299 results on '"Oh, S. Paul"'
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2. Bone Marrow-Derived Alk1 Mutant Endothelial Cells and Clonally Expanded Somatic Alk1 Mutant Endothelial Cells Contribute to the Development of Brain Arteriovenous Malformations in Mice.
3. BMP10 functions independently from BMP9 for the development of a proper arteriovenous network
4. Recent Advances in Basic Research for Brain Arteriovenous Malformation.
5. Pazopanib may reduce bleeding in hereditary hemorrhagic telangiectasia
6. Recent Advances in Basic Research for Brain Arteriovenous Malformation
7. Persistent infiltration and pro-inflammatory differentiation of monocytes cause unresolved inflammation in brain arteriovenous malformation
8. VEGFR2 Expression Correlates with Postnatal Development of Brain Arteriovenous Malformations in a Mouse Model of Type I Hereditary Hemorrhagic Telangiectasia
9. Correcting Smad1/5/8, mTOR, and VEGFR2 treats pathology in hereditary hemorrhagic telangiectasia models
10. TMEM100 is a key factor for specification of lymphatic endothelial progenitors
11. SnoN facilitates ALK1–Smad1/5 signaling during embryonic angiogenesis
12. Reduced Mural Cell Coverage and Impaired Vessel Integrity After Angiogenic Stimulation in the Alk1-deficient Brain
13. Reduced activin receptor-like kinase 1 activity promotes cardiac fibrosis in heart failure
14. Chapter 41 - Molecular Biology and Novel Treatments of Intracranial AVMs
15. 475 Local Conditional Induction of Brain Arteriovenous Malformations in Alk1-Inducible Knock-Out Mice
16. Overexpression of Activin Receptor-Like Kinase 1 in Endothelial Cells Suppresses Development of Arteriovenous Malformations in Mouse Models of Hereditary Hemorrhagic Telangiectasia
17. Supplementary Figure 2 from Tnk1/Kos1 Knockout Mice Develop Spontaneous Tumors
18. Supplementary Figure 3 from Tnk1/Kos1 Knockout Mice Develop Spontaneous Tumors
19. Supplementary Figure 5 from Tnk1/Kos1 Knockout Mice Develop Spontaneous Tumors
20. Supplementary Figure Legends 1-6 from Tnk1/Kos1 Knockout Mice Develop Spontaneous Tumors
21. Supplementary Figure 6 from Tnk1/Kos1 Knockout Mice Develop Spontaneous Tumors
22. Supplementary Figure 1 from Tnk1/Kos1 Knockout Mice Develop Spontaneous Tumors
23. Supplementary Figure 4 from Tnk1/Kos1 Knockout Mice Develop Spontaneous Tumors
24. Increasing brain angiotensin converting enzyme 2 activity decreases anxiety-like behavior in male mice by activating central Mas receptors
25. Highlights of the 13th International Hereditary Hemorrhagic Telangiectasia Scientific conference
26. TMEM100, a Lung-Specific Endothelium Gene
27. BMP10 functions independently from BMP9 for the development of a proper arteriovenous network
28. Conditional knockout of activin like kinase-1 (ALK-1) leads to heart failure without maladaptive remodeling
29. Emerging pathogenic mechanisms in human brain arteriovenous malformations: a contemporary review in the multiomics era
30. Novel experimental model of brain arteriovenous malformations using conditional Alk1 gene deletion in transgenic mice
31. CXCL12-CXCR4 signalling plays an essential role in proper patterning of aortic arch and pulmonary arteries
32. Bone morphogenetic protein-9 inhibits lymphatic vessel formation via activin receptor-like kinase 1 during development and cancer progression
33. Angiotensin-converting enzyme 2 inhibits high-mobility group box 1 and attenuates cardiac dysfunction post-myocardial ischemia
34. Adenine Nucleotide Translocase 4 Is Expressed Within Embryonic Ovaries and Dispensable During Oogenesis
35. Genetics and Emerging Therapies for Brain Arteriovenous Malformations
36. Suppression of BMP signaling by PHD2 deficiency in Pulmonary Arterial hypertension
37. VEGF neutralization can prevent and normalize arteriovenous malformations in an animal model for hereditary hemorrhagic telangiectasia 2
38. Restraint of angiogenesis by zinc finger transcription factor CTCF-dependent chromatin insulation
39. Contributors
40. Mitochondrial ATP transporter depletion protects mice against liver steatosis and insulin resistance
41. TGF-β signaling in endothelial cells, but not neuroepithelial cells, is essential for cerebral vascular development
42. Bone Marrow-Derived Alk1 Mutant Endothelial Cells and Clonally Expanded Somatic Alk1 Mutant Endothelial Cells Contribute to the Development of Brain Arteriovenous Malformations in Mice
43. ALK5- and TGFBR2-independent role of ALK1 in the pathogenesis of hereditary hemorrhagic telangiectasia type 2
44. Growth differentiation factor 11 signaling controls retinoic acid activity for axial vertebral development
45. Real-time imaging of de novo arteriovenous malformation in a mouse model of hereditary hemorrhagic telangiectasia
46. Dysregulation of intestinal crypt cell proliferation and villus cell migration in mice lacking Kruppel-like factor 9
47. A murine model for human Sepiapterin-reductase deficiency
48. Change in gene expression subsequent to induction of Pnn/DRS/memA: increase in p21cip1/waf1
49. Nonoverlapping expression patterns of ALK1 and ALK5 reveal distinct roles of each receptor in vascular development
50. Activin receptor patterning of foregut organogenesis
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