30 results on '"Paudyal, Anju"'
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2. Genetic, cellular and molecular defects in mouse mutants with severe neural tube defects
3. Dissecting the Genetic Complexity of Human 6p Deletion Syndromes by Using a Region-Specific, Phenotype-Driven Mouse Screen
4. An Improved Method for Growing Primary Neurons on Electron Microscopy Grids Co-Cultured with Astrocytes
5. Abstract P2005: Loss Of Full-length Mylk3 Causes Dilated Cardiomyopathy Via A MYL2-independent Mechanism
6. Abstract 14037: A Shorter Myosin Light Chain Kinase 3 Isoform Maintains Myosin Light Chain 2 Phosphorylation but Does Not Attenuate the Dilated Cardiomyopathy Seen in C57BL/6N Mice
7. Mylk3null C57BL/6N mice develop cardiomyopathy, whereasNntnull C57BL/6J mice do not
8. Mouse hitchhiker mutants have spina bifida, dorso-ventral patterning defects and polydactyly: identification of Tulp3 as a novel negative regulator of the Sonic hedgehog pathway
9. N-ethyl-N-nitrosourea-induced adaptor protein 2 sigma subunit 1 ( Ap2s1 ) mutations establish Ap2s1 loss-of-function mice
10. Gα11 mutation in mice causes hypocalcemia rectifiable by calcilytic therapy
11. Genetic interactions between planar cell polarity genes cause diverse neural tube defects in mice
12. Cinacalcet corrects hypercalcemia in mice with an inactivating Gα11 mutation
13. The exon junction complex senses energetic stress and regulates contractility and cell architecture in cardiac myocytes
14. Nuclear accumulation of myocyte muscle LIM protein is regulated by heme oxygenase 1 and correlates with cardiac function in the transition to failure
15. The Exon Junction Complex proteins are non‐AMP kinase dependent sensors of energetic stress and regulate contractility and sarcomere structure in cardiac myocytes
16. Abstract 14251: Nuclear Accumulation of Muscle LIM Protein is Regulated by Myocyte Contractility and Correlates With Cardiac Function in the Transition to Heart Failure
17. Interactions between planar cell polarity genes cause diverse neural tube defects
18. Modulation of stretch-induced myocyte remodeling and gene expression by nitric oxide: a novel role for lipoma preferred partner in myofibrillogenesis
19. Scribble is required for normal epithelial cell–cell contacts and lumen morphogenesis in the mammalian lung
20. The mouse Wnt/PCP protein Vangl2 is necessary for migration of facial branchiomotor neurons, and functions independently of Dishevelled
21. Scribble is required for normal lumen morphogenesis in the mammalian lung
22. The novel mouse mutant, chuzhoi, has disruption of Ptk7 protein and exhibits defects in neural tube, heart and lung development and abnormal planar cell polarity in the ear
23. 13-P098 Identification of chuzhoi, a novel mouse mutant of Ptk7 with severe neural tube defects
24. 13-P111 Identification of Tulp3 as a novel negative regulator of the Sonic Hedgehog
25. Cinacalcet corrects hypercalcemia in mice with an inactivating Gα11 mutation
26. Genetic, Cellular and Molecular Defects in Mouse Mutants with Severe Neural Tube Defects
27. Genetic, Cellular and Molecular Defects in Mouse Mutants with Severe Neural Tube Defects
28. Mylk3 null C57BL/6N mice develop cardiomyopathy, whereas Nnt null C57BL/6J mice do not.
29. G α 11 mutation in mice causes hypocalcemia rectifiable by calcilytic therapy.
30. Modulation of stretch-induced myocyte remodeling and gene expression by nitric oxide: a novel role for lipoma preferred partner in myofibrillogenesis.
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