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2. The Human Tumor Atlas Network: Charting Tumor Transitions across Space and Time at Single-Cell Resolution.

3. Loss of epigenetic information as a cause of mammalian aging

4. Correction: The SIRT1 Deacetylase Suppresses Intestinal Tumorigenesis and Colon Cancer Growth

6. Loss of epigenetic information as a cause of mammalian aging

9. The Making of a PreCancer Atlas: Promises, Challenges, and Opportunities

16. Dietary Restriction: Standing Up for Sirtuins [With Response]

19. CTCF-promoted RNA polymerase II pausing links DNA methylation to splicing

20. Loss of Epigenetic Information as a Cause of Mammalian Aging

23. DNA Break-Induced Epigenetic Drift as a Cause of Mammalian Aging

24. Erosion of the Epigenetic Landscape and Loss of Cellular Identity as a Cause of Aging in Mammals

25. The Making of a PreCancer Atlas: Promises, Challenges, and Opportunities

26. SIRT1 Activation Disrupts Maintenance of Myelodysplastic Syndrome Stem and Progenitor Cells by Restoring TET2 Function

27. The Making of a PreCancer Atlas: Promises, Challenges, and Opportunities

29. Replication Stress Shapes a Protective Chromatin Environment across Fragile Genomic Regions

30. TET2 Activity Is Modulated By SIRT1-Mediated Protein Deacetylation: A Potential Therapeutic Target in Myelodysplastic Syndrome

31. The Making of a PreCancer Atlas: Promises, Challenges, and Opportunities: (Trends in Cancer 4, 523–536, 2018)

34. Activation of SIRT1 Deacetylase As a Therapeutic Approach for Myelodysplastic Syndromes By Restoring TET2 Function

38. Ubiquitin-specific protease 21 stabilizes BRCA2 to control DNA repair and tumor growth.

43. Efficiency of RNA interference in the mouse hematopoietic system varies between cell types and developmental stages

47. SIRT1 Redistribution on Chromatin Promotes Genomic Stability but Alters Gene Expression during Aging

48. The SIRT1 Deacetylase Suppresses Intestinal Tumorigenesis and Colon Cancer Growth

50. A BRCA1-interacting lnc RNA regulates homologous recombination.

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