151 results on '"Perisic, Ljubica"'
Search Results
2. CDKN2B Regulates TGFβ Signaling and Smooth Muscle Cell Investment of Hypoxic Neovessels.
3. Reactive Oxygen-Forming Nox5 Links Vascular Smooth Muscle Cell Phenotypic Switching and Extracellular Vesicle-Mediated Vascular Calcification
4. Abstract 20471: Local Induction of miR21 Increases Fibrous Cap Stability in Vulnerable Atherosclerotic Lesions
5. Abstract 20282: Lipid Phosphate Phosphatase 3 and Lysophosphatidic Acid Receptors Expression in Human Atherosclerotic Plaques: Co-Localization of Relevant Lipid
6. Abstract 15801: The Long Noncoding RNA H19 Controls Endothelial Cell Aging
7. Abstract 15313: Long Non-Coding RNA MIAT Regulates Smooth Muscle Cell Plasticity and Macrophage Activity in Advanced Atherosclerosis Lesions
8. A systems-approach reveals human nestin is an endothelial-enriched, angiogenesis-independent intermediate filament protein
9. Nexilin/NEXN controls actin polymerization in smooth muscle and is regulated by myocardin family coactivators and YAP
10. Low TLR7 gene expression in atherosclerotic plaques is associated with major adverse cardio- and cerebrovascular events
11. Cyclin-dependent kinase inhibitor 2B regulates efferocytosis and atherosclerosis
12. MP28-09 NEXILIN WHICH IS TARGETED BY MYOCARDIN RELATED TRANSCRIPTION FACTORS LOCALIZES TO DENSE BODIES IN SMOOTH MUSCLE AND CONTROLS ACTIN POLYMERIZATION
13. Genomic profiling of human vascular cells identifies TWIST1 as a causal gene for common vascular diseases
14. Cyclin-dependent kinase inhibitor 2B regulates efferocytosis and atherosclerosis
15. Genetic susceptibility loci for cardiovascular disease and their impact on atherosclerotic plaques
16. Expression of endothelin type B receptors (EDNRB) on smooth muscle cells is controlled by MKL2, ternary complex factors, and actin dynamics
17. Genetic Susceptibility Loci for Cardiovascular Disease and Their Impact on Atherosclerotic Plaques
18. Long non-coding RNA H19 regulates endothelial cell aging via inhibition of STAT3 signalling
19. Expression of endothelin type B receptors (EDNRB) on smooth muscle cells is controlled by MKL2, ternary complex factors, and actin dynamics
20. Expression of endothelin type B receptors (EDNRB) on smooth muscle cells is controlled by ternary complex factors and the actin cytoskeleton
21. Long non-coding RNA H19 regulates endothelial cell aging via inhibition of STAT3 signalling
22. Long non-coding RNA MIAT regulates smooth muscle cell and macrophage activity in atherosclerotic lesions. A role for Lipoprotein(a)/Oxidized phospholipids
23. Therapeutically Targeting the Kinase Activity or Gene Expression of RIP1 Reduces Inflammation-driven Atherosclerosis and Promotes Plaque Stability
24. Expression of endothelin type B receptors (EDNRB) on smooth muscle cells is controlled by ternary complex factors and the actin cytoskeleton
25. miRNA Targeting of Oxysterol Binding Protein-like 6 (OSBPL6) Regulates Cholesterol Trafficking and Efflux
26. Low TLR7 gene expression in atherosclerotic plaques is associated with major adverse cardio- and cerebrovascular events
27. Abstract 13: Inhibition of Nudt6 Stabilizes Advanced Atherosclerotic Plaques
28. Abstract 95: MiRNA-210 Enhances Fibrous Cap Stability in Advanced Atherosclerotic Lesions
29. Abstract 219: Deficiency of the T Cell Regulator Cbl-B Aggravates Atherosclerosis by Inducing CD8 + T Cell-Mediated Macrophage Death
30. Therapeutically inhibiting macrophage necroptosis reduces inflammation‐driven atherosclerosis and promotes plaque stability
31. LowTLR7gene expression in atherosclerotic plaques is associated with major adverse cardio- and cerebrovascular events
32. Long non-coding RNA H19 regulates endothelial cell aging via inhibition of STAT3 signalling.
33. Low TLR7 gene expression in atherosclerotic plaques is associated with major adverse cardio- and cerebrovascular events
34. Functional Analysis of a Novel Genome-Wide Association Study Signal in SMAD3 That Confers Protection From Coronary Artery Disease
35. Integrative functional genomics identifies regulatory mechanisms at coronary artery disease loci
36. Integrative functional genomics identifies regulatory mechanisms at coronary artery disease loci
37. Targeting macrophage necroptosis for therapeutic and diagnostic interventions in atherosclerosis
38. Abstract 173: Proprotein Convertase Subtilisin/Kexin Type 6 is a Key Protease in the Control of Smooth Muscle Cell Function in Vascular Disease
39. Abstract 3: Targeting Macrophage Necroptosis for Therapeutic and Diagnostic Interventions to Treat Atherosclerosis
40. Abstract 19: Role of Adenosine-to-Inosine RNA Editing of Alu Elements in Human Vascular Inflammatory Diseases
41. miRNA Targeting of Oxysterol-Binding Protein-Like 6 Regulates Cholesterol Trafficking and Efflux
42. Abstract 512: The Long Non-coding Rna MIAT Regulates Smooth Muscle Cell Proliferation and Macrophage Activity in Advanced Atherosclerotic Lesions
43. Abstract 127: Induction of miR-21 Increases Fibrous Cap Stability in Vulnerable Atherosclerotic Lesions
44. Functional Analysis of a Novel Genome-Wide Association Study Signal in SMAD3 That Confers Protection From Coronary Artery Disease
45. Targeting macrophage necroptosis for therapeutic and diagnostic interventions to treat atherosclerosis
46. CDKN2B Regulates TGF β Signaling and Smooth Muscle Cell Investment of Hypoxic Neovessels
47. Schip1 Is a Novel Podocyte Foot Process Protein that Mediates Actin Cytoskeleton Rearrangements and Forms a Complex with Nherf2 and Ezrin
48. Macrophage Mitochondrial Energy Status Regulates Cholesterol Efflux and Is Enhanced by Anti-miR33 in Atherosclerosis
49. Coronary Artery Disease Associated Transcription Factor TCF21 Regulates Smooth Muscle Precursor Cells That Contribute to the Fibrous Cap
50. Correction: Schip1 Is a Novel Podocyte Foot Process Protein that Mediates Actin Cytoskeleton Rearrangements and Forms a Complex with Nherf2 and Ezrin
Catalog
Books, media, physical & digital resources
Discovery Service for Jio Institute Digital Library
For full access to our library's resources, please sign in.