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1. Elevated α-synuclein levels inhibit mitophagic flux

2. Tyrosyl-tRNA synthetase has a noncanonical function in actin bundling

3. Parkinsonism mutations in DNAJC6 cause lipid defects and neurodegeneration that are rescued by Synj1

4. The Alzheimer susceptibility gene BIN1 induces isoform-dependent neurotoxicity through early endosome defects

5. MAPRE2 mutations result in altered human cranial neural crest migration, underlying craniofacial malformations in CSC-KT syndrome

6. Endophilin-B regulates autophagy during synapse development and neurodegeneration

7. Presynaptic Autophagy and the Connection With Neurotransmission

8. A structure of substrate-bound Synaptojanin1 provides new insights in its mechanism and the effect of disease mutations

9. Trapping of Syntaxin1a in Presynaptic Nanoclusters by a Clinically Relevant General Anesthetic

10. Tau association with synaptic vesicles causes presynaptic dysfunction

11. In vivo single-molecule imaging of syntaxin1A reveals polyphosphoinositide- and activity-dependent trapping in presynaptic nanoclusters

12. Loss of Bin1 Promotes the Propagation of Tau Pathology

13. Endophilin-A Deficiency Induces the Foxo3a-Fbxo32 Network in the Brain and Causes Dysregulation of Autophagy and the Ubiquitin-Proteasome System

14. Mitochondrial uncouplers inhibit clathrin-mediated endocytosis largely through cytoplasmic acidification

15. Purification of Soluble Recombinant Human Tau Protein from Bacteria Using Double-tag Affinity Purification

16. Deficiency of parkin and PINK1 impairs age-dependent mitophagy in Drosophila

17. Synaptic Contacts Enhance Cell-to-Cell Tau Pathology Propagation

18. Chronological requirements of TDP-43 function in synaptic organization and locomotive control

19. HDAC6 Is a Bruchpilot Deacetylase that Facilitates Neurotransmitter Release

20. Correction: Publisher Correction: In vivo single-molecule imaging of syntaxin1A reveals polyphosphoinositide- and activity-dependent trapping in presynaptic nanoclusters

21. Erratum: In vivo single-molecule imaging of syntaxin1A reveals polyphosphoinositide- and activity-dependent trapping in presynaptic nanoclusters

23. Human intellectual disability genes form conserved functional modules in Drosophila.

24. Aconitase causes iron toxicity in Drosophila pink1 mutants.

25. Ubiquitin ligase HUWE1 regulates axon branching through the Wnt/β-catenin pathway in a Drosophila model for intellectual disability.

26. Near-infrared 808 nm light boosts complex IV-dependent respiration and rescues a Parkinson-related pink1 model.

27. The yeast complex I equivalent NADH dehydrogenase rescues pink1 mutants.

28. Synaptic mitochondria in synaptic transmission and organization of vesicle pools in health and disease

29. Drosophila NMNAT maintains neural integrity independent of its NAD synthesis activity.

30. Neurodegeneration cell per cell

31. Nano-positioning and tubulin conformation contribute to axonal transport regulation of mitochondria along microtubules

32. Synaptic proteostasis in Parkinson's disease

35. Neuronal identity defines α-synuclein and tau toxicity

36. EndophilinA-dependent coupling between activity-induced calcium influx and synaptic autophagy is disrupted by a Parkinson-risk mutation

37. Neuronal identity defines α-synuclein and tau toxicity

38. Maturation of neuronal AD-tau pathology involves site-specific phosphorylation of cytoplasmic and synaptic tau preceding conformational change and fibril formation

39. EndophilinA-dependent coupling between activity-dependent calcium influx and synaptic autophagy is disrupted by a Parkinson-risk mutation

40. Parkinson mutations in DNAJC6 cause lipid defects and neurodegeneration that are rescued by Synj1

41. Modular generation of cortical, striatal and ventral midbrain progenitor cells v2

42. Excess Lipin enzyme activity contributes to TOR1A recessive disease and DYT-TOR1A dystonia

43. Do we still need animals? Surveying the role of animal-free models in Alzheimer's and Parkinson's disease research

44. Synaptic tau and synaptogyrin‐3 are promising targets to tackle tauopathies

45. A reference human induced pluripotent stem cell line for large-scale collaborative studies

47. The Alzheimer susceptibility gene BIN1 induces isoform-dependent neurotoxicity through early endosome defects

48. Modular generation of cortical, striatal and ventral midbrain progenitor cells v1

49. Lowering Synaptogyrin-3 expression rescues Tau-induced memory defects and synaptic loss in the presence of microglial activation

50. Torsin and NEP1R1-CTDNEP1 phosphatase affect interphase nuclear pore complex insertion by lipid-dependent and lipid-independent mechanisms

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