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2. Blockade of dopamine D3 receptors improves hippocampal synaptic function and rescues age‐related cognitive phenotype.

8. The antineoplastic drug flavopiridol reverses memory impairment induced by Amyloid-ß1-42 oligomers in mice

14. List of Contributors

21. Fluoxetine and Vortioxetine Reverse Depressive-Like Phenotype and Memory Deficits Induced by Aβ1-42 Oligomers in Mice: A Key Role of Transforming Growth Factor-β1

22. Neuromodulatory Action of Picomolar Extracellular Aβ42 Oligomers on Presynaptic and Postsynaptic Mechanisms Underlying Synaptic Function and Memory

25. Role of Amyloid-β and Tau Proteins in Alzheimer’s Disease: Confuting the Amyloid Cascade

26. Role of Amyloid-β and Tau Proteins in Alzheimer’s Disease: Confuting the Amyloid Cascade

28. Fisiologia

33. LTP and memory impairment caused by extracellular Aβ and Tau oligomers is APP-dependent

34. Author response: LTP and memory impairment caused by extracellular Aβ and Tau oligomers is APP-dependent

35. Amyloid-β Peptide Is Needed for cGMP-Induced Long-Term Potentiation and Memory

36. Fluoxetine and Vortioxetine Reverse Depressive-Like Phenotype and Memory Deficits Induced by Aβ1-42 Oligomers in Mice: A Key Role of Transforming Growth Factor-β1.

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42. Extracellular Tau Oligomers Produce An Immediate Impairment of LTP and Memory

43. CL316,243, a β3-adrenergic receptor agonist, induces muscle hypertrophy and increased strength

44. Time-dependent reversal of synaptic plasticity induced by physiological concentrations of oligomeric Aβ42: an early index of Alzheimer’s disease

46. The antineoplastic drug flavopiridol reverses memory impairment induced by Amyloid-ß 1-42 oligomers in mice

50. A novel mechanism for cyclic adenosine monophosphate-mediated memory formation: role of amyloid beta

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