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Seventeen patients less than 12 years old sustained unstable pelvic ring fractures and were treated non-operatively. They were followed for 2 to 25 years. The subjective long-term results depended on the presence or absence of low back pain. This correlated strongly with pelvic asymmetry. Five patients complained about chronic back pain, two were functionally impaired by severe pelvic asymmetry. Healing of an unstable pelvic fracture in malposition with asymmetry causes poor results and must be prevented.

Ligamentous and osseous injuries of the cervical spine during growth are extremely rare and therapeutic principles for such lesions are ill-defined. In order to help solve some diagnostic and therapeutic problems a retrospective study of 30 children was carried out. There were 5 discoligamentous injuries, 5 fractures of the atlas, 1 fracture of the arcus C2, 8 fractures of the odontoid, and 11 vertebral body compression fractures. The injuries were caused by motor vehicle accident in 9, by diving into shallow water in 7, by falling from a height in 7, by a sports accident in 6, and by a direct trauma in 1 patient. Four children died, three of them due to concomitant brain injury. Two patients suffered permanent neurologic disability due to spinal cord trauma. Five patients underwent an operation. Conservative therapy of flexion-compression fractures of the vertebral bodies caused kyphotic deformity. Ventral interbody fusion in two patients had excellent long-term results. All odontoid fractures were treated by applying a plaster cast and healed promptly. Diagnostic problems were mainly caused by the hypermobility of the upper cervical spine.

The etiology of sinus node (SN) dysfunction after orthotopic heart transplantation as assessed by the origin of rhythm, heart rate (HR), and, provided the origin of rhythm was sinus, by corrected sinus node recovery time (CSNRT) was studied in 50 patients. The possible influences on postoperative donor SN function of donor age, recipient age, underlying pretransplant heart disease, pretransplant amiodarone (AMIO) treatment, date of surgery, ischemic time, surgical technique of atrial incision, the use of different cardioplegic solutions during the study period, and rejection were evaluated. The results thus obtained indicate that SN dysfunction is common after cardiac transplantation and pinpoint to a different etiology of transient (restoration of normal SN function within 4 postoperative weeks) and persistent (SN function still impaired at 3 months) SN dysfunction. Of the several demographic and perioperative variables evaluated, only ischemic time had an influence on postoperative SN function in that ischemic times were significantly longer in patients with impaired SN function when compared with patients exhibiting normal SN function (148 +/- 39 min vs. 110.5 +/- 36 min, respectively, P = 0.001). Further stratification according to the duration of SN dysfunction revealed significantly longer ischemic times in patients with transiently impaired SN function only (156.3 +/- 35 min vs. 110.5 +/- 36 min, P = 0.0026). No relation to persistent SN dysfunction of ischemic time (130.5 +/- 36 min vs. 110.5 +/- 36 min, P = ns) or any other factor investigated was found.

We assessed incidence and course of postoperative SN dysfunction in 42 cardiac transplant recipients. RHY, HR and CSNRT were compared in distinguishing between persistent (9 of 42 patients) and transient (11 of 42) SN dysfunction. Persistent SN dysfunction was distinguished by a significantly lower HR (day 14: 46.2 +/- 12 vs 67 +/- 19 bpm; day 21: 40.3 +/- 21 vs 70.8 +/- 16 bpm; p less than 0.05, respectively) and by RHY: postoperative AS (3 of 42) was found only in patients whose SN dysfunction persisted while patients with SR but prolonged CSNRT more often had transient impairment (6 of 42 vs 2 of 42); CSNRT, in contrast, is unlikely to be useful in distinguishing between transient and persistent SN dysfunction since exceptionally long CSNRTs (11,340 ms. 12,080 ms) occurred in patients with both types. Ischemic times were significantly longer in patients with transient impairment when compared with the group with normal SN function (155.1 +/- 36 vs 109.2 +/- 36 min, p less than 0.05) but did not differ significantly in patients with persistent SN dysfunction (124.8 +/- 32 min). Thus, (1) SN dysfunction after cardiac transplantation is common but most often transient; (2) RHY and HR are adequate follow-up parameters and best predictors of long-term SN function; (3) SN dysfunction after transplantation has important clinical implications given the unreliability of lower pacemakers to take over in 3 patients; (4) transient SN dysfunction is related to duration of ischemia during hypothermic preservation whereas pathogenesis of persistent SN dysfunction remains to be elucidated.

With the transvenous approach of atrial leads in 1978 a progressive physiological pacing concept was developed at the Second Surgical Department of the University of Vienna. Using multiprogrammable fully-automatic double-chamber pacers and the technology of biological sensors, the most suitable treatment for any individual patient can be chosen. Our recommendations are DDD stimulation for complete AV block and rate-responsive pacing of the atrium or the ventricle for the Sick-Sinus-Syndrome or atrial fibrillation, respectively. Second choices for AV block are rate-responsive single-chamber pacemakers or simple programmable pacemakers generally.