daniel, Yang, Qin, Kong, Dong, Banks, Alexander, Zhang, Lin, Rodgers, Joseph, Pirinen, Eija, Pulinilkunnil, Thomas, Gong, Fengying, Wang, Ya-chin, Cen, Yana, Sauve, Anthony, Asara, John, Peroni, Odile, Monia, Brett, Bhanot, Sanjay, Alhonen, Leena, Puigserver, Pere, and Kahn, Barbara

Subjects
Acetyltransferases, Adipocytes, Adipose Tissue, Adipose Tissue, White, Animals, Diabetes Mellitus, Type 2, Diet, Energy Metabolism, Fatty Liver, Gene Knockdown Techniques, Glucose Intolerance, Glucose Transporter Type 4, Insulin Resistance, Liver, Male, Mice, Mice, Inbred C57BL, NAD, Niacinamide, Nicotinamide N-Methyltransferase, Obesity, Ornithine Decarboxylase, Oxidoreductases Acting on CH-NH Group Donors, S-Adenosylmethionine, Sirtuin 1, Spermine, Thinness, Polyamine Oxidase
Abstract

In obesity and type 2 diabetes, Glut4 glucose transporter expression is decreased selectively in adipocytes. Adipose-specific knockout or overexpression of Glut4 alters systemic insulin sensitivity. Here we show, using DNA array analyses, that nicotinamide N-methyltransferase (Nnmt) is the most strongly reciprocally regulated gene when comparing gene expression in white adipose tissue (WAT) from adipose-specific Glut4-knockout or adipose-specific Glut4-overexpressing mice with their respective controls. NNMT methylates nicotinamide (vitamin B3) using S-adenosylmethionine (SAM) as a methyl donor. Nicotinamide is a precursor of NAD(+), an important cofactor linking cellular redox states with energy metabolism. SAM provides propylamine for polyamine biosynthesis and donates a methyl group for histone methylation. Polyamine flux including synthesis, catabolism and excretion, is controlled by the rate-limiting enzymes ornithine decarboxylase (ODC) and spermidine-spermine N(1)-acetyltransferase (SSAT; encoded by Sat1) and by polyamine oxidase (PAO), and has a major role in energy metabolism. We report that NNMT expression is increased in WAT and liver of obese and diabetic mice. Nnmt knockdown in WAT and liver protects against diet-induced obesity by augmenting cellular energy expenditure. NNMT inhibition increases adipose SAM and NAD(+) levels and upregulates ODC and SSAT activity as well as expression, owing to the effects of NNMT on histone H3 lysine 4 methylation in adipose tissue. Direct evidence for increased polyamine flux resulting from NNMT inhibition includes elevated urinary excretion and adipocyte secretion of diacetylspermine, a product of polyamine metabolism. NNMT inhibition in adipocytes increases oxygen consumption in an ODC-, SSAT- and PAO-dependent manner. Thus, NNMT is a novel regulator of histone methylation, polyamine flux and NAD(+)-dependent SIRT1 signalling, and is a unique and attractive target for treating obesity and type 2 diabetes.

Published
2014

25. Selective Chemical Inhibition of PGC-1α Gluconeogenic Activity Ameliorates Type 2 Diabetes

Author

Sharabi, Kfir, Lin, Hua, Tavares, Clint D.J., Dominy, John E., Camporez, Joao Paulo, Perry, Rachel J., Schilling, Roger, Rines, Amy K., Lee, Jaemin, Hickey, Marc, Bennion, Melissa, Palmer, Michelle, Nag, Partha P., Bittker, Joshua A., Perez, José, Jedrychowski, Mark P., Ozcan, Umut, Gygi, Steve P., Kamenecka, Theodore M., Shulman, Gerald I., Schreiber, Stuart L., Griffin, Patrick R., and Puigserver, Pere

Published
2017
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28. Controversies surrounding peripheral cannabinoid receptor 1 in fatty liver disease

Author

Mutlu, Beste and Puigserver, Pere

Subjects
Health care industry
Abstract

Cannabinoid receptor 1 (CB-1) antagonists are potential candidates for treating obesity and metabolic complications. Despite clear metabolic benefits, unwanted side effects in the brain pose issues for patients. With the hope of overcoming this obstacle, CB-1 in peripheral tissues has become a potential drug target. Previous studies had suggested that liver CB-1 would be an excellent target to prevent development of nonalcoholic steatohepatitis (NAFLD). However, in this issue of the JCI, Wang et al. showed that CB-1 was barely detectable in the liver and deletion of CB-1 in hepatocytes provided no metabolic benefits against NAFLD. These contradictory results raise substantial concerns about the potential benefits of peripheral CB-1 blockers against NAFLD., Sources of liver fat in metabolic diseases NAFLD is a continuum of liver pathologies, usually seen in overweight or obese people, that can be described in three progressive stages: (a) [...]

Published
2021
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30. The Public and Corporate Use of History: Commemorative Books at Catholic Schools.

Author

Moll Bagur, Sergi, Fullana Puigserver, Pere, and Matas Pastor, Joan Josep

Abstract

The present research is framed within the study of Catholic schools and more specifically, in the commemorative books that these centres promoted and published. In order to carry out the study, a total of 34 books have been analysed. They all deal with the history of different Catholic schools in the Balearic Islands (Spain). The involvement of educational community members (students, alumni, teachers, families, ecc.) in the production of the books, together with the institutional and reputational use made of them, link these historiographical products to both the genre of public history and the field of corporate history. In order to understand the nature of these links, as well as the idiosyncrasy of these monographs, this study aims to shed light on the "who", "what" and "how", that is, on the authorship, content and format of the commemorative books in question. The results obtained show that the commemorative books, apart from offering a wealth of useful data regarding the past of Catholic schools, help to better understand the evolution of a historiographical practice that has formed the basis of "new" historiographical genres related to the public and corporate use of history. [ABSTRACT FROM AUTHOR]

Published
2024

31. EL ATHLETIC CLUB EMBAJADOR OFICIOSO ANTE LA SANTA SEDE DURANTE EL PONTIFICADO DE PÍO XII.

Author

FULLANA PUIGSERVER, Pere and Mario DIÉGUEZ, Alejandro

Subjects
PAPACY, RELIGION & politics, INTERNATIONAL competition, ATHLETIC clubs, FRANCOISM
Abstract

Copyright of Materiales para la Historia del Deporte is the property of Polytechnic University of Madrid and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Published
2024
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40. Contributors

Author

Abdel-Wahab, Omar, primary, Abrahm, Janet L., additional, Adams, Sharon, additional, Adewoye, Adeboye H., additional, Allen, Carl, additional, Ambinder, Richard F., additional, Anasetti, Claudio, additional, Anastasi, John, additional, Anderson, Julia A., additional, Antin, Joseph H., additional, Antony, Aśok C., additional, Araten, David J., additional, Armand, Philippe, additional, Armstrong, Gillian, additional, Armstrong, Scott A., additional, Arnold, Donald M., additional, Artz, Andrew S., additional, Awan, Farrukh T., additional, Baglin, Trevor P., additional, Benson, Don M., additional, Benz, Edward J., additional, Berliner, Nancy, additional, Bhagat, Govind, additional, Bhardwaj, Nina, additional, Bhatia, Ravi, additional, Bhatia, Smita, additional, Bhatt, Mihir D., additional, Bhatt, Vijaya Raj, additional, Bitan, Menachem, additional, Blinderman, Craig D., additional, Bollard, Catherine M., additional, Braun, Benjamin S., additional, Brenner, Malcolm K., additional, Brittenham, Gary M., additional, Brodsky, Robert A., additional, Brown, Myles, additional, Broxmeyer, Hal E., additional, Brummel-Ziedins, Kathleen, additional, Brunner, Andrew M., additional, Buadi, Francis K., additional, Burkhardt, Birgit, additional, Burns, Melissa, additional, Byrd, John C., additional, Caimi, Paolo F., additional, Caligiuri, Michael A., additional, Canavan, Michelle, additional, Cantor, Alan B., additional, Carcao, Manuel, additional, Carroll, Michael C., additional, Carty, Shannon A., additional, Castillo, Jorge J., additional, Chan, Anthony K.C., additional, Chapin, John, additional, Chiu, April, additional, Chute, John P., additional, Clark, David B., additional, Coates, Thomas D., additional, Cogle, Christopher R., additional, Connell, Nathan T., additional, Cooke, Elizabeth, additional, Cooley, Sarah, additional, Corradini, Paolo, additional, Creager, Mark A., additional, Creger, Richard J., additional, Cromwell, Caroline, additional, Crowther, Mark A., additional, Cushing, Melissa M., additional, Cutler, Corey, additional, Dang, Chi V., additional, Danial, Nika N., additional, Dave, Sandeep S., additional, DeCaprio, James A., additional, Dinauer, Mary C., additional, Dinner, Shira, additional, Diz-Küçükkaya, Reyhan, additional, Dodd, Roger Y., additional, Donato, Michele L., additional, Dorshkind, Kenneth, additional, Dotti, Gianpietro, additional, Dror, Yigal, additional, Dunleavy, Kieron, additional, Dvorak, Christopher C., additional, Ebert, Benjamin L., additional, Eck, Michael J., additional, Eikelboom, John W., additional, Epperla, Narendranath, additional, Ershler, William B., additional, Evans, William E., additional, Faderl, Stefan, additional, Ferrara, James L.M., additional, Filipovich, Alexandra Hult, additional, Fischer, Martin, additional, Fredenburgh, James C., additional, Friedman, Kenneth D., additional, Fuchs, Ephraim, additional, Fuller, Stephen J., additional, Gailani, David, additional, Galipeau, Jacques, additional, Gallagher, Patrick G., additional, Ganapathi, Karthik A., additional, Gardner, Lawrence B., additional, Gee, Adrian P., additional, Gerson, Stanton L., additional, Gertz, Morie A., additional, Giardina, Patricia J., additional, Gibson, Christopher J., additional, Golan, Karin, additional, Golub, Todd R., additional, Gonzales, Matthew J., additional, Gotlib, Jason, additional, Gottschalk, Stephen, additional, Grant, Marianne A., additional, Graubert, Timothy A., additional, Gregg, Xylina T., additional, Gribben, John G., additional, Gross, Dawn M., additional, Gruber, Tanja A., additional, Guitart, Joan, additional, Gurbuxani, Sandeep, additional, Gur-Cohen, Shiri, additional, Gutierrez, Alejandro, additional, Hamadani, Mehdi, additional, Hari, Parameswaran N., additional, Hartwig, John H., additional, Hayman, Suzanne R., additional, Hayward, Catherine P.M., additional, Hebbel, Robert P., additional, Heslop, Helen E., additional, Hillis, Christopher, additional, Hillyer, Christopher D., additional, Ho, Karin, additional, Hockenbery, David M., additional, Hoffman, Ronald, additional, Hogg, Kerstin E., additional, Holtan, Shernan G., additional, Horny, Hans-Peter, additional, Hsu, Yen-Michael S., additional, Hunter, Zachary R., additional, Huntington, James A., additional, Iancu-Rubin, Camelia, additional, Iqbal, Ali, additional, Isenman, David E., additional, Israels, Sara J., additional, Italiano, Joseph E., additional, Jaffe, Elaine S., additional, Jaffer, Iqbal H., additional, Jagannath, Sundar, additional, Jäger, Ulrich, additional, Jain, Nitin, additional, James, Paula, additional, Jeha, Sima, additional, Jordan, Michael B., additional, Josephson, Cassandra D., additional, Jung, Moonjung, additional, Kager, Leo, additional, Kambayashi, Taku, additional, Kanakry, Jennifer A., additional, Kantarjian, Hagop M., additional, Kaplan, Jason, additional, Karafin, Matthew S., additional, Karsan, Aly, additional, Kaufman, Randal J., additional, Kaufman, Richard M., additional, Keller, Frank G., additional, Kelly, Kara M., additional, Kessler, Craig M., additional, Key, Nigel S., additional, Keyzner, Alla, additional, Khandoga, Alexander G., additional, Khanna-Gupta, Arati, additional, Khatib-Massalha, Eman, additional, Klein, Harvey G., additional, Knoechel, Birgit, additional, Kollet, Orit, additional, Konkle, Barbara A., additional, Kontoyiannis, Dimitrios P., additional, Koreth, John, additional, Koretzky, Gary A., additional, Kotecha, Dipak, additional, Kremyanskaya, Marina, additional, Kumari, Anju, additional, Kuzel, Timothy M., additional, Küppers, Ralf, additional, Lacy, Martha Q., additional, Ladas, Elana, additional, Landier, Wendy, additional, Lapid, Kfir, additional, Lapidot, Tsvee, additional, Larson, Peter J., additional, Levi, Marcel, additional, Lewis, Russell E., additional, Liebman, Howard A., additional, Lillicrap, David, additional, Lim, Wendy, additional, Lin, Judith C., additional, Lindblad, Robert, additional, Lip, Gregory Y.H., additional, Little, Jane A., additional, Lohr, Jens G., additional, López, José A., additional, Luscinskas, Francis W., additional, Maciejewski, Jaroslaw P., additional, Majhail, Navneet S., additional, Manches, Olivier, additional, Mandle, Robert J., additional, Mann, Kenneth G., additional, Manno, Catherine S., additional, Marcogliese, Andrea N., additional, Mariani, Guglielmo, additional, Marincola, Francesco M., additional, Mascarenhas, John, additional, Massberg, Steffen, additional, McEver, Rodger P., additional, McGrath, Emer, additional, McKinney, Matthew S., additional, Mehta, Rohtesh S., additional, Mentzer, William C., additional, Merlini, Giampaolo, additional, Merryman, Reid, additional, Michel, Marc, additional, Migliaccio, Anna Rita, additional, Miller, Jeffrey S., additional, Mims, Martha P., additional, Mondoro, Traci Heath, additional, Moorehead, Paul, additional, Muniz, Luciana R., additional, Munshi, Nikhil C., additional, Najfeld, Vesna, additional, Nayak, Lalitha, additional, Nazy, Ishac, additional, Neff, Anne T., additional, Ness, Paul M., additional, Notarangelo, Luigi D., additional, O'Brien, Sarah H., additional, O'Connor, Owen A., additional, O'Donnell, Martin, additional, Olson, Amanda, additional, Orkin, Stuart H., additional, Pai, Menaka, additional, Pai, Sung-Yun, additional, Paidas, Michael, additional, Panch, Sandhya R., additional, Pande, Reena L., additional, Papayannopoulou, Thalia, additional, Parikh, Rahul, additional, Petersdorf, Effie W., additional, Peterson, Shane E., additional, Pittaluga, Stefania, additional, Ponce, Doris M., additional, Popolo, Laura, additional, Prchal, Josef T., additional, Pui, Ching-Hon, additional, Puigserver, Pere, additional, Rak, Janusz, additional, Ramos, Carlos A., additional, Rand, Jacob H., additional, Rand, Margaret L., additional, Rao, Dinesh S., additional, Ravandi, Farhad, additional, Rawlings, David J., additional, Reddy, Pavan, additional, Reding, Mark T., additional, Reiter, Andreas, additional, Rice, Lawrence, additional, Riese, Matthew J., additional, Ritchey, Arthur Kim, additional, Roberts, David J., additional, Roman, Elizabeth, additional, Rooney, Cliona M., additional, Rosen, Steven T., additional, Rosenthal, David S., additional, Rossmann, Marlies P., additional, Rot, Antal, additional, Rowley, Scott D., additional, Rubnitz, Jeffrey E., additional, Rydz, Natalia, additional, Salama, Mohamed E., additional, Sauk, Steven, additional, Saunthararajah, Yogen, additional, Savage, William, additional, Scadden, David, additional, Schaefer, Kristen G., additional, Schiffman, Fred, additional, Schneidewend, Robert, additional, Schrier, Stanley L., additional, Schuchman, Edward H., additional, Scullion, Bridget Fowler, additional, Selvaggi, Kathy J., additional, Senoo, Keitaro, additional, Shaheen, Montaser, additional, Shaz, Beth H., additional, Shelburne, Samuel A., additional, Shpall, Elizabeth J., additional, Shurin, Susan B., additional, Siegal, Deborah, additional, Silberstein, Leslie E., additional, Silberstein, Lev, additional, Silverstein, Roy L., additional, Sloan, Steven R., additional, Smith, Franklin O., additional, Smith, James W., additional, Smith, Katy, additional, Steensma, David P., additional, Steinberg, Martin H., additional, Stock, Wendy, additional, Storry, Jill R., additional, Stramer, Susan L., additional, Strauss, Ronald G., additional, Stroncek, David F., additional, Taylor, Justin, additional, Thota, Swapna, additional, Treon, Steven P., additional, Tulpule, Anil, additional, Valdes, Roberto Ferro, additional, Valent, Peter, additional, Vedantham, Suresh, additional, Vercellotti, Gregory M., additional, Verneris, Michael R., additional, Vichinsky, Elliott P., additional, von Andrian, Ulrich H., additional, Vose, Julie M., additional, Wagner, Andrew J., additional, Wang, Ena, additional, Wang, Jia-huai, additional, Warkentin, Theodore E., additional, Wasserstein, Melissa P., additional, Webster, Ann, additional, Weisdorf, Daniel J., additional, Weitz, Jeffrey I., additional, Westhoff, Connie M., additional, Wheeler, Allison P., additional, Widick, Page, additional, Wiley, James S., additional, William, Basem M., additional, Williams, David A., additional, Wilson, Wyndham H., additional, Wolfe, Joanne, additional, Wolgast, Lucia R., additional, Wood, Deborah, additional, Wu, Jennifer, additional, Yahalom, Joachim, additional, Yee, Donald L., additional, Younes, Anas, additional, Young, Neal S., additional, and Zeller, Michelle P., additional

Published
2018
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