33 results on '"Olgasi, Cristina"'
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2. Factor VIII promotes angiogenesis and vessel stability regulating extracellular matrix proteins
3. Liver gene therapy with intein‐mediated F8 trans‐splicing corrects mouse haemophilia A
4. Current status and new avenues of stem cell-based preclinical and therapeutic approaches in amyotrophic lateral sclerosis
5. Identification and functional characterization of a novel splicing variant in the F8 coagulation gene causing severe hemophilia A
6. Hemostasis and endothelial functionality: the double face of coagulation factors
7. Therapeutic correction of hemophilia A by transplantation of hPSC-derived liver sinusoidal endothelial cell progenitors
8. Efficient and safe correction of hemophilia A by lentiviral vector-transduced BOECs in an implantable device
9. P.168: Biofabrication of a Functional Vascularized Endocrine Pancreas (VEP) for Type 1 Diabetes
10. Factor VIII Is an Endothelial Factor That Promotes Vessel Stability
11. Increase of Palmitic Acid Concentration Impairs Endothelial Progenitor Cell and Bone Marrow–Derived Progenitor Cell Bioavailability: Role of the STAT5/PPARγ Transcriptional Complex
12. Increase of palmitic acid concentration impairs endothelial progenitor cell and bone marrow--derived progenitor cell bioavailability: role of the STAT5/ PPARγ/transcriptional complex
13. Regulatory-Compliant Validation of a Highly Sensitive qPCR for Biodistribution Assessment of Hemophilia A Patient Cells
14. iPSC-Derived Liver Organoids: A Journey from Drug Screening, to Disease Modeling, Arriving to Regenerative Medicine
15. Patient-Specific iPSC-Derived Endothelial Cells Provide Long-Term Phenotypic Correction of Hemophilia A
16. A functional assay for the clinical annotation of genetic variants of uncertain significance in Diamond-Blackfan anemia
17. Identification and functional characterization of a novel splicing variant in the F8coagulation gene causing severe hemophilia A
18. Increase of Palmitic Acid Concentration Impairs Endothelial Progenitor Cell and Bone Marrow-Derived Progenitor Cell Bioavailability: Role of the Signal Transducer and Activator of Transcription 5/Peroxisome Proliferator-Activated Receptor γ Transcriptional Complex
19. La condizione di iperglicemia induce il de-differenziamento delle cellule staminali ottenute da tessuto adiposo viscerale (ASC): nuove prospettive per la terapia cellulare in corso di malattia diabetica
20. La grelina des-acilata (UAG) promuove la rigenerazione muscolare inducendo l’’espansione delle cellule satelliti (SCS) muscolari dopo danno ischemico: ruolo di SOD-2 e miR221/222
21. L’acido Palmitico, mediante l’attivazione del complesso trascrizionale STAT5/PPARgamma, inibisce l’espansione delle EPC: potenziali implicazioni nella patologia vascolare associata al diabete
22. I miR-221 e miR-222 regolando post-trascrizionalmente p27Kip1 e p57Kip2 inibiscono la crescita vascolare mediata dall’iperglicemia
23. Vaccination to kitL: a new approach to inhibit tumor angiogenesis
24. 179. Correcting the Bleeding Phenotype in Hemophilia Ausing Lentivirally FVIII-Corrected Endothelial Cells Differentiated from Hemophilic Induced Pluripotent Stem Cell (iPSC)
25. Ihibition of beta 1 integrin and IL-3R beta common subunit interaction hinders tumor angiogenesis
26. Inhibition of β1 integrin and IL-3Rβ common subunit interaction hinders tumour angiogenesis
27. miR-222 controls neovascularization by regulating STAT5A expression
28. DNA vaccination against membrane-bound Kit ligand: A new approach to inhibiting tumour growth and angiogenesis
29. High glucose via NOX-dependent ROS generation and AKT activity promotes adipose-derived stem cell de-differentiation
30. microRNA-222 Controls Neovascularization by Regulating Signal Transducer and Activator of Transcription 5A Expression
31. miR-221/222 control luminal breast cancer tumor progression by regulating different targets.
32. Efficient and safe correction of hemophilia A by lentiviral vector-transduced BOECs in an implantable device.
33. Regulatory-Compliant Validation of a Highly Sensitive qPCR for Biodistribution Assessment of Hemophilia A Patient Cells.
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