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4. ACKR3 agonism induces heterodimerization with chemokine receptor CXCR4 and attenuates platelet function.

5. Hemin‐induced platelet activation is regulated by the ACKR3 chemokine surface receptor and has implications for passivation of vulnerable atherosclerotic plaques.

7. Hemin-induced platelet activation is regulated via ACKR3 chemokine surface receptor - implications for passivation of vulnerable atherosclerotic plaque

14. Correction to “Cathepsin-Targeting SARS-CoV-2 Inhibitors: Design, Synthesis, and Biological Activity”

17. Cathepsin-Targeting SARS-CoV-2 Inhibitors: Design, Synthesis, and Biological Activity

25. Multi-omics for COVID-19: driving development of therapeutics and vaccines

34. Discovery and Development of First-in-Class ACKR3/CXCR7 Superagonists for Platelet Degranulation Modulation

35. Small-Molecule Thioesters as SARS-CoV-2 Main Protease Inhibitors: Enzyme Inhibition, Structure–Activity Relationships, Antiviral Activity, and X-ray Structure Determination

37. Small Molecule Thioesters as SARS-CoV-2 Main Protease inhibitors: Enzyme inhibition and mechanism, structure-activity relationship, antiviral activity, and X-ray structure determination

41. Discovery of Polyphenolic Natural Products as SARS-CoV-2 M pro Inhibitors for COVID-19.

44. Involvement of GPR17 in Neuronal Fibre Outgrowth

45. 2‐Substituted thienotetrahydropyridine derivatives: Allosteric ectonucleotidase inhibitors

46. 3CL Protease Inhibitors with an Electrophilic Arylketone Moiety as Anti-SARS-CoV-2 Agents

49. Design, Synthesis, and Unprecedented Interactions of Covalent Dipeptide-Based Inhibitors of SARS-CoV-2 Main Protease and Its Variants Displaying Potent Antiviral Activity

50. Involvement of GPR17 in Neuronal Fibre Outgrowth

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