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1. Senescence Induced by UVB in Keratinocytes Impairs Amino Acids Balance

4. Supplementary Tables 1 - 5, Figures 1 - 11 from Posttranscriptional Regulation of PER1 Underlies the Oncogenic Function of IREα

5. Data from Posttranscriptional Regulation of PER1 Underlies the Oncogenic Function of IREα

6. Dual IRE1 RNase functions dictate glioblastoma development

9. Dual IRE1 RNase functions dictate glioblastoma development

10. Senescence Induced by UVB in Keratinocytes Impairs Amino Acids Balance

13. The out-of-field dose in radiation therapy induces delayed tumorigenesis by senescence evasion

14. Dual IRE1 RNase functions dictate glioblastoma development

15. Author response: The out-of-field dose in radiation therapy induces delayed tumorigenesis by senescence evasion

22. Dengue virus serotype infection specifies the activation of the unfolded protein response

25. Dual IRE 1 RN ase functions dictate glioblastoma development

26. Protein biosynthesis, a target of sorafenib, interferes with the unfolded protein response (UPR) and ferroptosis in hepatocellular carcinoma cells

28. Endoplasmic reticulum stress induces p53 cytoplasmic localization and prevents p53-dependent apoptosis by a pathway involving glycogen synthase kinase-3 beta

29. Alterations of EDEM1 functions enhance ATF6 pro‐survival signaling.

30. ATF6α regulates morphological changes associated with senescence in human fibroblasts

31. Defective DNA single-strand break repair is responsible for senescence and neoplastic escape of epithelial cells

35. Posttranscriptional Regulation of PER1 Underlies the Oncogenic Function of IREα

37. Autocrine control of glioma cells adhesion/migration through Inositol Requiring enzyme 1α (IRE1α)-mediated cleavage of Secreted Protein Acidic Rich in Cysteine (SPARC) mRNA

50. Identification of a gene signature of a pretransformation process by senescence evasion in normal human epidermal keratinocytes.

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