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140 results on '"Porter, Robert E."'

14. Analyzing Rehearsal Interaction.

Author

Porter, Robert E.

Abstract

A paradigm of rehearsal interaction, based on the measurement and interpretation of behavioral transactions between actors and directors, is posited in this article. Verbal and nonverbal communication processes characteristic of actor/director interactions are operationally defined, and indexes of director/actor interaction, director/actor talk ratio, and director influence ratio are organized according to a general matrix analysis system. Suggestions are given for further investigations to describe and to evaluate the rehearsal process in order to improve the quality of interaction, stimulate the acquisition of effective skills, and encourage positive attitudes. (KS)

Published
1975

23. Specific-pathogen-free Turkey model for reoviral arthritis

Author

Ngunjiri, John M., primary, Ghorbani, Amir, additional, Jang, Hyesun, additional, Waliullah, Sumyya, additional, Elaish, Mohamed, additional, Abundo, Michael C., additional, KC, Mahesh, additional, Taylor, Kara J.M., additional, Porter, Robert E., additional, and Lee, Chang-Won, additional

Published
2019
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24. An Outbreak of Fowl Aviadenovirus A–Associated Gizzard Erosion and Ulceration in Captive Bobwhite Quail (Colinus virginianus).

Author

Kumar, Rahul, Sturos, Matt, Porter, Robert E., Singh, Azad, Armién, Anibal G., Goyal, Sagar M., and Mor, Sunil K.

Subjects
NORTHERN bobwhite, POULTRY, EPITHELIAL cells, EROSION, ADENOVIRUS diseases, TRANSMISSION electron microscopy
Abstract

Copyright of Avian Diseases is the property of American Association of Avian Pathologists, Inc. and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Published
2021
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34. Outbreak of highly pathogenic avian influenza in Minnesota in 2015

Author

Fitzpatrick, Ann, primary, Mor, Sunil K., additional, Thurn, Mary, additional, Wiedenman, Elizabeth, additional, Otterson, Tracy, additional, Porter, Robert E., additional, Patnayak, Devi P., additional, Lauer, Dale C., additional, Voss, Shauna, additional, Rossow, Stephanie, additional, Collins, James E., additional, and Goyal, Sagar M., additional

Published
2017
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48. Select 3-hydroxy-3-methylglutaryl-coenzyme A reductase inhibitors vary in their ability to reduce egg yolk cholesterol levels in laying hens through alteration of the hepatic cholesterol biosynthesis and plasma VLDL composition

Author

Elkin, Robert G., Yan, Zhihong, Zhong, Yuan, Donkin, Shawn S., Buhman, Kimberley K., Story, Jon A., Turek, Jo J., Porter, Robert E., Jr., Anderson, Maureen, Homan, Reynold, and Newton, Roger S.

Subjects
Chickens -- Food and nutrition, Cholesterol -- Research, Eggs -- Cholesterol content, Low density lipoproteins -- Research, Enzyme inhibitors -- Research, Coenzymes -- Research, Food/cooking/nutrition
Abstract

The inability to markedly attenuate cholesterol levels in chicken eggs has led to speculation that cholesterol is essential for yolk formation and that egg production would cease when yolk cholesterol deposition was inadequate for embryonic survival. However, this critical level hypothesis remains unproven. Here, we determine the relative responsiveness of laying hens to three select inhibitors of 3-hydroxy-3-methylglutaryl-coenzyme A reductase (HMGR), the rate-limiting enzyme of cholesterol biosynthesis. A control diet, either alone or supplemented with one of two dietary levels (0.03 or 0.06%) of atorvastatin, lovastatin, or simvastatin, was fed to White Leghorn hens for 5 wk. Liver cholesterol concentrations (mg/g tissue) were decreased (P [less than or equal to] 0.05) by each HMGR inhibitor; however, total liver cholesterol (mg) did not differ among treatments. Microsomal hepatic HMGR activities were increased one- to twofold in all HMGR inhibitor-treated groups, while HMGR mRNA levels were unaffected. Diameters of plasma VLDL particles, the main cholesterol-carrying yolk precursor macromolecules, were reduced (P [less than or equal to] 0.05) only in hens fed 0.06% atorvastatin, and the particles contained 38% less total cholesterol (P [less than or equal to] 0.05) than controls. Plasma total cholesterol concentrations were lowered (P [less than or equal to] 0.05) by both doses of atorvastatin (-56, -63%) and simvastatin (-36,-45%). Egg cholesterol contents were maximally reduced by 46% (P [less than or equal to] 0.05), 7% (P > 0.05), and 22% (P [less than or equal to] 0.05) in hens fed the 0.06% level of atorvastatin, lovastatin, and simvastatin, respectively, while overall egg production [-19% (P [less than or equal to] 0.05), + 4% (P > 0.05), and -3% (P > 0.05)], was much less affected. We concluded that cholesterol per se may not be an obligatory component for yolk formation in chickens and, as such, may be amenable to further pharmacological manipulation. KEY WORDS: chickens; cholesterol; egg; 3-hydroxy-3-methylglutaryl-coenzyme A reductase inhibitors; VLDL

Published
1999

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