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1. Late Outcome after Necrosectomy

Author

Nordback, I. H., Sand, J., Beger, Hans G., editor, Matsuno, Seiki, editor, Cameron, John L., editor, Rau, Bettina M., editor, Sunamura, Makoto, editor, and Schulick, Richard D., editor

Published
2008
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2. Diagnostic strategy and timing of intervention in infected necrotizing pancreatitis: an international expert survey and case vignette study

Author

van Grinsven, Janneke, van Brunschot, Sandra, Bakker, Olaf J., Bollen, Thomas L., Boermeester, Marja A., Bruno, Marco J., Dejong, Cornelis H., Dijkgraaf, Marcel G., van Eijck, Casper H., Fockens, Paul, van Goor, Harry, Gooszen, Hein G., Horvath, Karen D., van Lienden, Krijn P., van Santvoort, Hjalmar C., Besselink, Marc G., Abdelhafez, M, Andersson, R, Andren-Sandberg, A, Ashley, S, van Baal, M, Baron, T, Bassi, C, Bradley, E, Buchler, M, Cappendijk, V, Carter, R, Charnley, R, Coelho, D, Connor, S, Dellinger, P, Dervenis, C, Deviere, J, Doctor, N, Dudeja, V, En-qiang, M, Escourrou, J, Fagenholz, P, Farkas, G, Forsmark, C, Freeman, M, Freeny, P, French, J, Friess, H, Gardner, T, Goetzinger, P, Haveman, J, Hofker, S, Imrie, C, Isaji, S, Isenmann, R, Klar, E, Laméris, J, Lerch, M, Lévy, P, Lillemoe, K, Löhr, M, Mayerle, J, Mayumi, T, Mittal, A, Moessner, J, Morgan, D, Mortele, K, Nealon, W, Neoptolemos, J, Nieuwenhuijs, V, Nordback, I, Olah, A, Oppong, K, Padbury, R, Papachristou, G, Parks, R, Poley, J, Radenkovic, D, Raraty, M, Rau, B, Rebours, V, Rische, S, Runzi, M, Sainani, N, Sarr, M, Schaapherder, S, Seewald, S, Seifert, H, Shimosegawa, T, Silverman, S, Singh, V, Siriwardena, A, Steinberg, W, Sutton, R, Takeda, K, Timmer, R, Vege, S, Voermans, R, de Waele, J, Wang, Ch, Warshaw, A, Werner, J, Weusten, B, Whitcomb, D, Wig, J, Windsor, J, and Zyromski, N

Published
2015
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16. Immediate effect of vagotomy on pancreatic insulin secretion

Author

Nordback, I. and Harju, E.

Subjects
Gastrectomy -- Complications, Vagus nerve, Pancreatic beta cells -- Physiological aspects, Pancreas -- Physiological aspects, Health
Abstract

Total gastrectomy, or removal of the stomach, is a surgical procedure performed for a variety of disease conditions. Complications that frequently occur following gastrectomy are thought to result, at least in part, from abnormal carbohydrate absorption and metabolism. Animal studies indicate that the vagus nerve has important stimulatory effects on the secretion of insulin (the major hormone involved in the metabolism of carbohydrates). Some of the alterations in glucose handling seen in gastrectomy patients may result from damage to the vagus nerve during surgery. To evaluate this theory, glucagon-stimulated insulin secretion was measured in 11 patients with total gastrectomy and intact vagus nerves, and in 10 patients with total gastrectomy and total subdiaphragmatic vagotomy (severing all branches of the vagus nerve below the diaphragm). Glucagon-stimulated insulin response was measured twice in each subject at the time of surgery; once following induction of anesthesia before any resection had occurred, and 60 to 90 minutes later, when either gastrectomy or gastrectomy plus vagotomy had been accomplished. Patients with gastrectomy alone showed a 48 percent increase in C-peptide (an insulin precursor) concentration; vagotomized patients showed only a 13 percent increase. These results indicate that some of the abnormalities found in gastrectomy patients in whom the vagus nerve has been compromised (either intentionally or unintentionally) may result from damage to this nerve. (Consumer Summary produced by Reliance Medical Information, Inc.)

Published
1991

17. Diagnostic strategy and timing of intervention in infected necrotizing pancreatitis: an international expert survey and case vignette study

Author

van Grinsven, Janneke, primary, van Brunschot, Sandra, additional, Bakker, Olaf J., additional, Bollen, Thomas L., additional, Boermeester, Marja A., additional, Bruno, Marco J., additional, Dejong, Cornelis H., additional, Dijkgraaf, Marcel G., additional, van Eijck, Casper H., additional, Fockens, Paul, additional, van Goor, Harry, additional, Gooszen, Hein G., additional, Horvath, Karen D., additional, van Lienden, Krijn P., additional, van Santvoort, Hjalmar C., additional, Besselink, Marc G., additional, Abdelhafez, M., additional, Andersson, R., additional, Andren-Sandberg, A., additional, Ashley, S., additional, van Baal, M., additional, Baron, T., additional, Bassi, C., additional, Bradley, E., additional, Buchler, M., additional, Cappendijk, V., additional, Carter, R., additional, Charnley, R., additional, Coelho, D., additional, Connor, S., additional, Dellinger, P., additional, Dervenis, C., additional, Deviere, J., additional, Doctor, N., additional, Dudeja, V., additional, En-qiang, M., additional, Escourrou, J., additional, Fagenholz, P., additional, Farkas, G., additional, Forsmark, C., additional, Freeman, M., additional, Freeny, P., additional, French, J., additional, Friess, H., additional, Gardner, T., additional, Goetzinger, P., additional, Haveman, J., additional, Hofker, S., additional, Imrie, C., additional, Isaji, S., additional, Isenmann, R., additional, Klar, E., additional, Laméris, J., additional, Lerch, M., additional, Lévy, P., additional, Lillemoe, K., additional, Löhr, M., additional, Mayerle, J., additional, Mayumi, T., additional, Mittal, A., additional, Moessner, J., additional, Morgan, D., additional, Mortele, K., additional, Nealon, W., additional, Neoptolemos, J., additional, Nieuwenhuijs, V., additional, Nordback, I., additional, Olah, A., additional, Oppong, K., additional, Padbury, R., additional, Papachristou, G., additional, Parks, R., additional, Poley, J., additional, Radenkovic, D., additional, Raraty, M., additional, Rau, B., additional, Rebours, V., additional, Rische, S., additional, Runzi, M., additional, Sainani, N., additional, Sarr, M., additional, Schaapherder, S., additional, Seewald, S., additional, Seifert, H., additional, Shimosegawa, T., additional, Silverman, S., additional, Singh, V., additional, Siriwardena, A., additional, Steinberg, W., additional, Sutton, R., additional, Takeda, K., additional, Timmer, R., additional, Vege, S., additional, Voermans, R., additional, de Waele, J., additional, Wang, Ch., additional, Warshaw, A., additional, Werner, J., additional, Weusten, B., additional, Whitcomb, D., additional, Wig, J., additional, Windsor, J., additional, and Zyromski, N., additional

Published
2016
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18. Classification of acute pancreatitis--2012: revision of the Atlanta classification and definitions by international consensus

Author

Acosta, Jm, Amann, St, Andren Sandberg, A, Aranha, Gv, Asciutti, S, Banks, Pa, Barauskas, G, Baron, Th, Bassi, Claudio, Behrman, S, Behms, Ke, Belliappa, V, Berzin, Tm, Besselink, Mg, Bhasin, Dk, Biankin, A, Bishop, Md, Bollen, Tl, Bonini, Cj, Bradley, El, Buechler, M, Carter, Michael Ross, Cavestro, Gm, Chari, St, Chavez Rodriguez, Jj, da Cunha, Je, D'Agostino, H, De Campos, T, Delakidis, S, de Madaria, E, Deprez, Ph, Dervenis, C, Disario, Ja, Doria, C, Falconi, Massimo, Fernandez del Castillo, C, Freeny, Pc, Frey, Cf, Friess, H, Frossard, Jl, Fuchshuber, P, Gallagher, Sf, Gardner, Tb, Garg, Pk, Ghattas, G, Glasgow, R, Gonzalez, Ja, Gooszen, Hg, Gress, Tm, Gumbs, Aa, Halliburton, C, Helton, S, Hill, Mc, Horvath, Kd, Hoyos, S, Imrie, Cw, Isenmann, R, Izbicki, Jr, Johnson, Cd, Karagiannis, Ja, Klar, E, Kolokythas, O, Lau, J, Litvin, Aa, Longnecker, Ds, Lowenfels, Ab, Mackey, R, Mah'Moud, M, Malangoni, M, Mcfadden, Dw, Mishra, G, Moody, Fg, Morgan, De, Morinville, V, Mortele, Kj, Neoptolemos, Jp, Nordback, I, Pap, A, Papachristou, Gi, Parks, R, Pedrazolli, S, Pelaez Luna, M, Pezzilli, R, Pitt, Ha, Prosanto, C, Ramesh, H, Ramirez, Fc, Raper, Se, Rasheed, A, Reed, Dn, Romangnuolo, J, Rossaak, J, Sanabria, J, Sarr, Mg, Schaefer, C, Schmidt, J, Schmidt, Pn, Serrablo, A, Senkowski, Ck, Sharma, M, Sigman, Km, Singh, P, Stefanidis, G, Steinberg, W, Steiner, J, Strasberg, S, Strum, W, Takada, T, Tanaka, M, Thoeni, Rf, Tsiotos, Gg, Van Santvoort, H, Vaccaro, M, Vege, Ss, Villavicencio, Rl, Vrochides, D, Wagner, M, Warshaw, Al, Wilcox, Cm, Windsor, Ja, Wysocki, P, Yadav, D, Zenilman, Me, Zyromski, N. j., Banks, P, Bollen, T, Dervenis, C, Gooszen, H, Johnson, C, Sarr, M, Tsiotos, G, Vege, S, Cavestro, GIULIA MARTINA, and ACUTE PANCREATITIS CLASSIFICATION WORKING, Group

Subjects
Clinical deffinitions, medicine.medical_specialty, CIENCIAS MÉDICAS Y DE LA SALUD, Exacerbation, MEDLINE, Medicina Clínica, Disease, Guideline, Severity of Illness Index, Atlanta classification, Cystogastrostomy, purl.org/becyt/ford/3.2 [https], Severity of illness, medicine, Humans, Acute Disease, Disease Progression, Pancreatitis, Tomography, X-Ray Computed, Ranson criteria, Intensive care medicine, Tomography, business.industry, Gastroenterology, medicine.disease, Acute pancreatitis, X-Ray Computed, Surgery, Evaluation of complex medical interventions [NCEBP 2], purl.org/becyt/ford/3 [https], Medicina Critica y de Emergencia, business
Abstract

Background and objective: The Atlanta classification of acute pancreatitis enabled standardised reporting of research and aided communication between clinicians. Deficiencies identified and improved understanding of the disease make a revision necessary. Methods: A web-based consultation was undertaken in 2007 to ensure wide participation of pancreatologists. After an initial meeting, the Working Group sent a draft document to 11 national and international pancreatic associations. This working draft was forwarded to all members. Revisions were made in response to comments, and the web-based consultation was repeated three times. The final consensus was reviewed, and only statements based on published evidence were retained. Results: The revised classification of acute pancreatitis identified two phases of the disease: early and late. Severity is classified as mild, moderate or severe. Mild acute pancreatitis, the most common form, has no organ failure, local or systemic complications and usually resolves in the first week. Moderately severe acute pancreatitis is defined by the presence of transient organ failure, local complications or exacerbation of co-morbid disease. Severe acute pancreatitis is defined by persistent organ failure, that is, organ failure >48 h. Local complications are peripancreatic fluid collections, pancreatic and peripancreatic necrosis (sterile or infected), pseudocyst and walled-off necrosis (sterile or infected). We present a standardised template for reporting CT images. Conclusions: This international, web-based consensus provides clear definitions to classify acute pancreatitis using easily identified clinical and radiologic criteria. The wide consultation among pancreatologists to reach this consensus should encourage widespread adoption. Fil: Banks, Peter A.. Harvard Medical School; Estados Unidos Fil: Bollen, Thomas L.. St Antonius Hospital; Países Bajos Fil: Dervenis, Christos. Agia Olga Hospital; Grecia Fil: Gooszen, Hein G.. Radboud Universiteit Nijmegen; Países Bajos Fil: Johnson, Colin D.. University Hospital Southampton; Reino Unido Fil: Sarr, Michael G.. Mayo Clinic; Estados Unidos Fil: Tsiotos, Gregory G.. Metropolitan Hospital; Grecia Fil: Vege, Santhi Swaroop. Metropolitan Hospital; Grecia Fil: Vaccaro, Maria Ines. Consejo Nacional de Investigaciones Científicas y Técnicas; Argentina Fil: Acute Pancreatitis Classification Working Group. No especifica

Published
2013

19. IAP/APA evidence-based guidelines for the management of acute pancreatitis

Author

Besselink, M, van Santvoort, H, Freeman, M, Gardner, T, Mayerle, J, Vege, Ss, Werner, J, Banks, P, Mckay, C, Fernandez del Castillo, C, French, J, Gooszen, H, Johnson, C, Sarr, M, Takada, T, Windsor, J, Saluja, A, Liddle, R, Papachristou, G, Singh, V, Rünzi, M, Wu, B, Bollen, T, Morgan, D, Mortele, K, Mittal, A, En qiang, M, de Waele, J, Petrov, M, Dellinger, P, Lerch, Mm, Anderson, R, Mcclave, S, Hartwig, W, Bruno, Mariasole, Oria, A, Baron, T, Castillo, Cf, Fagenholz, P, Horvath, K, van Baal, M, Nealon, W, Andren Sandberg, A, Bakker, O, Bassi, Claudio, Buchler, M, Boermeester, M, Bradley, E, Chari, S, Charnley, R, Connor, S, Dervenis, C, Deviere, J, Dudeja, V, Fockens, P, Forsmark, C, Friess, H, Isaji, S, Isenmann, R, Klar, E, Lévy, P, Lillemoe, K, Liu, X, Löhr, Cm, Mayumi, T, Mossner, J, Neoptolemos, J, Nordback, I, Olah, Anett, Padbury, R, Parks, R, Radenkovic, D, Rau, B, Rebours, V, Seewald, S, Seifert, H, Shimosegawa, T, Siriwardena, A, Steinberg, W, Sutton, R, Tanaka, M, Takeda, K, Tse, F, van Goor, H, Warshaw, A, Wang, C, Whitcomb, D, Zhao, Y, Zyromski, N., Surgery, and Gastroenterology and Hepatology

Subjects
musculoskeletal diseases, medicine.medical_specialty, Acute pancreatitis, Diagnosis, Gastroenterology, Guidelines, Intervention, Nutrition, Pancreas, Prediction, Prevention, Radiology, Surgery, Treatment, Acute Disease, Cholecystectomy, Disease Management, Fluid Therapy, Humans, Pancreatitis, Pancreatitis, Acute Necrotizing, Evidence-based practice, Endocrinology, Diabetes and Metabolism, medicine.medical_treatment, viruses, education, Medicine and Health Sciences, medicine, Disease management (health), Intensive care medicine, Grading (education), Hepatology, business.industry, Sciences bio-médicales et agricoles, medicine.disease, body regions, Clinical research, Acute Necrotizing, Etiology, Evaluation of complex medical interventions Tissue engineering and pathology [NCEBP 2], biological phenomena, cell phenomena, and immunity, business
Abstract

There have been substantial improvements in the management of acute pancreatitis since the publication of the International Association of Pancreatology (IAP) treatment guidelines in 2002. A collaboration of the IAP and the American Pancreatic Association (APA) was undertaken to revise these guidelines using an evidence-based approach., 0, Working Group IAP/APA Acute Pancreatitis Guidelines., info:eu-repo/semantics/published

Published
2013

20. Editorial

Author

Nordback I

Subjects
chemistry.chemical_compound, medicine.medical_specialty, Nutrition and Dietetics, chemistry, business.industry, Endocrinology, Diabetes and Metabolism, Internal medicine, medicine, Pancreatitis, Alcohol, medicine.disease, business, Gastroenterology
Published
1998

25. Distal Pancreatectomy Using a No-Touch Isolation Technique

Author

Hirota, M., primary, Hashimoto, D., additional, Ishiko, T., additional, Satoh, N., additional, Takamori, H., additional, Chikamoto, A., additional, Tanaka, H., additional, Sugita, H., additional, Sand, J., additional, Nordback, I., additional, and Baba, H., additional

Published
2012
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28. Prognostic value of analysis of DNA in pancreatic adenocarcinoma by flow cytometry

Author

Hyöty M, Tapio Visakorpi, Op, Kallioniemi, Mattila J, Laippala P, and Nordback I

Subjects
Adult, Male, Ploidies, DNA, Neoplasm, Adenocarcinoma, Middle Aged, Flow Cytometry, Prognosis, Pancreatic Neoplasms, Survival Rate, Multivariate Analysis, Humans, Female, Aged
Abstract

DNA was analysed by flow cytometry in paraffin-embedded tumour specimens from 58 patients who had undergone resection of pancreatic ductal adenocarcinoma, to see if there was any correlation between DNA analysis and survival. Because of the poor quality of the DNA histogram five cases were excluded from the analysis of DNA-ploidy and 10 from the analysis of the synthesis phase fraction. Aneuploidy was detected in 11 (21%) of the 53 tumours. Neither aneuploidy nor synthesis phase fraction correlated significantly with size, stage or differentiation of the tumours. The survival of the patients with DNA-aneuploid tumours (median 9 months) did not differ significantly from that of the patients with diploid tumours (median 14 months). Four patients survived for 5 years or longer, two of whom had aneuploid tumours. Synthesis phase fraction did not correlate with survival. Using univariate survival analyses, location of the tumour in the head of the pancreas (p = 0.02), a tumour diameter of less than 3.8 cm (p = 0.003), tumour stages I and II (p = 0.03), and a well differentiated tumour (p = 0.04) correlated with favourable survival. In a Cox's regression analysis, however, only the diameter of the tumour had independent prognostic value. We conclude that DNA analysis by flow cytometry is not helpful in selecting patients with resectable pancreatic carcinoma who will benefit from resection.

Published
1991

50. Direct effect of thyroxine on pig sphincter of Oddi contractility.

Author

Inkinen, Johanna, Sand, Juhani, Arvola, Pertti, Pörsti, Ilkka, Nordback, Isto, Inkinen, J, Sand, J, Arvola, P, Pörsti, I, and Nordback, I

Abstract

Sphincter of Oddi (SO) motility has an important role in the regulation of bile flow. SO function disturbances (stenosis or dyskinesia) may prevent normal bile flow and thus enhance the probability of common bile duct (CBD) stone formation. Previously we have shown that there is an increased prevalence of diagnosed hypothyroidism in CBD stone patients, compared with gallbladder stone patients or age-, sex-, and hospital-admission-adjusted controls. The present study was done to test the hypothesis that thyroxine directly effects the SO. The specificity of the effects of thyroxine were studied by comparing with triiodothyronine (T3), progesterone, cortisone, estrogen, and testosterone. For ex vivo studies three or four successive 1 to 1.5-mm SO rings were prepared from each pig and placed between two hooks in oxygenated physiologic salt solution at 37°C. SO contraction was measured with isometric force displacement transducers and registered on a polygraph. Each SO ring was stimulated with KCl (125 mM), acetylcholine (ACh; 10 or 100 μM) and histamine (Hist; 10 or 100 μM) with and without thyroxine (10-10 or 10 -8 M), T3 (10-9 or 10-7 M), progesterone (1 μM), cortisone (1 μM), estrogen (1 μM), or testosterone (1 nM) in the medium. KCl, ACh, and Hist induced strong contractions in the SO rings. The addition of thyroxine did not influence significantly the KCl-induced contractions, but the ACh- and Hist-induced contractions decreased by a mean of 37–44% (P < 0.001) and 54–56% (P < 0.001), respectively, as compared to the contractions without thyroxine. Triiodothyronine had a similar inhibitory effect to thyroxine, whereas cortisone, estrogen, and testosterone had no effect. Progesterone decreased the KCl-, ACh-, and Hist-induced SO contractions. In conclusion, physiological concentrations of thyroxine have an inhibitory effect on receptor-mediated ACh and Hist, but not on the nonspecific KCl-induced SO contraction ex vivo. The inhibitory effect is similar in thyroxine and triiodothyronine. Of the steroid hormones, only progesterone nonspecifically ameliorates SO contractions ex vivo. Because the effect of thyroxine on the SO is prorelaxing, the lack of thyroxine may result in an increased tension of the SO. [ABSTRACT FROM AUTHOR]

Published
2001
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