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269 results on '"Osuka, Koji"'

4. Suprapterional keyhole approach for anteromedial skull base lesions: How I do it.

Author

Inomo, Toshiaki, Iwami, Kenichiro, Watanabe, Tadashi, and Osuka, Koji

Subjects
SKULL base, CRANIOTOMY, SCALP, ANGLES
Abstract

Background: For a minimally invasive treatment approach to the anteromedial part of the anterior cranial fossa (ACF), a small incision and craniotomy of the posterolateral part of the ACF are preferable. Method: We described the concept and technique of suprapterional keyhole approach (SPKA), which uses an exoscope and endoscope to treat ACF lesions. Conclusion: The SPKA enables ACF observation from the lateral direction; the endoscope's extended viewing angles enable the observation of the anteromedial part of the ACF, including the bilateral olfactory groove. Facial skin and large scalp incisions are avoided, making this approach efficient for ACF lesions. [ABSTRACT FROM AUTHOR]

Published
2024
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17. Growing skull fracture with an atypical mechanism: a case report

Author

Aoyama, Masahiro, Joko, Masahiro, Niwa, Aichi, Iwami, Kenichiro, Hara, Masahito, Osuka, Koji, and Miyachi, Shigeru

Subjects
pediatric, trauma, growing skull fracture, cranioplasty, duraplasty
Abstract

Growing skull fractures (GSFs) are well-known but rare causes of pediatric head trauma. They generally occur several months after a head injury, and the main lesion is located under the periosteum. We herein report a case involving a 3-month-old boy with GSF that developed by a different mechanism than previously consid-ered. It developed 18 days after the head injury. A large mass containing cerebrospinal fluid and brain tissue was present within the periosteum. A good outcome was obtained with early strategic surgery. Injury to the inner layer of the periosteum and sudden increase in intracranial pressure might be related to GSF in this case.

Published
2020

24. Complement C5 Contributes to Brain Injury After Subarachnoid Hemorrhage

Author

van Dijk, Bart J, Meijers, Joost C M, Kloek, Anne T, Knaup, Veronique L, Rinkel, Gabriel J E, Morgan, B Paul, van der Kamp, Marije J, Osuka, Koji, Aronica, Eleonora, Ruigrok, Ynte M, van de Beek, Diederik, Brouwer, Matthijs, Pekna, Marcela, Hol, Elly M, Vergouwen, Mervyn D I, van Dijk, Bart J, Meijers, Joost C M, Kloek, Anne T, Knaup, Veronique L, Rinkel, Gabriel J E, Morgan, B Paul, van der Kamp, Marije J, Osuka, Koji, Aronica, Eleonora, Ruigrok, Ynte M, van de Beek, Diederik, Brouwer, Matthijs, Pekna, Marcela, Hol, Elly M, and Vergouwen, Mervyn D I

Abstract

Previous studies showed that complement activation is associated with poor functional outcome after aneurysmal subarachnoid hemorrhage (SAH). We investigated whether complement activation is underlying brain injury after aneurysmal SAH (n = 7) and if it is an appropriate treatment target. We investigated complement expression in brain tissue of aneurysmal SAH patients (n = 930) and studied the role of common genetic variants in C3 and C5 genes in outcome. We analyzed plasma levels (n = 229) to identify the functionality of a single nucleotide polymorphism (SNP) associated with outcome. The time course of C5a levels was measured in plasma (n = 31) and CSF (n = 10). In an SAH mouse model, we studied the extent of microglia activation and cell death in wild-type mice, mice lacking the C5a receptor, and in mice treated with C5-specific antibodies (n = 15 per group). Brain sections from aneurysmal SAH patients showed increased presence of complement components C1q and C3/C3b/iC3B compared to controls. The complement component 5 (C5) SNP correlated with C5a plasma levels and poor disease outcome. Serial measurements in CSF revealed that C5a was > 1400-fold increased 1 day after aneurysmal SAH and then gradually decreased. C5a in plasma was 2-fold increased at days 3-10 after aneurysmal SAH. In the SAH mouse model, we observed a ≈ 40% reduction in both microglia activation and cell death in mice lacking the C5a receptor, and in mice treated with C5-specific antibodies. These data show that C5 contributes to brain injury after experimental SAH, and support further study of C5-specific antibodies as novel treatment option to reduce brain injury and improve prognosis after aneurysmal SAH.

Published
2020

25. Complement C5 Contributes to Brain Injury After Subarachnoid Hemorrhage

Author

TN groep Hol, Brain, Neurologie, Circulatory Health, Pathologie, ZL Cerebrovasculaire Ziekten Medisch, Cancer, van Dijk, Bart J, Meijers, Joost C M, Kloek, Anne T, Knaup, Veronique L, Rinkel, Gabriel J E, Morgan, B Paul, van der Kamp, Marije J, Osuka, Koji, Aronica, Eleonora, Ruigrok, Ynte M, van de Beek, Diederik, Brouwer, Matthijs, Pekna, Marcela, Hol, Elly M, Vergouwen, Mervyn D I, TN groep Hol, Brain, Neurologie, Circulatory Health, Pathologie, ZL Cerebrovasculaire Ziekten Medisch, Cancer, van Dijk, Bart J, Meijers, Joost C M, Kloek, Anne T, Knaup, Veronique L, Rinkel, Gabriel J E, Morgan, B Paul, van der Kamp, Marije J, Osuka, Koji, Aronica, Eleonora, Ruigrok, Ynte M, van de Beek, Diederik, Brouwer, Matthijs, Pekna, Marcela, Hol, Elly M, and Vergouwen, Mervyn D I

Published
2020

31. Physical disuse contributes to widespread chronic mechanical hyperalgesia, tactile allodynia, and cold allodynia through neurogenic inflammation and spino-parabrachio-amygdaloid pathway activation

Author

Ohmichi, Yusuke, primary, Ohmichi, Mika, additional, Tashima, Ryoichi, additional, Osuka, Koji, additional, Fukushige, Kaori, additional, Kanikowska, Dominika, additional, Fukazawa, Yugo, additional, Yawo, Hiromu, additional, Tsuda, Makoto, additional, Naito, Munekazu, additional, and Nakano, Takashi, additional

Published
2020
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33. Complement C5 Contributes to Brain Injury After Subarachnoid Hemorrhage

Author

van Dijk, Bart J., primary, Meijers, Joost C.M., additional, Kloek, Anne T., additional, Knaup, Veronique L., additional, Rinkel, Gabriel J.E., additional, Morgan, B. Paul, additional, van der Kamp, Marije J., additional, Osuka, Koji, additional, Aronica, Eleonora, additional, Ruigrok, Ynte M., additional, van de Beek, Diederik, additional, Brouwer, Matthijs, additional, Pekna, Marcela, additional, Hol, Elly M., additional, and Vergouwen, Mervyn D.I., additional

Published
2019
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46. Skull Base Invasion Patterns of Malignant Head and Neck Tumors: A Neurosurgical Perspective.

Author

Iwami, Kenichiro, Fujii, Masazumi, Nishio, Naoki, Maruo, Takashi, Fujimoto, Yasushi, Takanari, Keisuke, Kamei, Yuzuru, Yamada, Masayuki, Ogawa, Tetsuya, Osuka, Koji, and Saito, Kiyoshi

Subjects
HEAD tumors, NECK tumors, SPHENOID bone, TEMPORAL bone, SKULL base, EXTERNAL ear
Abstract

Objective  Craniofacial resection (CFR) and temporal bone resection (TBR) on malignant head and neck tumors (MHNTs) invading skull base require accurate and precise determination of the tumor invasion. We investigated tumor skull base invasion patterns and surgical results in CFR and TBR cases. Methods  We performed either CFR or TBR for 75 selected patients with the possibility of en bloc resection over the period between 2011 and 2018. The medical charts of the selected patients were reviewed. Results  Primary tumor onset site (TOS) groups were: (1) nasal cavity/ethmoid sinus, 20 cases; (2) orbit, 10 cases; (3) maxillary sinus, 28 cases; and (4) external ear/temporomandibular joint, 17 cases. Grades for tumor invasion depth (TID) included: (I) extracranial invasion and skull base bone invasion; (II) extradural invasion; or (III) intradural invasion. Patients in groups 1 and 2 had a significantly higher frequency of grade II and III invasions than patients in groups 3 and 4. The main invasion site was nasal cavity superior wall and ethmoid sinus superior wall for group 1 tumors, orbit superior wall, and lateral skull base sphenoid bone for group 2 and 3 tumors, and lateral skull base temporal bone for group 4 tumors. Positive resection margins represented a significant negative prognostic factor. TID and TOS did not affect skull base margin status. Conclusion  Skull base invasion of MHNTs exhibits certain fixed patterns in sites susceptible to invasion based on the TOS. The frequencies of extradural and intradural invasions differed, indicating the importance for accurate preoperative tumor evaluation. [ABSTRACT FROM AUTHOR]

Published
2021
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47. Physical disuse contributes to widespread chronic mechanical hyperalgesia, tactile allodynia, and cold allodynia through neurogenic inflammation and spino-parabrachio-amygdaloid pathway activation.

Author

Yusuke Ohmichi, Mika Ohmichi, Ryoichi Tashima, Koji Osuka, Kaori Fukushige, Dominika Kanikowska, Yugo Fukazawa, Hiromu Yawo, Makoto Tsuda, Munekazu Naito, Takashi Nakano, Ohmichi, Yusuke, Ohmichi, Mika, Tashima, Ryoichi, Osuka, Koji, Fukushige, Kaori, Kanikowska, Dominika, Fukazawa, Yugo, Yawo, Hiromu, and Tsuda, Makoto

Published
2020
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50. Additional file 1: Table S1. of Interleukin-6, MCP-1, IP-10, and MIG are sequentially expressed in cerebrospinal fluid after subarachnoid hemorrhage

Author

Aichi Niwa, Osuka, Koji, Nakura, Takahiro, Matsuo, Naoki, Takeya Watabe, and Takayasu, Masakazu

Abstract

Correlation coefficients showing the relationships among the levels of interleukin-6 (IL-6), monocyte chemoattractant protein-1 (MCP-1), interferon-Îł-inducible protein-10 (IP-10), and monokine induced by interferon-Îł (MIG). The maximum value of each cytokine or chemokine was adopted as its peak value. (DOC 28 kb)

Published
2016
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