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7. Cardiovascular assessment I

Author

Belghith, M., Nouira, S., Lanore, J. J., Mira, J. P., Hamy, I., Renaud, B., Brunet, F., Dall’Ava, J., Dhainaut, J. F., Lovell, A. T., Hanson, G. C., Piccinni, P., Nolli, M. L., Tripepi, A., Rao, T. Polamarasetti, Meneghetti, L., Rossaro, L., Bassi, N., Tedeschi, U., Boccagni, P., Brolese, A., Burra, P., Patrassi, G. M., D’Amico, D. F., Valtier, B., Jardin, F., de Lassence, A., Brun-Ney, D., Dubourg, O., Bourdarias, J. -P., DeMonte, A., Baigorri, F., Bertolissi, M., Joseph, D., Fernàndez, R., Blanch, U., Artigas, A., Kisch, H. W., Leucht, S., Lichtwarck-Aschoff, M., Zimmermann, G., Pfeiffer, U. J., and Blümel, G.

Published
1992
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8. High vs. low doses of low‐molecular‐weight heparin for the treatment of superficial vein thrombosis of the legs: a double‐blind, randomized trial

Author

Girolami, A., Prandoni, P., Marchiori, A., Bernardi, E., Cogo, A., Cuppini, S., Rudelli, G., Alvino, S., Recchia, G., Stragliotto, E., Zerbini, O., Gonsalvi, M., Morelli, P., Bucherini, E., Tormene, D., Mosena, L., Rosi, G., Lu Priore, A., Ambrosio, G., Parisi, R., Bortoluzzi, C., Contavalli, C., Andreozzi, G. M., Verlato, F., Camporese, G., Aluigi, L., Astorino, G., Pagnan, A., Salsa, F., Bullo, C., Visona, A., Lusiani, L., Bonanome, A., Pesavento, R., Patrassi, G. M., Scarano, L., Marcon, L., Faresin, F., Frausini, G., Francolini, G., Imberti, D., Prati, C., Santonastaso, M., Zanatta, N., Viani, M. P., Malacarne, Z., Pinto, A., Prestipino, F., Ghirarduzzi, A., Iotti, M., Cattabiani, M., Todini, A. R., Paiella, M. L., Allegra, C., Carlizza, A., Venco, A., Dentali, F., Scannapieco, G., Villalta, S., and Di Salvo, M. M.

Subjects
Male, medicine.medical_specialty, Superficial vein thrombosis, medicine.drug_class, Premedication, Deep vein, Low molecular weight heparin, Asymptomatic, law.invention, Double-Blind Method, Randomized controlled trial, law, Thromboembolism, medicine, Humans, Saphenous Vein, Low-molecular-weight heparin, Aged, Venous Thrombosis, Leg, Dose-Response Relationship, Drug, Heparin, business.industry, Incidence, Great saphenous vein, Thrombophlebitis, Venous thromboembolism, Hematology, Heparin, Low-Molecular-Weight, Middle Aged, medicine.disease, Thrombosis, Surgery, Treatment Outcome, medicine.anatomical_structure, Female, medicine.symptom, business, medicine.drug
Abstract

In contrast with extensive information on the management of deep vein thrombosis of the lower extremities, little is known on the most appropriate treatment of the superficial vein thrombosis (SVT). In a multicenter, prospective, controlled, double-blind, double-dummy clinical trial, 164 consecutive patients with acute SVT of the great saphenous vein were randomized to receive the s.c. administration of either fixed prophylactic doses (2850 a-Xa IU) or body-weight adjusted therapeutic doses of nadroparin once daily for 1 month. The main study outcome was to compare the rate of asymptomatic and symptomatic extension of SVT and/or venous thromboembolic (VTE) complications during a 3-month follow-up period. Of the 81 patients randomized to the prophylactic doses, seven [8.6%; 95% confidence interval (CI), 3.5-17.0] developed SVT progression or VTE complications as compared with six of the 83 (7.2%; 95% CI, 2.8-15.1) allocated to the treatment group (absolute difference, 1.4; 96% CI, -6.9 to 9.7; P = 0.74). No patient in either group developed major bleeding. Our findings suggest that therapeutic doses of low-molecular-weight heparin, administered for 1 month in patients with SVT of the greater saphenous vein do not improve results obtained by prophylactic doses, administered for the same period, over a 3-month follow-up period.

Published
2005

14. Versicherungsrechtliche Medizin

Author

Weimann, Stern, F., Ziemke, Reichardt, Lochte, Foerster, Friedemann, Adolf, Critchley, Scherer, H. J., Fischer, Ö., Patrassi, G., Panse, Schmitt, Mendel, Kurt, Hessberg, Jendralski, Stiefler, G., Sattler, Holländer, Alfred, Duschl, L., Plenz, Junghanns, Deus, Klages, Fr., Fuss, S., Michael, Holländer, Kalmus, Rosenthal-Deussen, Engelhardt, Oppenheim, M., and Rosenthal-Deussen, Erika

Published
1932
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15. Vergiftungen

Author

Ullmann, K., Panse, Timm, Fraenckel, P., Jakowlew, Elisabeth, Kerl, Wilhelm, Scholz, H., Ehrismann, Wohlwill, Fr., Steidle, H., Engelhardt, Lanke, Wachholz, Strassmann, Gg., Wiethold, Wilcke, Wolff, E. K., Bettzieche, Ritter, Peiser, B., Patrassi, G., Jahnel, Altschul, R., Petri, Else, Feigenbaum, Arieh, Laubender, Walther, Kornfeld, Klix, Fröhlich, A., Capaldi, Grünthal, E., Hallervorden, Eisner, Klieneberger, Zeehuisen, Mendel, Kurt, Janisch-Rašković, Vilma, Lamers, Badt, Giese, Hári, Paul, Zangger, Rosenthal-Deussen, E., Spitta, and Sprinz

Published
1932
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16. Versicherungsrechtliche Medizin

Author

Spiecker, Fraenckel, P., Ziemke, Löwenstein, K., Mendel, Kurt, Goldstein, K., Hager, Patrassi, G., Stern, F., Wartenberg, Sonntag, A., Winkler, G., Weimann, Kayser-Petersen, Kalmus, Autoreferat, Schwarz, Bernstein, Lochte, Mayer, R. L., Roscher, Hook, Zillmer, Feldmann, E., Friedemann, Adolf, Gottstein, G., and Strassmann, G.

Published
1933
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17. Vergiftungen

Author

Schönberg, Strassmann, F., Staub, H., Giese, Lanke, Bálint, Steindorff, Kurt, Lickint, Kornfeld, Scholz, H., Krambach, Patrassi, G., Weiss, Laubender, Walther, Hesse, Wolff, P., Hulst, J. P. L., Salinger, Meyer, A., Ehrismann, Lehmann, Lendle, Oelkers, H. A., Sjövall, Einar, Badt, Prange, Linden, H., Trommsdorff, Hegler, C., Petri, Else, Timm, Gullmann, L., Jagdhold, Reuter, Rosenthal-Deussen, Erika, Friedemann, Adolf, Kalmus, Panse, Katz, Rudolf, and Wilcke

Published
1933
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20. Vergiftungen

Author

Schönberg, Kärber, Ehrismann, Flaschenträger, B., Riebeling, Estler, Wilcke, Weiss, G., Ganter, Vitray, Schaltenbrand, Laehr, Sjövall, Einar, Nissen, Karl, Marx, Neuhaus, C., Critchley, Frick, Pfister, H., Grüninger, Behrens, Heidepriem, Curt, Hulst, J. P. L., Panse, Lochte, Eichler, Higier, Roth, Dittrich, Popoviciu, Virgil, Ruickoldt, Orth, Hans, Weber, v. Neureiter, Fröhlich, A., Patrassi, G., Ruge, Schwarzacher, Scholz, H., Strassmann, G., Lade, Fritz, Rubner, Max H., Klemperer, Weinig, E., Fraenckel, P., Warstadt, Arno, Merkel, Michelsson, G., Bomskov, Ganter, and Hahn, F.

Published
1935
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21. Vergiftungen

Author

Schönberg, Böhmer, Meyer, M., Ganter, Petri, Else, Kalmus, Gluschke, Kornfeld, Schrader, Weissfeiler, Zeitz, Friederike, Steindorff, Kurt, Rosenthal-Deussen, Erika, Feldberg, Peiser, B., Patrassi, G., Lomholt, Svend, Anselmino, K. J., Trendtel, Fleischhacker, Baumgart, Bassia, v. Neureiter, Mendel, Kurt, Flury, Scholz, H., Lehmann, Hans, Wilcke, and Autoreferat

Published
1932
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36. The contact phase of blood coagulation and renin activation in essential hypertension before and after captopril.

Author

PATRASSI, G. M., FALLO, F., MARTINELLI, S., MANTERO, F., BOERI, G., and GIROLAMI, A.

Abstract

The contact phase of blood coagulation in a group of patients suffering from essential hypertension was studied before and after captopril administration. The baseline levels of factor XII, factor XI and plasminogen were significantly higher than in normals and correlated with baseline diastolic blood pressure levels. On the contrary, plasma prekallikrein was not significantly different from normal. These results suggest the presence of a hypercoagulable state in essential hypertension. After captopril administration, factor XII, factor XI and prekallikrein rapidly decreased, perhaps as a consequence of the drug's effect on the vascular endothelial surface. There was no correlation between the changes of active and inactive renin and the changes of prekallikrein and plasminogen levels. Our data do not support the view that factor XII-plasma kallikrein or plasmin dependent pathways are involved in the activation of inactive renin in vivo. Captopril, by provoking rapid pressure changes, appears to be able to affect the clotting system. [ABSTRACT FROM PUBLISHER]

Published
1984
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39. Rheological and fibrinolytic findings in multiple sclerosis.

Author

Brunetti, A, Ricchieri, G L, Patrassi, G M, Girolami, A, and Tavolato, B

Abstract

Blood viscosity indices, fibrinolytic activity and other serum proteins related to haemocoagulation have been studied in 36 patients with multiple sclerosis. The whole blood viscosity was found to be increased in multiple sclerosis. The increase was caused by a decrease in erythrocyte deformability since plasma viscosity and haematocrit were normal. Plasminogen, fibrinogen and alpha 2 antitrypsin levels were found to be lower than normal. Such alterations were not observed in a group of patients with other non-immunological neurological diseases. In the latter group some coagulation indices were even higher than normal. The higher mean age of the pathological controls could explain the observed levels. The abnormalities observed in multiple sclerosis patients are considered to be a consequence of a non-specific activation of the coagulative system in a chronic immunological disease. [ABSTRACT FROM AUTHOR]

Published
1981

40. Heterozygous Protein C Deficiency Associated with Multiple Congenital Hemangiomas--A Case Report.

Author

Simioni, P., Zanon, G., Lazzaro, A. R., Patrassi, G. M., and Girolami, A.

Subjects
PROTEIN C deficiency, HEMANGIOMAS, CIRCULATING anticoagulants, BLOOD-vessel tumors, VENOUS thrombosis, DEFICIENCY diseases
Abstract

Congenital protein C deficiency is described as associated with recurrent thrombotic manifestations. The proband, a fourteen-year-old female, has a history of severe and frequent thrombotic disease, moreover, she presents congenital multiple hemangiomas. Family history was positive for protein C deficiency since the mother of the proposita is also affected and has manifested deep venous thrombophlebitis. Two additional relatives on the maternal side were not available for study but were reported to be symptomatic. None of the family members presented angiomatosis. This case report represents the first description of the association of protein C deficiency with multiple hemangiomas. [ABSTRACT FROM AUTHOR]

Published
1991
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50. Clotting Changes in Two Patients with Longitudinal Sinus Thrombosis

Author

Girolami, A., Pardatscher, K., Scanarini, M., Job, I., and Patrassi, G.

Abstract

Two patients with angiographically proven longitudinal sinus thrombosis were studied from a coagulation point of view. In the first case, marked primary thrombocytosis was found. This patient was treated with aspirin, dipyridamole and radioactive phosphorus. In the second case, fibrinogen and whole blood plasma viscosity were elevated. This patient developed deep vein thrombosis of the left leg a few weeks after the cerebral episode and was treated with coumarin drugs. The importance of a coagulation study in every patient with cerebral vein thrombosis is emphasized.

Published
1980
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