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16 results on '"Patra, Nabanita"'

8. Contributors

Author

Abd-Elsalam, Kamel A., Abigail, M. Evy Alice, Afreen, Shagufta, Ahmed, Farah K., Ajayi, Fanelwa R., Alghuthaymi, Mousa, Almoammar, Hassan, Ashfaq, Mohammad, Asim-Mansha, Behera, Anindita, Chauhan, Divya, Chen, Xuan, da Silveira, João Vinícios Wirbitzki, Dewangan, Khemchand, Dutta, Sayan Deb, El Hamaky, Ahmed M., Farid-Un-Nisa, Ferreira, Danielle Cristine Mota, Gacem, Mohamed Amine, Gad, Mohamed A., Ganguly, Keya, Giraldo, Gina Alejandra G., Hashim, Ayat F., Hassan, Atef A., Hussain, Tajamal, Iravani, Siavash, Jampílek, Josef, Jebasingh, S. Emmanuel Joshua, Kalia, Anu, Katti, Kattesh V., Kaur, Harleen, Kaur, Harsimran, Kráľová, Katarína, Kurrey, Ramsingh, Li, Huan, Li, Ming-ju, Li, Xinghui, Lim, Ki-Taek, Majeed, Muhammad Irfan, Mali, Suzana, Mangalanagasundari, S., Mansour, Mogda K., Mantovan, Janaína, Marim, Beatriz M., Mittu, Bharti, Murugan, K., Muthu, K., Nadeem, Nimra, Naqvi, Syed Ali Raza, Njobeh, Patrick B., Oraby, Noha H., Padhi, Santwana, Paraliker, Priti, Parvin, Fahmida, Patel, Dinesh K., Patle, Tarun Kumar, Patra, Nabanita, Paulkumar, K., Pelissari, Franciele Maria, Rai, Mahendra, Rajiv, P., Reeta, T. Jesi, Rehaman, Sumayya, Rikta, Sharmin Yousuf, Roberto, Sergio Ruffo, Sayed El Ahl, Rasha M., Sharma, Sat Pal, Shin, Woo-Chul, Shrivas, Kamlesh, Singh, Jagvir, de Souza, Amanda Lélis, Tahir, Noor, Talreja, Neetu, Tareq, Shafi Mohammad, Thatyana, Maxwell, Thipe, Velaphi C., Vanathi, P., Youssef, Khamis, and Zahid, Muhammad

Published
2020
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9. Green synthesis of multi-metallic nanocubes

Author

Patra, Nabanita, primary, Taviti, Ashoka Chary, additional, Sahoo, Anupam, additional, Pal, Abhisek, additional, Beuria, Tushar Kant, additional, Behera, Anindita, additional, and Patra, Srikanta, additional

Published
2017
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11. A novel histone deacetylase (HDAC) inhibitor MHY219 induces apoptosis via up-regulation of androgen receptor expression in human prostate cancer cells

Author

Patra, Nabanita, primary, De, Umasankar, additional, Kim, Tae Hyung, additional, Lee, Young Ju, additional, Ahn, Mee Young, additional, Kim, Nam Deuk, additional, Yoon, Jung Hyun, additional, Choi, Wahn Soo, additional, Moon, Hyung Ryong, additional, Lee, Byung Mu, additional, and Kim, Hyung Sik, additional

Published
2013
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12. Comparisons of polybrominated diphenyl ethers levels in paired South Korean cord blood, maternal blood, and breast milk samples

Author

Kim, Tae Hyung, primary, Bang, Du Yeon, additional, Lim, Hyun Jung, additional, Jin Won, A., additional, Ahn, Mee Young, additional, Patra, Nabanita, additional, Chung, Ki Kyung, additional, Kwack, Seung Jun, additional, Park, Kui Lea, additional, Han, Soon Young, additional, Choi, Wahn Soo, additional, Han, Jung Yeol, additional, Lee, Byung Mu, additional, Oh, Jeong-Eun, additional, Yoon, Jeong-Hyun, additional, Lee, Jaewon, additional, and Kim, Hyung Sik, additional

Published
2012
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13. Functional Role of Phospholipase D (PLD) in Di(2-Ethylhexyl) Phthalate-Induced Hepatotoxicity in Sprague-Dawley Rats

Author

Kim, Na Young, primary, Kim, Tae Hyung, additional, Lee, Ena, additional, Patra, Nabanita, additional, Lee, Jaewon, additional, Shin, Mi Ok, additional, Kwack, Seung Jun, additional, Park, Kui Lea, additional, Han, Soon Young, additional, Kang, Tae Seok, additional, Kim, Seung Hee, additional, Lee, Byung Mu, additional, and Kim, Hyung Sik, additional

Published
2010
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16. Functional Role of Phospholipase D (PLD) in Di(2-Ethylhexyl) Phthalate-Induced Hepatotoxicity in Sprague-Dawley Rats.

Author

Na Young Kim, Tae Hyung Kim, Ena Lee, Patra, Nabanita, Jaewon Lee, Mi Ok Shin, Seung Jun Kwack, Kui Lea Park, Soon Young Han, Tae Seok Kang, Seung Hee Kim, Byung Mu Lee, and Hyung Sik Kim

Subjects
PHOSPHOLIPASES, PHTHALATE esters, HEPATOTOXICOLOGY, LABORATORY rats, HYDROLYSIS, LECITHIN, PHOSPHATIDIC acids, CELL proliferation
Abstract

Phospholipase D (PLD) is an enzyme that catalyzes the hydrolysis of phosphatidyl choline (PC) to generate phosphatidic acid (PA) and choline. PLD is believed to play an important role in cell proliferation, survival signaling, cell transformation, and tumor progression. However, it remains to be determined whether enhanced expression of PLD in liver is sufficient to induce hepatotoxicity. The aim of this study was to investigate the possible role of PLD in di(2-ethylhexyl) phthalate (DEHP)-induced hepatotoxicity in Sprague-Dawley rats. The phthalate, DEHP (500 mg/kg/d), was administered orally, daily to prepubertal rats (4 wk of age, weighing approximately 70-90 g) for 1, 7, or 28 d. In this study, protein expression levels of PLD1/2, peroxisome proliferator-activated receptor (PPAR), and cytochrome P-450 (CYP) were determined by Western blot analysis using specific antibodies. Liver weight was significantly increased in the DEHP treatment groups. Immunohistochemical analysis demonstrated that DEHP produced strong staining of proliferating cell nuclear antigen (PCNA) at 28 d of exposure, suggestive of hepatocyte proliferation. A significant rise in PLD1/2 expression was observed in liver of DEHP-exposed rats after 7 d. Further, PPARα, constitutive androstane receptor (CAR), pregnane X receptor (PXR), and CYP2B1 protein expression levels were markedly elevated in DEHP-treated groups. Our results suggest that DEHP significantly enhanced the expression of PLD, which may be correlated with PPARα-induced hepatotoxicity through a complex interaction with nuclear receptors including CAR and PXR. [ABSTRACT FROM AUTHOR]

Published
2010
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