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4. PD-1H/VISTA mediates immune evasion in acute myeloid leukemia

5. Associations of T-cell fitness prior to B-cell maturation antigen (BCMA)–targeted chimeric antigen receptor T-cell (CART) and bispecific T-cell engager (BiTE) therapies and efficacy/toxicity in relapsed/refractory multiple myeloma (RRMM).

9. PD-1H/VISTA mediates immune evasion in acute myeloid leukemia

13. Sputum Proteomics Reveals a Shift in Vitamin D-binding Protein and Antimicrobial Protein Axis in Tuberculosis Patients

14. Abstract 6100: The Fanconi Anemia pathway protein complex FANCI/FANCD2 couples the DNA damage response to R-loop regulation through SRSF1-mediated mRNA export

15. Supplemental Figures from Fibroblast Subtypes Regulate Responsiveness of Luminal Breast Cancer to Estrogen

16. Supplemental Table 1 from Fibroblast Subtypes Regulate Responsiveness of Luminal Breast Cancer to Estrogen

17. Supplemental Methods from Fibroblast Subtypes Regulate Responsiveness of Luminal Breast Cancer to Estrogen

18. Supplemental Figure Legends from Fibroblast Subtypes Regulate Responsiveness of Luminal Breast Cancer to Estrogen

19. Supplemental Table 2 from Fibroblast Subtypes Regulate Responsiveness of Luminal Breast Cancer to Estrogen

22. Transcription elongation defects link oncogenic splicing factor mutations to targetable alterations in chromatin landscape

23. Rationally designed inhibitors of the Musashi protein-RNA interaction by hotspot mimicry

25. Activation of targetable inflammatory immune signaling is seen in myelodysplastic syndromes with SF3B1 mutations

26. Author response: Activation of targetable inflammatory immune signaling is seen in myelodysplastic syndromes with SF3B1 mutations

29. Integrative genome-wide analysis reveals EIF3A as a key downstream regulator of translational repressor protein Musashi 2 (MSI2)

40. Challenges in the Evaluation and Management of Toxicities Arising From Immune Checkpoint Inhibitor Therapy for Patients With Myeloid Malignancies

50. Multi-Omics Investigation of Innate Navitoclax Resistance in Triple-Negative Breast Cancer Cells

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