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5. Supplementary Figure 2 from Loss of the CBX7 Gene Expression Correlates with a Highly Malignant Phenotype in Thyroid Cancer

6. Supplementary Methods and Figure Legends 1-2 from Loss of the CBX7 Gene Expression Correlates with a Highly Malignant Phenotype in Thyroid Cancer

7. Supplementary Figure 1 from Loss of the CBX7 Gene Expression Correlates with a Highly Malignant Phenotype in Thyroid Cancer

11. Down Syndrome Fetal Fibroblasts Display Alterations of Endosomal Trafficking Possibly due to SYNJ1 Overexpression

14. High-mobility group A1 inhibits p53 by cytoplasmic relocalization of its proapoptotic activator HIPK2

24. OC.4- Dysregulation of autophagy has a potential role in SYNJ1-associated early-onset parkinsonism

26. PERK-Mediated Unfolded Protein Response Activation and Oxidative Stress in PARK20 Fibroblasts

28. Retraction: Overexpression of Proteins HMGA1 Induces Cell Cycle Deregulation and Apoptosis in Normal Rat Thyroid Cells

29. Retraction: Suppression of HMGA2 Protein Synthesis Could Be a Tool for the Therapy of Well Differentiated Liposarcomas Overexpressing HMGA2

33. High mobility group A1 protein modulates autophagy in cancer cells

34. Glix 13, a New Drug Acting on Glutamatergic Pathways in Children and Animal Models of Autism Spectrum Disorders

39. HIPK2 deficiency causes chromosomal instability by cytokinesis failure and increases tumorigenicity

40. Identification of new high mobility group A1 associated proteins

44. Hmga1/Hmga2 double knock-out mice display a “superpygmy” phenotype

47. Convergent Effects of Resveratrol and PYK2 on Prostate Cells.

50. PIT1 upregulation by HMGA proteins has a role in pituitary tumorigenesis

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