990 results on '"Oh, Goo Taeg"'
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2. Pancreatic β-cell mitophagy as an adaptive response to metabolic stress and the underlying mechanism that involves lysosomal Ca2+ release
3. A resource of targeted mutant mouse lines for 5,061 genes
4. Complexation of drug and hapten-conjugated aptamer with universal hapten antibody for pancreatic cancer treatment
5. miR-125a-5p attenuates macrophage-mediated vascular dysfunction by targeting Ninjurin1
6. The antioxidant enzyme Peroxiredoxin-1 controls stroke-associated microglia against acute ischemic stroke
7. Ninjurin1 deficiency differentially mitigates colorectal cancer induced by azoxymethane and dextran sulfate sodium in male and female mice.
8. Peroxiredoxin 3 Deficiency Exacerbates DSS-Induced Acute Colitis via Exosomal miR-1260b-Mediated Barrier Disruption and Proinflammatory Signaling.
9. Peroxiredoxin 3 deficiency induces cardiac hypertrophy and dysfunction by impaired mitochondrial quality control
10. Single-cell transcriptomics reveal cellular diversity of aortic valve and the immunomodulation by PPARγ during hyperlipidemia
11. 2′–5′ oligoadenylate synthetase‑like 1 (OASL1) protects against atherosclerosis by maintaining endothelial nitric oxide synthase mRNA stability
12. ER-associated CTRP1 regulates mitochondrial fission via interaction with DRP1
13. Autophagy induction promotes renal cyst growth in polycystic kidney disease
14. SOD1 suppresses pro-inflammatory immune responses by protecting against oxidative stress in colitis
15. CD137 Signaling Regulates Acute Colitis via RALDH2-Expressing CD11b−CD103+ DCs
16. Deficiency of peroxiredoxin 2 exacerbates angiotensin II-induced abdominal aortic aneurysm
17. Oxidized LDL induces vimentin secretion by macrophages and contributes to atherosclerotic inflammation
18. CORTICOSTERONE PROTECTS AGAINST STROKE DAMAGE BY ELEVATING PRDX1 ANTIOXIDANT LEVELS
19. IMPROVED CARDIOPROTECTION IN CARDIOVASCULAR DISEASE BY MYELOID-PCSK9 DEFICIENCY BEYOND LDL-C
20. Mitochondrial H2O2 Is a Central Mediator of Diclofenac-Induced Hepatocellular Injury
21. Current pharmacotherapies for atherosclerotic cardiovascular diseases
22. The adipokine Retnla deficiency increases responsiveness to cardiac repair through adiponectin-rich bone marrow cells
23. Intragenic CpG islands play important roles in bivalent chromatin assembly of developmental genes
24. Anti-Inflammatory Actions of Soluble Ninjurin-1 Ameliorate Atherosclerosis
25. Ninjurin1 positively regulates osteoclast development by enhancing the survival of prefusion osteoclasts
26. N-α-acetyltransferase 10 (NAA10) in development: the role of NAA10
27. Indoleamine 2,3-Dioxygenase-Expressing Aortic Plasmacytoid Dendritic Cells Protect against Atherosclerosis by Induction of Regulatory T Cells
28. C1q/TNF-α–Related Protein 1 (CTRP1) Maintains Blood Pressure Under Dehydration Conditions
29. Mitochondrial H 2 O 2 Is a Central Mediator of Diclofenac-Induced Hepatocellular Injury.
30. Progerinin, an Inhibitor of Progerin, Alleviates Cardiac Abnormalities in a Model Mouse of Hutchinson–Gilford Progeria Syndrome
31. Supplementary Methods and Materials, Tables 1-4, Figure Legends 1-3 from Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis
32. Supplementary Figure 3 from Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis
33. Supplementary Figure 1 from Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis
34. Supplementary Figure 5 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
35. Supplementary Figure 2 from Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis
36. Supplementary Materials and Methods, Figure Legends 1-7, Table 1 Legend from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
37. Data from Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis
38. Data from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
39. Supplementary Figure 3 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
40. Supplementary Figure 4 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
41. Supplementary Figure 6 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
42. Supplementary Figure 2 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
43. Supplementary Figure 7 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
44. Supplementary Figure 1 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
45. Supplementary Table 1 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
46. Negligible effect of eNOS palmitoylation on fatty acid regulation of contraction in ventricular myocytes from healthy and hypertensive rats
47. Unbalanced Redox With Autophagy in Cardiovascular Disease
48. Endothelial PTP4A1 mitigates vascular inflammation via USF1/A20 axis-mediated NF-κB inactivation
49. Ninjurin1 Enhances the Basal Motility and Transendothelial Migration of Immune Cells by Inducing Protrusive Membrane Dynamics
50. Hypoxia-Induced Endothelial Progenitor Cell Function Is Blunted in Angiotensinogen Knockout Mice
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