110 results on '"Päth, G"'
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2. Knockdown of Etv5 enhances ROS production and impairs viability in pancreatic beta cells
3. Beta-cell replacement and regeneration: Strategies of cell-based therapy for type 1 diabetes mellitus
4. Human Krüppel-like factor 11 inhibits human proinsulin promoter activity in pancreatic beta cells
5. Effects of a novel corticotropin-releasing-hormone receptor type I antagonist on human adrenal function
6. In vitro (re)programming of human bone marrow stromal cells towards insulin producing phenotypes: O/FRI/2/03
7. The role of interleukin-6 in the human adrenal gland
8. Knockdown of Etv5 enhances nitric oxide production and caspase-independent cell death in pancreatic INS-1E-cells
9. ETV5 knockdown enhances loss of streptozotocin- and palmitate-induced viability in pancreatic INS-1E beta cells
10. Mesenchymal stem cells (MSC) reduce necrosis and apoptosis of pancreatic INS-1E beta cells during alloxan and streptozotocin exposure
11. The T risk allele of TCF7L2 rs 7903146 is associated with impaired insulin secretion rather than insulin resistance: A clinical pilot study in women with a history of gestational diabetes (GDM)
12. Exocrine AR42J acinar cells enhance viability of cocultured endocrine INS-1E beta cells
13. Mesenchymal Stem Cell (MSC)-derived humoral factors promote survival of insulin producing pancreatic β-cells during intrinsic cellular stress via Akt and ERK1/2 signalling
14. Insulinabgabegenauigkeiten von Patch-Pumpen versus Schlauchpumpen – Eine systematische Analyse unter Laborbedingungen
15. Mesenchymal stem cell (MSC)-mediated activation of ERK1/2 signaling does not contribute to maintained survival of cocultured alloxan-treated INS-1E beta cells
16. Effects of the gestational diabetes-risk polymorphism rs10501320 of MADD gene on metabolic phenotype and glucose homeostasis in women with history of gestational diabetes
17. Preserved insulin response of Nupr1-transgenic islets during diabetogenic injury is correlated with reduced secretion of IL-1beta and enhanced secretion of IL-1 receptor antagonist
18. The macrophage migration inhibitory factor (MIF)/CXCR4 axis regulates migration of bone marrow derived mesenchymal stem cells towards pancreatic ß-cells under lipotoxic conditions
19. Type 2 Diabetes mellitus genetic risk variant T in TCF7L2 rs 7903146 in women is associated with an increased risk for the development of gestational diabetes mellitus (GDM) with necessity of insulin therapy
20. The anti-inflammatory protein NUPR1 protects pancreatic islets from diabetogenic lipotoxic injury ex vivo
21. Der mit einer gestörten Insulinbiosynthese assoziierte Polymorphismus rs10501320 im MADD Gen erhöht das Risiko für das Auftreten von Gestationsdiabetes
22. Humoral factors by human bone marrow-derived MSC enhance beta cell proliferation via ERK1/2 signalling
23. The anti-inflammatory protein NUPR1 (p8) is a novel intracellular mediator of pancreatic β-cell protection during diabetogenic stress in vitro and in vivo
24. The insulin-biosynthesis-impairing MADD rs10501320 genetic polymorphism is associated with gestational diabetes in European populations
25. Type 2 Diabetes mellitus genetic risk variant T in TCF7L2 rs7903146 in women is associated with an increased probability of insulin therapy in gestational diabetes mellitus (GDM)
26. p8 is a novel intracellular mediator of pancreatic β-cell protection that preserves insulin secretory function during inflammatory cell stress in vivo
27. The anti-inflammatory protein p8 protects pancreatic islets from diabetogenic lipotoxic injury ex vivo
28. p8 is a novel intracellular mediator of pancreatic beta cell protection that preserves insulin secretory function during inflammatory stress in vivo
29. Humoral factors by bone marrow-derived MSC enhance survival of injured β-cells by preserving AKT signalling
30. Intracellular p8 mediates potent protection of insulin secretory function during inflammatory beta cell stress
31. Bone marrow-derived MSC enhance survival of injured β-cells by preserving Akt signalling via humoral factors
32. Der genetische Typ 2 Diabetes Risikomarker TCF7L2 rs7903146 ist differenziell assoziiert mit dem Auftreten eines Gestationsdiabetes bei deutschen und griechischen Frauen
33. Differenzielle Regulation des humanen Insulinpromotors in β-Zellen und nicht-β-Zellen durch Interaktion von Krüppel-like factor 11 (KLF11) mit dem P300-PDX1-Transaktivierungskomplex
34. Humorale Faktoren aus MSC schützen kokultivierte β-Zellen vor Schädigung durch Alloxan und induzieren Proliferation
35. Verbesserte Glucosetoleranz in transgenen Mäusen mit β-zellspezifischer Überproduktion des Proteins p8 nach Hochfettdiät und Insulitis
36. Mesenchymale Stromazellen aus dem Knochenmark (MSC) reduzieren Streptozotozin (STZ)-induzierte Schädigung von Insulin produzierenden ß-Zellen durch humorale Faktoren in vitro
37. Verbesserte Insulinfunktion unter Hochfettdiät-induzierter Lipotoxizität in transgenen Mäusen mit ektoper ß-Zell-spezifischer p8-Überexpression
38. Polycystisches Ovarsyndrom (PCOS): 5-Jahres Follow up Metabolischer Parameter
39. Protein p8 ist ein Akutfaktor bei diabetischem Gewebestress durch STZ und inhibiert die Apoptose-Effektormoleküle Caspase 3 und 7 in beta-Zellen
40. Das Protein p8 wird akut und temporär in STZ geschädigten Betazellen hochreguliert und verhindert Apoptose durch Inhibition der Caspasen 3 und 7
41. STZ-geschädigte beta-Zellen induzieren Chemokinrezeptoren und Migration in humanen Telomerase-immortalisierten mesenchymalen Knochenmarksstammzellen (hMSC-TERT) in vitro
42. PDX1 is involved in KLF11-induced inhibition of the human insulin promoter in rodent beta-cells
43. Kurzzeit „Lifestyle-Intervention“ im Rahmen einer innerbetrieblichen Gesundheitsförderung verbessert Parameter des Metabolischen Syndroms
44. Stable overexpression of p8 increases survival of INS-1E beta cells in response to streptozotocin (STZ)-induced beta cell damage in vitro
45. KLF11 differentially regulates PDX1-induced activation of the human insulin promoter in rodent beta cells and human non beta cells
46. The stress-related protein p8 protects beta cells from streptozotocin (STZ)-induced cell death in vitro
47. Glucose-dependent expansion of pancreatic beta-cells by the protein p8 in vitro and in vivo
48. Human pancreatic islet-derived precursor cells display mesenchymal stem cell features and differentiation capacity
49. Human Krüppel-like transcription factor 11 (KLF11) and CACCC box mutation inhibit activity of the human proinsulin promoter in pancreatic beta-cells
50. Inhibition of human proinsulin promoter activity by Krueppel-like transcription factor 11 (KLF11) is mediated through functional interactions with PDX-1 and a proximal CACCC box
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