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1. Real-world analysis of ruxolitinib in myelofibrosis: interim results focusing on patients who were naïve to JAK inhibitor therapy treated within the JAKoMo non-interventional, phase IV trial

2. GPRC5C drives branched-chain amino acid metabolism in leukemogenesis

7. Symptom burden profile in myelofibrosis patients with thrombocytopenia: Lessons and unmet needs

12. Oncogenic JAK2V617F causes PD-L1 expression, mediating immune escape in myeloproliferative neoplasms

20. Kidney Dysfunction Is Associated with Thrombosis and Disease Severity in Myeloproliferative Neoplasms: Implications from the German Study Group for MPN Bioregistry

21. Kidney dysfunction is associated with thrombosis and disease severity in myeloproliferative neoplasms: implications from the German Study Group for MPN Bioregistry

33. The ER-overload response: activation of NF-kappa

35. A phase II trial of pegylated interferon (alpha)-2b therapy for polycythemia vera and essential thrombocythemia: feasibility, clinical and biologic effects, and impact on quality of life

37. Tyrosine phosphorylation of IkappaB-alpha, activates NF-kappaB without proteolytic degradation of IkappaB-alpha

38. Activation of transcription factor NF-kappa-B by the adenovirus E3/19K protein requires its ER retention

44. Enhanced expression of the sphingosine-1-phosphate-receptor-3 causes acute myelogenous leukemia in mice

47. Gene mutations and clonal architecture in myelodysplastic syndromes and changes upon progression to acute myeloid leukaemia and under treatment

49. Oncogenic JAK2 V617F causes PD-L1 expression, mediating immune escape in myeloproliferative neoplasms

50. Associations between gender, disease features and symptom burden in patients with myeloproliferative neoplasms : an analysis by the MPN QOL International Working Group

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